GluN1 deletions in D1- and A2A-expressing cell types reveal distinct modes of behavioral regulation

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GluN1 deletions in D1- and A2A-expressing cell types reveal distinct modes of behavioral regulation is …
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scholarly articleQ13442814

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P356DOI10.1016/J.NEUROPHARM.2016.03.026
P932PMC publication ID5031515
P698PubMed publication ID27012890

P50authorMax E JoffeQ86145961
Brad A. GrueterQ42822736
P2093author name stringSophie R Vitter
P2860cites workCloning and expression of human and rat D1 dopamine receptorsQ24306738
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Expression of D1 receptor, D2 receptor, substance P and enkephalin messenger RNAs in the neurons projecting from the nucleus accumbensQ48522950
P433issuePt A
P407language of work or nameEnglishQ1860
P1104number of pages9
P304page(s)172-180
P577publication date2016-03-21
P1433published inNeuropharmacologyQ15332439
P1476titleGluN1 deletions in D1- and A2A-expressing cell types reveal distinct modes of behavioral regulation
P478volume112

Reverse relations

cites work (P2860)
Q97692907Dendritic spine density is increased on nucleus accumbens D2 neurons after chronic social defeat
Q38814541Do specific NMDA receptor subunits act as gateways for addictive behaviors?
Q39015823Emerging Role for Nucleus Accumbens Medium Spiny Neuron Subtypes in Depression
Q89456486Genetic loss of GluN2B in D1-expressing cell types enhances long-term cocaine reward and potentiation of thalamo-accumbens synapses
Q92987643The molecular and cellular mechanisms of depression: a focus on reward circuitry
Q47810676Toll-like receptor 4 deficiency alters nucleus accumbens synaptic physiology and drug reward behavior

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