scholarly article | Q13442814 |
P356 | DOI | 10.1093/EMBOJ/16.11.3303 |
P953 | full work available at URL | http://emboj.embopress.org/content/16/11/3303.full.pdf |
https://europepmc.org/articles/PMC1169946 | ||
https://europepmc.org/articles/PMC1169946?pdf=render | ||
https://doi.org/10.1093/emboj/16.11.3303 | ||
https://api.wiley.com/onlinelibrary/tdm/v1/articles/10.1093%2Femboj%2F16.11.3303 | ||
https://onlinelibrary.wiley.com/doi/full/10.1093/emboj/16.11.3303 | ||
P932 | PMC publication ID | 1169946 |
P698 | PubMed publication ID | 9214645 |
P5875 | ResearchGate publication ID | 14004147 |
P50 | author | Susan M Rosenberg | Q89466623 |
P2093 | author name string | R. S. Harris | |
C. Thulin | |||
J. Nagendran | |||
J. Torkelson | |||
M. J. Lombardo | |||
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P433 | issue | 11 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | evolution | Q1063 |
mutagenesis | Q149299 | ||
genetic recombination | Q211675 | ||
bacterial genome | Q4839988 | ||
P304 | page(s) | 3303-3311 | |
P577 | publication date | 1997-06-01 | |
1997-06-02 | |||
P1433 | published in | The EMBO Journal | Q1278554 |
P1476 | title | Genome-wide hypermutation in a subpopulation of stationary-phase cells underlies recombination-dependent adaptive mutation | |
P478 | volume | 16 |
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Q34599104 | Amplification of lac cannot account for adaptive mutation to Lac+ in Escherichia coli. |
Q34012598 | Amplification-mutagenesis: evidence that "directed" adaptive mutation and general hypermutability result from growth with a selected gene amplification |
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Q36499774 | Bacterial stationary-state mutagenesis and Mammalian tumorigenesis as stress-induced cellular adaptations and the role of epigenetics |
Q39847729 | Carbon starvation of Salmonella typhimurium does not cause a general increase of mutation rates |
Q35760045 | Catastrophe and what to do about it if you are a bacterium: the importance of frameshift mutants |
Q35013064 | Cell-selfish modes of evolution and mutations directed after transcriptional bypass |
Q73197734 | Characterization of an OprF-deficient mutant suggests that OprF is an essential protein for Pseudomonas fluorescens strain MF0 |
Q33943859 | Clusters of mutations from transient hypermutability |
Q33594339 | Competition of Escherichia coli DNA polymerases I, II and III with DNA Pol IV in stressed cells |
Q38446575 | Competitive growth advantage of nontoxigenic mutants in the stationary phase in archival cultures of pathogenic Vibrio cholerae strains |
Q28658067 | Constraint and opportunity in genome innovation |
Q33692357 | Contingency loci, mutator alleles, and their interactions. Synergistic strategies for microbial evolution and adaptation in pathogenesis |
Q55162233 | Death and population dynamics affect mutation rate estimates and evolvability under stress in bacteria. |
Q50130080 | Detection of mutator subpopulations in Salmonella typhimurium LT2 by reversion of his alleles |
Q39680847 | Different spectra of stationary-phase mutations in early-arising versus late-arising mutants of Pseudomonas putida: involvement of the DNA repair enzyme MutY and the stationary-phase sigma factor RpoS. |
Q37173424 | DinB upregulation is the sole role of the SOS response in stress-induced mutagenesis in Escherichia coli |
Q39477342 | Effect of drug concentration on emergence of macrolide resistance in Mycobacterium avium |
Q28346858 | Effect of endogenous carotenoids on "adaptive" mutation in Escherichia coli FC40 |
Q34195488 | Effect of growth under selection on appearance of chromosomal mutations in Salmonella enterica |
Q42719517 | Effect of translesion DNA polymerases, endonucleases and RpoS on mutation rates in Salmonella typhimurium |
Q36788747 | Empirical laws of survival and evolution: their universality and implications |
Q35028595 | Engineered proteins detect spontaneous DNA breakage in human and bacterial cells |
Q41028053 | Environmental change drives accelerated adaptation through stimulated copy number variation |
Q35058954 | Environmental regulation of mutation rates at specific sites. |
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Q33941534 | Environmentally directed mutations and their impact on industrial biotransformation and fermentation processes |
Q35582961 | Error-Prone DNA Polymerases: When Making a Mistake is the Only Way to Get Ahead |
Q39753960 | Error-prone polymerase, DNA polymerase IV, is responsible for transient hypermutation during adaptive mutation in Escherichia coli. |
Q34049897 | Error‐prone DNA polymerase IV is controlled by the stress‐response sigma factor, RpoS, in Escherichia coli |
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Q34643555 | General stress response regulator RpoS in adaptive mutation and amplification in Escherichia coli |
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Q34603856 | Hypermutability in carcinogenesis |
Q37922538 | Hypermutation and stress adaptation in bacteria |
Q77681420 | Hypermutation as a means to globally re-stabilize the genome following environmental stress |
Q33770256 | Hypermutation in bacteria and other cellular systems |
Q28776505 | Hypermutation in derepressed operons of Escherichia coli K12 |
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Q35037445 | In pursuit of a molecular mechanism for adaptive gene amplification |
Q34606847 | Increased episomal replication accounts for the high rate of adaptive mutation in recD mutants of Escherichia coli |
Q39694895 | Induction of a DNA nickase in the presence of its target site stimulates adaptive mutation in Escherichia coli. |
Q34102811 | Interactions among strategies associated with bacterial infection: pathogenicity, epidemicity, and antibiotic resistance |
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Q35990867 | Ionizing radiation and restriction enzymes induce microhomology-mediated illegitimate recombination in Saccharomyces cerevisiae |
Q24651873 | Is evolution Darwinian or/and Lamarckian? |
Q33783262 | Isolation, propagation and characterisation of Cryptosporidium. |
Q34569161 | Isothermal amplification and molecular typing of the obligate intracellular pathogen Mycobacterium leprae isolated from tissues of unknown origins |
Q33728224 | Levels of the Vsr endonuclease do not regulate stationary-phase reversion of a Lac- frameshift allele in Escherichia coli. |
Q36369839 | Long-term survival during stationary phase: evolution and the GASP phenotype |
Q33692333 | Mechanisms of genome-wide hypermutation in stationary phase |
Q33692291 | Mechanisms of mutation in nondividing cells. Insights from the study of adaptive mutation in Escherichia coli |
Q33847662 | Mechanisms of stationary phase mutation: a decade of adaptive mutation |
Q42771933 | Microbiology. Antibiotic resistance, not shaken or stirred |
Q35028138 | Minisatellite alterations in ZRT1 mutants occur via RAD52-dependent and RAD52-independent mechanisms in quiescent stationary phase yeast cells |
Q33724726 | Mismatch repair in Escherichia coli cells lacking single-strand exonucleases ExoI, ExoVII, and RecJ |
Q35190848 | Mismatch repair protein MutL becomes limiting during stationary-phase mutation |
Q24628966 | Multiple pathways for SOS-induced mutagenesis in Escherichia coli: an overexpression of dinB/dinP results in strongly enhancing mutagenesis in the absence of any exogenous treatment to damage DNA |
Q35125285 | Multiple pathways of selected gene amplification during adaptive mutation. |
Q33373675 | Mutability and importance of a hypermutable cell subpopulation that produces stress-induced mutants in Escherichia coli |
Q41749616 | Mutation and cancer: the antecedents to our studies of adaptive mutation. |
Q35869358 | Mutation as a stress response and the regulation of evolvability |
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Q34471714 | Plasmid copy number underlies adaptive mutability in bacteria |
Q73255932 | Prokaryote and eukaryote evolvability |
Q33282780 | Protecting exons from deleterious R-loops: a potential advantage of having introns |
Q24548000 | Rates of spontaneous mutation |
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Q64389096 | Recombination-based mechanisms for somatic hypermutation |
Q34617476 | Regulating general mutation rates: examination of the hypermutable state model for Cairnsian adaptive mutation. |
Q42324867 | Role of Ribonucleotide Reductase in Bacillus subtilis Stress-Associated Mutagenesis |
Q34470485 | Roles of E. coli double-strand-break-repair proteins in stress-induced mutation |
Q36365259 | Roles of Nucleoid-Associated Proteins in Stress-Induced Mutagenic Break Repair in Starving Escherichia coli |
Q33953638 | SOS mutator DNA polymerase IV functions in adaptive mutation and not adaptive amplification |
Q50186955 | Selection-Enhanced Mutagenesis of lac Genes Is Due to Their Co-amplification with dinB Encoding an Error-Prone DNA Polymerase |
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Q34514283 | Single-strand-specific exonucleases prevent frameshift mutagenesis by suppressing SOS induction and the action of DinB/DNA polymerase IV in growing cells |
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Q61552520 | Starvation-associated mutagenesis in yeast Saccharomyces cerevisiae is affected by Ras2/cAMP signaling pathway |
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Q35544150 | Stationary phase mutagenesis: mechanisms that accelerate adaptation of microbial populations under environmental stress |
Q37096423 | Stationary-phase mutation in the bacterial chromosome: recombination protein and DNA polymerase IV dependence |
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Q40459862 | Stress-Induced Mutagenesis. |
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Q36961683 | Stress-induced mutagenesis in bacteria. |
Q38541782 | The Origin of Mutants Under Selection: How Natural Selection Mimics Mutagenesis (Adaptive Mutation) |
Q33903483 | The SOS response regulates adaptive mutation |
Q39326888 | The Small RNA GcvB Promotes Mutagenic Break Repair by Opposing the Membrane Stress Response |
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