scholarly article | Q13442814 |
P50 | author | Richard Lamont | Q89232703 |
George Hajishengallis | Q39672238 | ||
P2860 | cites work | Porphyromonas gingivalis facilitates the development and progression of destructive arthritis through its unique bacterial peptidylarginine deiminase (PAD) | Q21089593 |
Low-abundance biofilm species orchestrates inflammatory periodontal disease through the commensal microbiota and complement | Q24594632 | ||
Porphyromonas gingivalis as a potential community activist for disease | Q24616806 | ||
The C5a receptor impairs IL-12-dependent clearance of Porphyromonas gingivalis and is required for induction of periodontal bone loss | Q24618908 | ||
Gingipain-dependent interactions with the host are important for survival of Porphyromonas gingivalis | Q24630104 | ||
Intrinsic apoptotic pathways of gingival epithelial cells modulated by Porphyromonas gingivalis | Q24633584 | ||
P. gingivalis accelerates gingival epithelial cell progression through the cell cycle | Q24650372 | ||
Porphyromonas gingivalis-host interactions: open war or intelligent guerilla tactics? | Q24655246 | ||
Porphyromonas gingivalis: an invasive and evasive opportunistic oral pathogen | Q27002394 | ||
Tyrosine phosphorylation and bacterial virulence | Q27009174 | ||
Microbial manipulation of receptor crosstalk in innate immunity | Q30499616 | ||
Identification of a second lipopolysaccharide in Porphyromonas gingivalis W50 | Q33319075 | ||
Efficacy of antibiotics against periodontopathogenic bacteria within epithelial cells: an in vitro study | Q45162034 | ||
Integrin activation by bacterial fimbriae through a pathway involving CD14, Toll-like receptor 2, and phosphatidylinositol-3-kinase | Q45288377 | ||
Activation of complement components C3 and C5 by a cysteine proteinase (gingipain-1) from Porphyromonas (Bacteroides) gingivalis. | Q46312320 | ||
Fimbrial proteins of porphyromonas gingivalis mediate in vivo virulence and exploit TLR2 and complement receptor 3 to persist in macrophages. | Q54476982 | ||
'Blooming' in the gut: how dysbiosis might contribute to pathogen evolution | Q63968591 | ||
Gingival epithelial cell signalling and cytoskeletal responses to Porphyromonas gingivalis invasion | Q64449425 | ||
Persistence of extracrevicular bacterial reservoirs after treatment of aggressive periodontitis. | Q64943983 | ||
Cutting Edge: TLR2 is required for the innate response to Porphyromonas gingivalis: activation leads to bacterial persistence and TLR2 deficiency attenuates induced alveolar bone resorption | Q79401044 | ||
The quorum-sensing molecule N-3-oxododecanoyl homoserine lactone (3OC12-HSL) enhances the host defence by activating human polymorphonuclear neutrophils (PMN) | Q80102959 | ||
Biphasic effect of gingipains from Porphyromonas gingivalis on the human complement system | Q80358557 | ||
Phagocytosis of staphylococci biofilms by polymorphonuclear neutrophils: S. aureus and S. epidermidis differ with regard to their susceptibility towards the host defense | Q84781883 | ||
Destruction of Bacterial Biofilms by Polymorphonuclear Neutrophils: Relative contribution of Phagocytosis, DNA Release, and Degranulation | Q85125504 | ||
Immune defense against S. epidermidis biofilms: components of the extracellular polymeric substance activate distinct bactericidal mechanisms of phagocytic cells | Q87443896 | ||
Ability of bacteria associated with chronic inflammatory disease to stimulate E-selectin expression and promote neutrophil adhesion | Q39821679 | ||
Differential interactions of fimbriae and lipopolysaccharide from Porphyromonas gingivalis with the Toll-like receptor 2-centred pattern recognition apparatus | Q40230062 | ||
TLR2 transmodulates monocyte adhesion and transmigration via Rac1- and PI3K-mediated inside-out signaling in response to Porphyromonas gingivalis fimbriae | Q40271727 | ||
Actinobacillus actinomycetemcomitans, Porphyromonas gingivalis, and Tannerella forsythensis are components of a polymicrobial intracellular flora within human buccal cells. | Q40459069 | ||
Complement factor D̄-like activity of Porphyromonas gingivalis W83 | Q41132518 | ||
Porphyromonas gingivalis infection sequesters pro-apoptotic Bad through Akt in primary gingival epithelial cells | Q41577708 | ||
The gingival tissues: the architecture of periodontal protection | Q41754677 | ||
ATP-dependent activation of an inflammasome in primary gingival epithelial cells infected by Porphyromonas gingivalis | Q41818799 | ||
A Porphyromonas gingivalis tyrosine phosphatase is a multifunctional regulator of virulence attributes | Q41828590 | ||
Intercellular spreading of Porphyromonas gingivalis infection in primary gingival epithelial cells | Q41859338 | ||
Porphyromonas gingivalis SerB-mediated dephosphorylation of host cell cofilin modulates invasion efficiency | Q41982299 | ||
Human Toll-like receptor 4 responses to P. gingivalis are regulated by lipid A 1- and 4'-phosphatase activities | Q42714693 | ||
Microbial hijacking of complement-toll-like receptor crosstalk | Q42911622 | ||
Periodontitis in RA-the citrullinated enolase connection. | Q42916208 | ||
ATP scavenging by the intracellular pathogen Porphyromonas gingivalis inhibits P2X7-mediated host-cell apoptosis. | Q43185488 | ||
Complement receptor 3 blockade promotes IL-12-mediated clearance of Porphyromonas gingivalis and negates its virulence in vivo | Q43985683 | ||
Involvement of integrins in fimbriae-mediated binding and invasion by Porphyromonas gingivalis. | Q44004516 | ||
C5a negatively regulates toll-like receptor 4-induced immune responses | Q44924554 | ||
Host defence against Staphylococcus aureus biofilms infection: phagocytosis of biofilms by polymorphonuclear neutrophils (PMN). | Q33414914 | ||
Proteomics of Porphyromonas gingivalis within a model oral microbial community | Q33447901 | ||
Pathway analysis for intracellular Porphyromonas gingivalis using a strain ATCC 33277 specific database | Q33499084 | ||
Neutrophil fate in gingival crevicular fluid | Q33523593 | ||
Temperature-dependent modulation of Porphyromonas gingivalis lipid A structure and interaction with the innate host defenses. | Q33789766 | ||
Epithelial cell pro-inflammatory cytokine response differs across dental plaque bacterial species | Q33975334 | ||
Dichotomy of gingipains action as virulence factors: from cleaving substrates with the precision of a surgeon's knife to a meat chopper-like brutal degradation of proteins. | Q34076596 | ||
Periodontal innate immune mechanisms relevant to atherosclerosis and obesity | Q34083329 | ||
The human oral microbiome | Q34127792 | ||
Review: Pathogen-induced inflammation at sites distant from oral infection: bacterial persistence and induction of cell-specific innate immune inflammatory pathways. | Q34181823 | ||
Role of Porphyromonas gingivalis phosphoserine phosphatase enzyme SerB in inflammation, immune response, and induction of alveolar bone resorption in rats | Q34290681 | ||
The keystone-pathogen hypothesis. | Q34297138 | ||
Pathogen-mediated proteolysis of the cell death regulator RIPK1 and the host defense modulator RIPK2 in human aortic endothelial cells. | Q34299368 | ||
Periodontal diseases. | Q34468885 | ||
Beyond the red complex and into more complexity: the polymicrobial synergy and dysbiosis (PSD) model of periodontal disease etiology. | Q34470176 | ||
Structural Dissection and In Vivo Effectiveness of a Peptide Inhibitor of Porphyromonas gingivalis Adherence to Streptococcus gordonii | Q34484719 | ||
Microbial interactions in building of communities | Q34518087 | ||
The subgingival microbiome in health and periodontitis and its relationship with community biomass and inflammation | Q34543230 | ||
The serine phosphatase SerB of Porphyromonas gingivalis suppresses IL-8 production by dephosphorylation of NF-κB RelA/p65 | Q34699747 | ||
The atherogenic bacterium Porphyromonas gingivalis evades circulating phagocytes by adhering to erythrocytes | Q34740007 | ||
Roles of the host oxidative immune response and bacterial antioxidant rubrerythrin during Porphyromonas gingivalis infection. | Q34862986 | ||
Review of osteoimmunology and the host response in endodontic and periodontal lesions | Q34920792 | ||
A Porphyromonas gingivalis haloacid dehalogenase family phosphatase interacts with human phosphoproteins and is important for invasion | Q34984268 | ||
Corruption of innate immunity by bacterial proteases | Q35003110 | ||
Origin and function of the cellular components in gingival crevice fluid | Q35092082 | ||
The pathogenic persona of community-associated oral streptococci | Q35640402 | ||
"Gum bug, leave my heart alone!"--epidemiologic and mechanistic evidence linking periodontal infections and atherosclerosis. | Q35866675 | ||
Distinct and complex bacterial profiles in human periodontitis and health revealed by 16S pyrosequencing | Q35979362 | ||
Porphyromonas gingivalis promotes Th17 inducing pathways in chronic periodontitis | Q36157309 | ||
Microbial carriage state of peripheral blood dendritic cells (DCs) in chronic periodontitis influences DC differentiation, atherogenic potential. | Q36284016 | ||
Local complement-targeted intervention in periodontitis: proof-of-concept using a C5a receptor (CD88) antagonist | Q36417215 | ||
Role of TLR2-dependent IL-10 production in the inhibition of the initial IFN-γ T cell response to Porphyromonas gingivalis. | Q36476646 | ||
The role of T cells in periodontal disease: homeostasis and autoimmunity | Q36703937 | ||
Role of differential expression of streptococcal arginine deiminase in inhibition of fimA expression in Porphyromonas gingivalis | Q36747267 | ||
Porphyromonas gingivalis-nucleoside-diphosphate-kinase inhibits ATP-induced reactive-oxygen-species via P2X7 receptor/NADPH-oxidase signalling and contributes to persistence. | Q36763229 | ||
A novel class of lipoprotein lipase-sensitive molecules mediates Toll-like receptor 2 activation by Porphyromonas gingivalis | Q36804694 | ||
Bacterial invasion of epithelial cells and spreading in periodontal tissue | Q36824616 | ||
Exploring the genetic basis of chronic periodontitis: a genome-wide association study | Q36839033 | ||
Subgingival biofilm formation | Q36880067 | ||
Suppression of T-cell chemokines by Porphyromonas gingivalis | Q36970761 | ||
Binding of complement inhibitor C4b-binding protein contributes to serum resistance of Porphyromonas gingivalis | Q37015215 | ||
Induction of bone loss by pathobiont-mediated Nod1 signaling in the oral cavity | Q37040382 | ||
Importance of TLR2 in early innate immune response to acute pulmonary infection with Porphyromonas gingivalis in mice | Q37059185 | ||
The human microbiome: from symbiosis to pathogenesis | Q37064798 | ||
Porphyromonas gingivalis RgpA-Kgp proteinase-adhesin complexes penetrate gingival tissue and induce proinflammatory cytokines or apoptosis in a concentration-dependent manner | Q37099426 | ||
Serine lipids of Porphyromonas gingivalis are human and mouse Toll-like receptor 2 ligands | Q37123738 | ||
A new inflammatory cytokine on the block: re-thinking periodontal disease and the Th1/Th2 paradigm in the context of Th17 cells and IL-17. | Q37249268 | ||
Modulation of TLR2 protein expression by miR-105 in human oral keratinocytes | Q37371743 | ||
Negative correlation of distributions of Streptococcus cristatus and Porphyromonas gingivalis in subgingival plaque | Q37451753 | ||
Disulfide formation in the ER and mitochondria: two solutions to a common process. | Q37508527 | ||
Neutrophil homeostasis and periodontal health in children and adults | Q37594566 | ||
Crosstalk pathways between Toll-like receptors and the complement system | Q37691806 | ||
Comparison of neutrophil functions in aggressive and chronic periodontitis | Q37733777 | ||
Genetic and environmental risk factors for chronic periodontitis and aggressive periodontitis | Q37733780 | ||
Periodontitis: a polymicrobial disruption of host homeostasis | Q37761827 | ||
Destructive and protective roles of cytokines in periodontitis: a re-appraisal from host defense and tissue destruction viewpoints | Q37782516 | ||
Targeting IL-17 and TH17 cells in chronic inflammation | Q38047820 | ||
Mucosal innate immune cells regulate both gut homeostasis and intestinal inflammation | Q38177725 | ||
Mechanisms of resistance of Porphyromonas gingivalis to killing by serum complement | Q38481466 | ||
Distinct transcriptional profiles characterize oral epithelium-microbiota interactions | Q38520459 | ||
Microbial complexes in subgingival plaque | Q38553206 | ||
Analysis of the intracellular fate of Legionella pneumophila mutants | Q38559824 | ||
Porphyromonas gingivalis induction of microRNA-203 expression controls suppressor of cytokine signaling 3 in gingival epithelial cells | Q38686982 | ||
Local chemokine paralysis, a novel pathogenic mechanism for Porphyromonas gingivalis. | Q39570767 | ||
Exit of intracellular Porphyromonas gingivalis from gingival epithelial cells is mediated by endocytic recycling pathway | Q39620387 | ||
Streptococcus cristatus ArcA interferes with Porphyromonas gingivalis pathogenicity in mice | Q39745708 | ||
P433 | issue | 2 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | Porphyromonas gingivalis | Q3214147 |
P304 | page(s) | 328-338 | |
P577 | publication date | 2014-02-01 | |
P1433 | published in | European Journal of Immunology | Q5412727 |
P1476 | title | Breaking bad: manipulation of the host response by Porphyromonas gingivalis | |
P478 | volume | 44 |
Q47364041 | A novel peptidic inhibitor derived from Streptococcus cristatus ArcA attenuates virulence potential of Porphyromonas gingivalis. |
Q99233987 | A therapeutic oxygen carrier isolated from Arenicola marina decreased P. gingivalis induced inflammation and tissue destruction |
Q38756340 | Activation of the TREM-1 pathway in human monocytes by periodontal pathogens and oral commensal bacteria. |
Q47214655 | Analysis of differential expression of tight junction proteins in cultured oral epithelial cells altered by Porphyromonas gingivalis, Porphyromonas gingivalis lipopolysaccharide, and extracellular adenosine triphosphate |
Q64081308 | Apoptosis Transcriptional Profile Induced by HmuY |
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Q35932948 | Basic biology and role of interleukin-17 in immunity and inflammation |
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Q57295298 | Characterization and development of SAPP as a specific peptidic inhibitor that targets Porphyromonas gingivalis |
Q41566377 | Characterization of Innate Immune Responses of Human Endothelial Cells Induced by Porphyromonas gingivalis and Their Derived Outer Membrane Vesicles |
Q42721477 | Characterization of a bacterial tyrosine kinase in Porphyromonas gingivalis involved in polymicrobial synergy |
Q89900618 | Characterization of intratissue bacterial communities and isolation of Escherichia coli from oral lichen planus lesions |
Q35241492 | Cigarette smoke condensate modulates migration of human gingival epithelial cells and their interactions with Porphyromonas gingivalis. |
Q104079208 | Clinical significance and prognostic value of Porphyromonas gingivalis infection in lung cancer |
Q41361397 | Combined antioxidant effects of Neem extract, bacteria, red blood cells and Lysozyme: possible relation to periodontal disease. |
Q54228902 | Community Development between Porphyromonas gingivalis and Candida albicans Mediated by InlJ and Als3. |
Q39165180 | Dental biofilm: ecological interactions in health and disease. |
Q38697615 | Differential Regulation of Mas-Related G Protein-Coupled Receptor X2-Mediated Mast Cell Degranulation by Antimicrobial Host Defense Peptides and Porphyromonas gingivalis Lipopolysaccharide |
Q39848642 | Differential capacity for complement receptor-mediated immune evasion by Porphyromonas gingivalis depending on the type of innate leukocyte |
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Q92090423 | Fetal Weight Outcomes in C57BL/6J and C57BL/6NCrl Mice after Oral Colonization with Porphyromonas gingivalis |
Q40534389 | Filifactor alocis Promotes Neutrophil Degranulation and Chemotactic Activity. |
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Q92773036 | Gene expression of Porphyromonas gingivalis ATCC 33277 when growing in an in vitro multispecies biofilm |
Q30582298 | Genetic and intervention studies implicating complement C3 as a major target for the treatment of periodontitis |
Q37695490 | Genome Sequence of Porphyromonas gingivalis Strain A7A1-28. |
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Q91723711 | Identification of PGN_1123 as the Gene Encoding Lipid A Deacylase, an Enzyme Required for Toll-Like Receptor 4 Evasion, in Porphyromonas gingivalis |
Q38687979 | Identification of Streptococcus cristatus peptides that repress expression of virulence genes in Porphyromonas gingivalis. |
Q93022516 | Identification of functional domains of the minor fimbrial antigen involved in the interaction of Porphyromonas gingivalis with oral streptococci |
Q41130655 | Identification of quantitative trait loci influencing inflammation-mediated alveolar bone loss: insights into polygenic inheritance of host-biofilm disequilibria in periodontitis |
Q91649623 | Implication of Porphyromonas gingivalis in colitis and homeostasis of intestinal epithelium |
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Q40294338 | Insights into Dynamic Polymicrobial Synergy Revealed by Time-Coursed RNA-Seq |
Q92358830 | Is there a link between genetic defects in the complement cascade and Porphyromonas gingivalis in Alzheimer's disease? |
Q91641184 | Killing mechanism of bacteria within multi-species biofilm by blue light |
Q35688116 | MFG-E8, a novel homeostatic regulator of osteoclastogenesis |
Q40541431 | Mannose-binding lectin gene polymorphism in relation to periodontal infection. |
Q38722715 | Marginal bone loss around non-submerged implants is associated with salivary microbiome during bone healing |
Q55279386 | Maturation of the Mfa1 Fimbriae in the Oral Pathogen Porphyromonas gingivalis. |
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Q41945242 | Modulation of host responses by oral commensal bacteria |
Q26768562 | Modulation of inflammasome activity by Porphyromonas gingivalis in periodontitis and associated systemic diseases |
Q26750529 | Neutrophil Functions in Periodontal Homeostasis |
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Q34203498 | Neutrophil mobilization by surface-glycan altered Th17-skewing bacteria mitigates periodontal pathogen persistence and associated alveolar bone loss |
Q64226778 | Nonulosonic acids contribute to the pathogenicity of the oral bacterium |
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Q38983345 | Phosphoglycerol dihydroceramide, a distinctive ceramide produced by Porphyromonas gingivalis, promotes RANKL-induced osteoclastogenesis by acting on non-muscle myosin II-A (Myh9), an osteoclast cell fusion regulatory factor |
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