Role for c-jun N-terminal kinase in treatment-refractory acute myeloid leukemia (AML): signaling to multidrug-efflux and hyperproliferation

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Role for c-jun N-terminal kinase in treatment-refractory acute myeloid leukemia (AML): signaling to multidrug-efflux and hyperproliferation is …
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scholarly articleQ13442814

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P6179Dimensions Publication ID1052387031
P356DOI10.1038/SJ.LEU.2402457
P698PubMed publication ID11986940
P5875ResearchGate publication ID11383026

P2093author name stringCripe LD
Jung SH
Phillips CA
Hartman AD
Smith EA
Mercola D
Boswell HS
Burgess GS
Gabig TG
Fyffe J
Gelfanov VM
Kneebone P
Spigel DR
P2860cites workRegulation of JNK signaling by GSTpQ24533822
Control of cell cycle progression by c-Jun is p53 dependentQ24607483
The jun proto-oncogene is positively autoregulated by its product, Jun/AP-1Q28291340
Identification of a putative antioxidant response element in the 5'-flanking region of the human gamma-glutamylcysteine synthetase heavy subunit geneQ28300319
Biochemistry of multidrug resistance mediated by the multidrug transporterQ29616476
The Bcr-Abl leukemia oncogene activates Jun kinase and requires Jun for transformationQ33720267
The ARF/p53 pathwayQ33840421
Role of glutathione in the export of compounds from cells by the multidrug-resistance-associated proteinQ33999305
Mitogenic signaling mediated by oxidants in Ras-transformed fibroblastsQ34418354
The presence of a FLT3 internal tandem duplication in patients with acute myeloid leukemia (AML) adds important prognostic information to cytogenetic risk group and response to the first cycle of chemotherapy: analysis of 854 patients from the UniteQ34517403
Detoxication of base propenals and other alpha, beta-unsaturated aldehyde products of radical reactions and lipid peroxidation by human glutathione transferasesQ35053135
Cellular transformation and malignancy induced by ras require c-junQ36561672
Oncoprotein-mediated signalling cascade stimulates c-Jun activity by phosphorylation of serines 63 and 73Q36821325
Coordinated induction of MRP/GS-X pump and gamma-glutamylcysteine synthetase by heavy metals in human leukemia cellsQ38356356
High-intensity Raf signal causes cell cycle arrest mediated by p21Cip1.Q40023426
Regulation of the fission yeast transcription factor Pap1 by oxidative stress: requirement for the nuclear export factor Crm1 (Exportin) and the stress-activated MAP kinase Sty1/Spc1Q40444338
The mammalian UV response: c-Jun induction is required for exit from p53-imposed growth arrestQ40834838
Biology of the multidrug resistance-associated protein, MRP.Q41078120
Glutathione and related enzymes in multidrug resistanceQ41078132
Modulation of activity of the promoter of the human MDR1 gene by Ras and p53.Q41643433
Regulation of human glutathione S-transferase pi gene transcription: influence of 5'-flanking sequences and trans-activating factors which recognize AP-1-binding sitesQ41737010
Role of the stress-activated/c-Jun NH2-terminal protein kinase pathway in the cellular response to adriamycin and other chemotherapeutic drugsQ42527540
Amplification of P-glycoprotein genes in multidrug-resistant mammalian cell linesQ42818012
The Jun kinase/stress-activated protein kinase pathway functions to regulate DNA repair and inhibition of the pathway sensitizes tumor cells to cisplatinQ46727285
Role of Atf1 and Pap1 in the induction of the catalase gene of fission yeast schizosaccharomyces pombe.Q52169787
Molecular evolution of acute myeloid leukaemia in relapse: unstable N-ras and FLT3 genes compared with p53 gene.Q54100578
Expression of multidrug resistance protein/GS-X pump and gamma-glutamylcysteine synthetase genes is regulated by oxidative stress.Q54117225
P53 gene mutations in acute myelogenous leukaemiaQ67549556
Direct involvement of the Y-box binding protein YB-1 in genotoxic stress-induced activation of the human multidrug resistance 1 geneQ74299207
P433issue5
P921main subjectacute myeloid leukemiaQ264118
childhood acute myeloid leukemiaQ55779816
P304page(s)799-812
P577publication date2002-05-01
P1433published inLeukemiaQ6534498
P1476titleRole for c-jun N-terminal kinase in treatment-refractory acute myeloid leukemia (AML): signaling to multidrug-efflux and hyperproliferation
P478volume16