editorial | Q871232 |
scholarly article | Q13442814 |
P50 | author | Arthur Kaser | Q88612415 |
P2093 | author name string | Richard S Blumberg | |
Michal Tomczak | |||
P2860 | cites work | Genome-wide association defines more than 30 distinct susceptibility loci for Crohn's disease | Q22251069 |
XBP1 links ER stress to intestinal inflammation and confers genetic risk for human inflammatory bowel disease | Q22252318 | ||
Genome-wide association study of 14,000 cases of seven common diseases and 3,000 shared controls | Q24550675 | ||
Virus-plus-susceptibility gene interaction determines Crohn's disease gene Atg16L1 phenotypes in intestine | Q24604466 | ||
Homeostasis and inflammation in the intestine | Q24613509 | ||
Paneth cells directly sense gut commensals and maintain homeostasis at the intestinal host-microbial interface | Q24644721 | ||
Genome-wide association study identifies new susceptibility loci for Crohn disease and implicates autophagy in disease pathogenesis | Q24656576 | ||
Hypoxia: an alarm signal during intestinal inflammation | Q27691319 | ||
Signal integration in the endoplasmic reticulum unfolded protein response | Q27860577 | ||
Genome-wide association identifies multiple ulcerative colitis susceptibility loci | Q27865246 | ||
Secretion of microbicidal alpha-defensins by intestinal Paneth cells in response to bacteria | Q28204776 | ||
Regulation of hepatic lipogenesis by the transcription factor XBP1 | Q28507784 | ||
XBP-1 is required for biogenesis of cellular secretory machinery of exocrine glands | Q28510479 | ||
The transcription factor XBP-1 is essential for the development and survival of dendritic cells | Q28512302 | ||
Nod2-dependent regulation of innate and adaptive immunity in the intestinal tract | Q28587612 | ||
Regulation mechanisms and signaling pathways of autophagy | Q29547416 | ||
The gut microbiota shapes intestinal immune responses during health and disease | Q29547727 | ||
A key role for autophagy and the autophagy gene Atg16l1 in mouse and human intestinal Paneth cells | Q29614565 | ||
Sequence variants in the autophagy gene IRGM and multiple other replicating loci contribute to Crohn's disease susceptibility | Q29614872 | ||
A genome-wide association scan of nonsynonymous SNPs identifies a susceptibility variant for Crohn disease in ATG16L1 | Q29614873 | ||
Inflammatory bowel disease | Q29615500 | ||
Inflammatory bowel disease | Q29616286 | ||
Intestinal crypt homeostasis results from neutral competition between symmetrically dividing Lgr5 stem cells | Q29619410 | ||
Investigation of Crohn's disease risk loci in ulcerative colitis further defines their molecular relationship | Q34899429 | ||
TLR activation of the transcription factor XBP1 regulates innate immune responses in macrophages. | Q35041384 | ||
The endoplasmic reticulum stress response in immunity and autoimmunity | Q37231726 | ||
Endoplasmic reticulum stress in the intestinal epithelium and inflammatory bowel disease | Q37399734 | ||
Ischemia activates the ATF6 branch of the endoplasmic reticulum stress response | Q37447782 | ||
Enteric defensins are essential regulators of intestinal microbial ecology | Q37483232 | ||
The genetics of Crohn's disease | Q37488205 | ||
Endoplasmic reticulum stress and intestinal inflammation | Q37623437 | ||
Endoplasmic reticulum stress: implications for inflammatory bowel disease pathogenesis | Q37760005 | ||
Disruption of Paneth and goblet cell homeostasis and increased endoplasmic reticulum stress in Agr2-/- mice | Q41977500 | ||
B- and T-cell development both involve activity of the unfolded protein response pathway. | Q51790130 | ||
Level of activation of the unfolded protein response correlates with Paneth cell apoptosis in human small intestine exposed to ischemia/reperfusion. | Q53294250 | ||
The Paneth cell alpha-defensin deficiency of ileal Crohn's disease is linked to Wnt/Tcf-4 | Q80819683 | ||
P433 | issue | 2 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | reperfusion injury | Q1413991 |
P304 | page(s) | 393-396 | |
P577 | publication date | 2010-12-18 | |
P1433 | published in | Gastroenterology | Q4039279 |
P1476 | title | "ER stress(ed out)!": Paneth cells and ischemia-reperfusion injury of the small intestine | |
P478 | volume | 140 |
Q38270128 | Animal models of ischemia-reperfusion-induced intestinal injury: progress and promise for translational research. |
Q36431001 | Mucosal Immune System and M Cell-targeting Strategies for Oral Mucosal Vaccination |
Q51069633 | Protective effect of glutamine on the main and adjacent organs damaged by ischemia-reperfusion in rats. |
Q35085852 | The cell biology of the unfolded protein response. |
Q27676939 | The α-defensin salt-bridge induces backbone stability to facilitate folding and confer proteolytic resistance |
Q90446620 | Unfolded protein response (UPR) integrated signaling networks determine cell fate during hypoxia |
Q37745551 | Upregulation of miR-665 promotes apoptosis and colitis in inflammatory bowel disease by repressing the endoplasmic reticulum stress components XBP1 and ORMDL3. |
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