Genetic susceptibility to increased bacterial translocation influences the response to biological therapy in patients with Crohn's disease.

scientific article published in February 2013

Genetic susceptibility to increased bacterial translocation influences the response to biological therapy in patients with Crohn's disease. is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1136/GUTJNL-2012-303557
P698PubMed publication ID23376290
P5875ResearchGate publication ID235396551

P50authorJosé M González-NavajasQ39836999
Pedro ZapaterQ39837042
Rubén FrancésQ59698583
Laura SempereQ60316164
P2093author name stringMichael Scharl
Gerhard Rogler
Reiner Wiest
Ernst Holler
Ana Gutiérrez
José Such
Isabel Almenta
P2860cites workInflammatory Bowel DiseaseQ22248115
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Nod2 is a general sensor of peptidoglycan through muramyl dipeptide (MDP) detectionQ24292675
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Genome-wide meta-analysis increases to 71 the number of confirmed Crohn's disease susceptibility lociQ24618592
Genome-wide association study identifies new susceptibility loci for Crohn disease and implicates autophagy in disease pathogenesisQ24656576
Host recognition of bacterial muramyl dipeptide mediated through NOD2. Implications for Crohn's diseaseQ28201834
Maintenance infliximab for Crohn's disease: the ACCENT I randomised trialQ28202504
Infliximab maintenance therapy for fistulizing Crohn's diseaseQ28246982
Toll-like receptor signalling in macrophages links the autophagy pathway to phagocytosisQ29614481
A genome-wide association scan of nonsynonymous SNPs identifies a susceptibility variant for Crohn disease in ATG16L1Q29614873
Impaired autophagy of an intracellular pathogen induced by a Crohn's disease associated ATG16L1 variantQ33375721
Infliximab for the treatment of fistulas in patients with Crohn's diseaseQ33860666
ATG16L1 and NOD2 interact in an autophagy-dependent antibacterial pathway implicated in Crohn's disease pathogenesisQ34258672
Association of NOD2 (CARD 15) genotype with clinical course of Crohn's disease: a cohort studyQ34523337
From symptom to diagnosis: clinical distinctions among various forms of intestinal inflammationQ35787421
Systematic review: is ingestion of paracetamol or non-steroidal anti-inflammatory drugs associated with exacerbations of inflammatory bowel disease?Q35966079
Experimental models of inflammatory bowel disease reveal innate, adaptive, and regulatory mechanisms of host dialogue with the microbiotaQ36209737
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Individual medicine in inflammatory bowel disease: monitoring bioavailability, pharmacokinetics and immunogenicity of anti-tumour necrosis factor-alpha antibodiesQ37367325
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Distribution and manifestations of inflammatory bowel disease in Asians, Hispanics, and African Americans: a systematic reviewQ37496554
What is the optimal therapy for Crohn's disease: step-up or top-down?Q37720746
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CpG motifs of bacterial DNA essentially contribute to the perpetuation of chronic intestinal inflammationQ38321426
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The molecular classification of the clinical manifestations of Crohn's diseaseQ40651190
Epistasis between Toll-like receptor-9 polymorphisms and variants in NOD2 and IL23R modulates susceptibility to Crohn's diseaseQ45758087
Muramyl dipeptide and toll-like receptor sensitivity in NOD2-associated Crohn's diseaseQ46504948
Differential effects of NOD2 variants on Crohn's disease risk and phenotype in diverse populations: a metaanalysis.Q52970473
Efficacy and safety of retreatment with anti-tumor necrosis factor antibody (infliximab) to maintain remission in Crohn's disease.Q53540951
Infliximab, azathioprine, or combination therapy for Crohn's diseaseQ56988763
P433issue2
P407language of work or nameEnglishQ1860
P304page(s)272-280
P577publication date2013-02-01
P1433published inGutQ5621669
P1476titleGenetic susceptibility to increased bacterial translocation influences the response to biological therapy in patients with Crohn's disease
P478volume63

Reverse relations

cites work (P2860)
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