| review article | Q7318358 |
| scholarly article | Q13442814 |
| P2093 | author name string | David Murray | |
| Razmik Mirzayans | |||
| P2860 | cites work | Adaptive mutation: implications for evolution | Q24623723 |
| Endopolyploidy in irradiated p53-deficient tumour cell lines: persistence of cell division activity in giant cells expressing Aurora-B kinase | Q24650663 | ||
| Classification of cell death: recommendations of the Nomenclature Committee on Cell Death 2009 | Q24653924 | ||
| Stress-triggered atavistic reprogramming (STAR) addiction: driving force behind head and neck cancer? | Q26739748 | ||
| Cell Fusion in the War on Cancer: A Perspective on the Inception of Malignancy | Q26740111 | ||
| The Dark Side of Cell Fusion | Q26748224 | ||
| The Growing Complexity of Cancer Cell Response to DNA-Damaging Agents: Caspase 3 Mediates Cell Death or Survival? | Q26749064 | ||
| Fusion of bone marrow-derived cells with cancer cells: metastasis as a secondary disease in cancer | Q27000378 | ||
| The "virgin birth", polyploidy, and the origin of cancer | Q27024916 | ||
| Mechanisms of disseminated cancer cell dormancy: an awakening field | Q27027015 | ||
| Stem cells, senescence, neosis and self-renewal in cancer | Q27497212 | ||
| p53-mediated senescence impairs the apoptotic response to chemotherapy and clinical outcome in breast cancer. | Q27851827 | ||
| Tumor Budding, Micropapillary Pattern, and Polyploidy Giant Cancer Cells in Colorectal Cancer: Current Status and Future Prospects | Q28075498 | ||
| Budded karyoplasts from multinucleated fibroblast cells contain centrosomes and change their morphology to mitotic cells | Q40346120 | ||
| Spontaneous cell transformation: karyoplasts derived from multinucleated cells produce new cell growth in senescent human epithelial cell cultures | Q40505229 | ||
| Release of mitotic descendants by giant cells from irradiated Burkitt's lymphoma cell line.. | Q40858282 | ||
| Self-inflicted DNA double-strand breaks sustain tumorigenicity and stemness of cancer cells | Q41070253 | ||
| Essential roles of Caspase-3 in facilitating Myc-induced genetic instability and carcinogenesis | Q41329913 | ||
| Role of p16(INK4A) in Replicative Senescence and DNA Damage-Induced Premature Senescence in p53-Deficient Human Cells | Q42286334 | ||
| Vital dyes and virtual deaths | Q43130530 | ||
| Measuring apoptosis by microscopy and flow cytometry | Q44375924 | ||
| Autophagy, senescence and tumor dormancy in cancer therapy | Q44950633 | ||
| A molecular signature for anastasis, recovery from the brink of apoptotic cell death. | Q45952373 | ||
| Drug-tolerant persister cancer cells are vulnerable to GPX4 inhibition. | Q46265031 | ||
| Aberrant p21WAF1-dependent growth arrest as the possible mechanism of abnormal resistance to ultraviolet light cytotoxicity in Li-Fraumeni syndrome fibroblast strains heterozygous for TP53 mutations | Q47684754 | ||
| Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018. | Q47843948 | ||
| Cancer. Fusion for moving | Q48887336 | ||
| Cell-cell fusion as a mechanism of DNA exchange in cancer | Q49787586 | ||
| A distinct oncogenerative multinucleated cancer cell serves as a source of stemness and tumor heterogeneity. | Q50027351 | ||
| Sulforhodamine B colorimetric assay for cytotoxicity screening. | Q51032435 | ||
| Neosis: a novel type of cell division in cancer. | Q51830250 | ||
| Caspase-3 regulates the migration, invasion and metastasis of colon cancer cells. | Q52430740 | ||
| The Biology and Therapeutic Implications of Tumor Dormancy and Reactivation. | Q52608415 | ||
| Endoreplication: The Good, the Bad, and the Ugly. | Q52724873 | ||
| Neosis and its potential role in cancer development and chemoresistance. | Q53371158 | ||
| DNA damage and its repair in human normal or xeroderma pigmentosum fibroblasts treated with 4-nitroquinoline 1-oxide or its 3-methyl derivative. | Q53573640 | ||
| Resistance to cancer chemotherapy as an atavism? (retrospective on DOI 10.1002/bies.201300170). | Q53764237 | ||
| Apoptosis Reversal Promotes Cancer Stem Cell-Like Cell Formation. | Q53841482 | ||
| Apoptotic protease activating factor 1 (Apaf-1)-independent cell death suppression by Bcl-2. | Q36368462 | ||
| The role of transient hypermutators in adaptive mutation in Escherichia coli | Q36384221 | ||
| Polyploidy Formation in Doxorubicin-Treated Cancer Cells Can Favor Escape from Senescence | Q36397010 | ||
| The number of polyploid giant cancer cells and epithelial-mesenchymal transition-related proteins are associated with invasion and metastasis in human breast cancer | Q36402074 | ||
| Effects of the protein kinase inhibitors wortmannin and KN62 on cellular radiosensitivity and radiation-activated S phase and G1/S checkpoints in normal human fibroblasts | Q36619914 | ||
| β-Lactam antibiotics promote bacterial mutagenesis via an RpoS-mediated reduction in replication fidelity | Q36737307 | ||
| Low levels of Caspase-3 predict favourable response to 5FU-based chemotherapy in advanced colorectal cancer: Caspase-3 inhibition as a therapeutic approach | Q36847557 | ||
| Imaging caspase-3 activation as a marker of apoptosis-targeted treatment response in cancer | Q36920403 | ||
| Multiplex caspase activity and cytotoxicity assays | Q37050828 | ||
| Generation of cancer stem-like cells through the formation of polyploid giant cancer cells | Q37351339 | ||
| Molecular signature of anastasis for reversal of apoptosis | Q37643379 | ||
| MOS, aneuploidy and the ploidy cycle of cancer cells | Q37777023 | ||
| Dying cell clearance and its impact on the outcome of tumor radiotherapy | Q38042950 | ||
| Cell fusion is a potent inducer of aneuploidy and drug resistance in tumor cell/ normal cell hybrids | Q38067161 | ||
| Coming full circle-from endless complexity to simplicity and back again | Q38200253 | ||
| Size Does Matter: Why Polyploid Tumor Cells are Critical Drug Targets in the War on Cancer. | Q38218129 | ||
| Dormant Cells: The Original Cause of Tumor Recurrence and Metastasis | Q38306308 | ||
| Is Senescence Reversible? | Q38572733 | ||
| Premature aging/senescence in cancer cells facing therapy: good or bad? | Q38578680 | ||
| EGF receptor inhibition radiosensitizes NSCLC cells by inducing senescence in cells sustaining DNA double-strand breaks | Q38609721 | ||
| Do Multiwell Plate High Throughput Assays Measure Loss of Cell Viability Following Exposure to Genotoxic Agents? | Q38647105 | ||
| Multinucleated polyploidy drives resistance to Docetaxel chemotherapy in prostate cancer | Q38710996 | ||
| Multinucleated Giant Cancer Cells Produced in Response to Ionizing Radiation Retain Viability and Replicate Their Genome | Q38716153 | ||
| Dying glioma cells establish a proangiogenic microenvironment through a caspase 3 dependent mechanism | Q38732164 | ||
| Altered interactions between unicellular and multicellular genes drive hallmarks of transformation in a diverse range of solid tumors | Q38796992 | ||
| Spontaneous γH2AX Foci in Human Solid Tumor-Derived Cell Lines in Relation to p21WAF1 and WIP1 Expression | Q38871254 | ||
| Radiation-induced homotypic cell fusions of innately resistant glioblastoma cells mediate their sustained survival and recurrence | Q38888602 | ||
| Therapeutic resistance and cancer recurrence mechanisms: Unfolding the story of tumour coming back | Q38942907 | ||
| Significance of Wild-Type p53 Signaling in Suppressing Apoptosis in Response to Chemical Genotoxic Agents: Impact on Chemotherapy Outcome | Q39269601 | ||
| Cancer adaptations: Atavism, de novo selection, or something in between? | Q39425125 | ||
| Characterization of hybrid cells derived from spontaneous fusion events between breast epithelial cells exhibiting stem-like characteristics and breast cancer cells | Q39638952 | ||
| Decreased Expression of the DNA Mismatch Repair Gene Mlh1 under Hypoxic Stress in Mammalian Cells | Q39745939 | ||
| Tumor cells can escape DNA-damaging cisplatin through DNA endoreduplication and reversible polyploidy | Q39972815 | ||
| Active caspase 3 and DNA fragmentation as markers for apoptotic cell death in primary and metastatic liver tumours | Q40047713 | ||
| Co-repression of mismatch repair gene expression by hypoxia in cancer cells: role of the Myc/Max network | Q40175307 | ||
| Mitotic catastrophe results in cell death by caspase-dependent and caspase-independent mechanisms. | Q40345586 | ||
| Improving anticancer drug development begins with cell culture: misinformation perpetrated by the misuse of cytotoxicity assays | Q28077848 | ||
| Ataxia telangiectasia mutated and p21CIP1 modulate cell survival of drug-induced senescent tumor cells: implications for chemotherapy | Q28272930 | ||
| Evaluation of a soluble tetrazolium/formazan assay for cell growth and drug sensitivity in culture using human and other tumor cell lines | Q28297800 | ||
| A chromatin-mediated reversible drug-tolerant state in cancer cell subpopulations | Q29614275 | ||
| The NCI60 human tumour cell line anticancer drug screen | Q29614975 | ||
| New colorimetric cytotoxicity assay for anticancer-drug screening | Q29615418 | ||
| Mitochondria and cell death: outer membrane permeabilization and beyond | Q29615459 | ||
| The senescence-associated secretory phenotype: the dark side of tumor suppression | Q29620106 | ||
| Reversibility of apoptosis in cancer cells | Q30485754 | ||
| Comparison of in vitro anticancer-drug-screening data generated with a tetrazolium assay versus a protein assay against a diverse panel of human tumor cell lines | Q30617388 | ||
| Three steps to the immortality of cancer cells: senescence, polyploidy and self-renewal | Q33590178 | ||
| Apoptosis, p53, and tumor cell sensitivity to anticancer agents | Q33590964 | ||
| Caspase 3 promotes surviving melanoma tumor cell growth after cytotoxic therapy | Q33610993 | ||
| Senescent cells as a source of inflammatory factors for tumor progression | Q33833881 | ||
| Genome-wide hypermutation in a subpopulation of stationary-phase cells underlies recombination-dependent adaptive mutation | Q33886793 | ||
| The SOS response regulates adaptive mutation | Q33903483 | ||
| Polyploid giant cells provide a survival mechanism for p53 mutant cells after DNA damage. | Q33915750 | ||
| Polyploid giant cancer cells with budding and the expression of cyclin E, S-phase kinase-associated protein 2, stathmin associated with the grading and metastasis in serous ovarian tumor | Q34062677 | ||
| Death receptor signals to mitochondria | Q34139596 | ||
| Spontaneous cancer-stromal cell fusion as a mechanism of prostate cancer androgen-independent progression | Q34374537 | ||
| Adaptive mutations, mutator DNA polymerases and genetic change strategies of pathogens | Q34392595 | ||
| Action of x-rays on mammalian cells | Q34648824 | ||
| Feasibility of drug screening with panels of human tumor cell lines using a microculture tetrazolium assay | Q34661407 | ||
| Molecular mechanisms involved in tumor repopulation after radiotherapy | Q34948795 | ||
| Caspase 3-mediated stimulation of tumor cell repopulation during cancer radiotherapy | Q35093740 | ||
| Co-cultivation of murine BMDCs with 67NR mouse mammary carcinoma cells give rise to highly drug resistant cells | Q35102680 | ||
| Strategies for tracking anastasis, a cell survival phenomenon that reverses apoptosis | Q35162256 | ||
| The reverse evolution from multicellularity to unicellularity during carcinogenesis | Q35573684 | ||
| Tumorigenic hybrids between mesenchymal stem cells and gastric cancer cells enhanced cancer proliferation, migration and stemness | Q35820438 | ||
| A Fraction of CD133+ CNE2 Cells Is Made of Giant Cancer Cells with Morphological Evidence of Asymmetric Mitosis | Q35831564 | ||
| Mutation as a stress response and the regulation of evolvability | Q35869358 | ||
| Fusion with stem cell makes the hepatocellular carcinoma cells similar to liver tumor-initiating cells. | Q35914875 | ||
| Cell survival, DNA damage, and oncogenic transformation after a transient and reversible apoptotic response | Q36030459 | ||
| Spontaneous formation of tumorigenic hybrids between breast cancer and multipotent stromal cells is a source of tumor heterogeneity | Q36044675 | ||
| Neosis--a paradigm of self-renewal in cancer | Q36326465 | ||
| Crystal Violet Assay for Determining Viability of Cultured Cells. | Q54340969 | ||
| A switch from high-fidelity to error-prone DNA double-strand break repair underlies stress-induced mutation. | Q54478818 | ||
| Role of mutator alleles in adaptive evolution. | Q54564129 | ||
| Evolution of oncogenic signatures of mutation hotspots in tyrosine kinases supports the atavistic hypothesis of cancer. | Q54959144 | ||
| Roles of Polyploid/Multinucleated Giant Cancer Cells in Metastasis and Disease Relapse Following Anticancer Treatment. | Q55057212 | ||
| Novel roles of apoptotic caspases in tumor repopulation, epigenetic reprogramming, carcinogenesis, and beyond | Q57162853 | ||
| Senescence and senotherapeutics: a new field in cancer therapy | Q57169113 | ||
| Unconventional Ways to Live and Die: Cell Death and Survival in Development, Homeostasis, and Disease | Q57183216 | ||
| Cell fusion potentiates tumor heterogeneity and reveals circulating hybrid cells that correlate with stage and survival | Q57214280 | ||
| Dormant, quiescent, tolerant and persister cells: four synonyms for the same target in cancer | Q58575308 | ||
| Anastasis: recovery from the brink of cell death | Q58715274 | ||
| Viability Assessment Following Anticancer Treatment Requires Single-Cell Visualization | Q58805130 | ||
| The 150 most important questions in cancer research and clinical oncology series: questions 94-101 : Edited by Cancer Communications | Q59808518 | ||
| Global cancer statistics 2018: GLOBOCAN estimates of incidence and mortality worldwide for 36 cancers in 185 countries | Q60142201 | ||
| The Fate of Fusions | Q61811592 | ||
| HER2-Targeted Tyrosine Kinase Inhibitors Cause Therapy-Induced-Senescence in Breast Cancer Cells | Q64243102 | ||
| Leukocyte⁻Cancer Cell Fusion-Genesis of a Deadly Journey | Q64245343 | ||
| Stress-Induced Mutagenesis: Implications in Cancer and Drug Resistance | Q64389723 | ||
| The dualistic origin of human tumors. | Q64914786 | ||
| Multinucleated cells can continuously generate mononucleated cells in the absence of mitosis: a study of cells of the avian osteoclast lineage | Q71825230 | ||
| Caspase-3 activity as a prognostic factor in colorectal carcinoma | Q73877869 | ||
| A senescence-like phenotype distinguishes tumor cells that undergo terminal proliferation arrest after exposure to anticancer agents | Q78125095 | ||
| Induction of senescence with doxorubicin leads to increased genomic instability of HCT116 cells | Q81419187 | ||
| Cell fusion promotes chemoresistance in metastatic colon carcinoma | Q84507421 | ||
| HepG2 cells recovered from apoptosis show altered drug responses and invasiveness | Q88059987 | ||
| Two-Step Senescence-Focused Cancer Therapies | Q88731404 | ||
| Analysis of Cell Viability by the MTT Assay | Q88954879 | ||
| Polyploid Giant Cancer Cells (PGCCs): The Evil Roots of Cancer | Q89399158 | ||
| To the edge of cell death and back | Q90152760 | ||
| A Unique Morphological Phenotype in Chemoresistant Triple-Negative Breast Cancer Reveals Metabolic Reprogramming and PLIN4 Expression as a Molecular Vulnerability | Q90182242 | ||
| Formation of Polyploid Giant Cancer Cells Involves in the Prognostic Value of Neoadjuvant Chemoradiation in Locally Advanced Rectal Cancer | Q90318544 | ||
| The Systemic-Evolutionary Theory of the Origin of Cancer (SETOC): A New Interpretative Model of Cancer as a Complex Biological System | Q90461191 | ||
| Targeting Cancer Cell Dormancy | Q90908025 | ||
| aCLS cancers: Genomic and epigenetic changes transform the cell of origin of cancer into a tumorigenic pathogen of unicellular organization and lifestyle | Q90923626 | ||
| Adaptive mutability of colorectal cancers in response to targeted therapies | Q91172844 | ||
| Flow Cytometric Detection of Newly-formed Breast Cancer Stem Cell-like Cells After Apoptosis Reversal | Q91447283 | ||
| Diversity of the Senescence Phenotype of Cancer Cells Treated with Chemotherapeutic Agents | Q91468929 | ||
| Syncytin 1, CD9, and CD47 regulating cell fusion to form PGCCs associated with cAMP/PKA and JNK signaling pathway | Q91536060 | ||
| The role of heterogeneous environment and docetaxel gradient in the emergence of polyploid, mesenchymal and resistant prostate cancer cells | Q91960249 | ||
| Polyploid giant cancer cells: Unrecognized actuators of tumorigenesis, metastasis, and resistance | Q92380631 | ||
| Tumor Microenvironment and Cell Fusion | Q92409943 | ||
| Molecular profiling of anastatic cancer cells: potential role of the nuclear export pathway | Q92421775 | ||
| Apoptotic cell-derived exosomes: messages from dying cells | Q92503806 | ||
| Cancer cell fusion: a potential target to tackle drug-resistant and metastatic cancer cells | Q92517244 | ||
| Patterns of Early p21 Dynamics Determine Proliferation-Senescence Cell Fate after Chemotherapy | Q92781059 | ||
| Nonlinearities in the cellular response to ionizing radiation and the role of p53 therein | Q92966595 | ||
| Molecular mechanisms of biogenesis of apoptotic exosome-like vesicles and their roles as damage-associated molecular patterns | Q93335813 | ||
| P275 | copyright license | Creative Commons Attribution | Q6905323 |
| P6216 | copyright status | copyrighted | Q50423863 |
| P433 | issue | 4 | |
| P921 | main subject | neoplasm | Q1216998 |
| genetic heterogeneity | Q3733697 | ||
| P577 | publication date | 2020-02-15 | |
| P1433 | published in | International Journal of Molecular Sciences | Q3153277 |
| P1476 | title | Intratumor Heterogeneity and Therapy Resistance: Contributions of Dormancy, Apoptosis Reversal (Anastasis) and Cell Fusion to Disease Recurrence | |
| P478 | volume | 21 |
Search more.