scholarly article | Q13442814 |
P50 | author | Rodrigo S Galhardo | Q42557808 |
P2093 | author name string | Joseph F Petrosino | |
Susan M Rosenberg | |||
Liza D Morales | |||
P2860 | cites work | Inhibition of mutation and combating the evolution of antibiotic resistance | Q21146100 |
Construction of Escherichia coli K-12 in-frame, single-gene knockout mutants: the Keio collection | Q22122301 | ||
SOS-induced DNA polymerases enhance long-term survival and evolutionary fitness | Q24530758 | ||
Multiple pathways for SOS-induced mutagenesis in Escherichia coli: an overexpression of dinB/dinP results in strongly enhancing mutagenesis in the absence of any exogenous treatment to damage DNA | Q24628966 | ||
beta-Lactamases in laboratory and clinical resistance | Q24669605 | ||
Adaptive amplification and point mutation are independent mechanisms: evidence for various stress-inducible mutation mechanisms | Q24796250 | ||
One-step inactivation of chromosomal genes in Escherichia coli K-12 using PCR products | Q27860842 | ||
Signal transduction and regulatory mechanisms involved in control of the sigma(S) (RpoS) subunit of RNA polymerase | Q28220384 | ||
Comparative gene expression profiles following UV exposure in wild-type and SOS-deficient Escherichia coli | Q28364148 | ||
cAMP-dependent SOS induction and mutagenesis in resting bacterial populations. | Q33719467 | ||
Genome-wide hypermutation in a subpopulation of stationary-phase cells underlies recombination-dependent adaptive mutation | Q33886793 | ||
The SOS response regulates adaptive mutation | Q33903483 | ||
Mutation frequencies and antibiotic resistance | Q33945503 | ||
SOS mutator DNA polymerase IV functions in adaptive mutation and not adaptive amplification | Q33953638 | ||
Adaptive reversion of a frameshift mutation in Escherichia coli | Q33958142 | ||
Two enzymes, both of which process recombination intermediates, have opposite effects on adaptive mutation in Escherichia coli. | Q33966542 | ||
Opposing roles of the holliday junction processing systems of Escherichia coli in recombination-dependent adaptive mutation | Q33966750 | ||
Escherichia coli DNA polymerase IV mutator activity: genetic requirements and mutational specificity | Q33994529 | ||
Proofreading-defective DNA polymerase II increases adaptive mutation in Escherichia coli. | Q34019746 | ||
Error‐prone DNA polymerase IV is controlled by the stress‐response sigma factor, RpoS, in Escherichia coli | Q34049897 | ||
Ultraviolet mutagenesis and inducible DNA repair in Escherichia coli | Q34072546 | ||
Chromosomal system for studying AmpC-mediated beta-lactam resistance mutation in Escherichia coli | Q34107615 | ||
The simple sequence contingency loci of Haemophilus influenzae and Neisseria meningitidis | Q34187179 | ||
A set of lacZ mutations in Escherichia coli that allow rapid detection of each of the six base substitutions | Q34290231 | ||
AmpD, essential for both beta-lactamase regulation and cell wall recycling, is a novel cytosolic N-acetylmuramyl-L-alanine amidase | Q34313267 | ||
Adaptive, or stationary-phase, mutagenesis, a component of bacterial differentiation in Bacillus subtilis | Q34436201 | ||
High frequency of hypermutable Pseudomonas aeruginosa in cystic fibrosis lung infection. | Q34508854 | ||
Fidelity of Escherichia coli DNA polymerase IV. Preferential generation of small deletion mutations by dNTP-stabilized misalignment | Q44049947 | ||
Stress-induced mutagenesis in bacteria | Q44459277 | ||
General stress response master regulator rpoS is expressed in human infection: a possible role in chronicity | Q44676175 | ||
High mutation frequencies among Escherichia coli and Salmonella pathogens | Q48057867 | ||
To be a mutator, or how pathogenic and commensal bacteria can evolve rapidly | Q50132690 | ||
Emergence of multidrug-resistant mutants is increased under antibiotic selective pressure in Pseudomonas aeruginosa. | Q54073909 | ||
Quinolone action in Escherichia coli cells carrying gyrA and gyrB mutations. | Q54249642 | ||
A switch from high-fidelity to error-prone DNA double-strand break repair underlies stress-induced mutation. | Q54478818 | ||
Genetic analysis and molecular cloning of the Escherichia coli ruv gene. | Q54484834 | ||
Cytosolic intermediates for cell wall biosynthesis and degradation control inducible beta-lactam resistance in gram-negative bacteria. | Q54568935 | ||
Adaptive mutation by deletions in small mononucleotide repeats. | Q54630365 | ||
Recombination in adaptive mutation. | Q54635736 | ||
Construction of a umuDC operon substitution mutation in Escherichia coli | Q54682895 | ||
Highly Variable Mutation Rates in Commensal and Pathogenic Escherichia coli | Q56944671 | ||
Purification and mutant analysis of Citrobacter freundii AmpR, the regulator for chromosomal AmpC beta-lactamase | Q68047757 | ||
Plasmid pKM101-mediated mutagenesis in Escherichia coli is inducible | Q70256183 | ||
Specificity of mutagenesis resulting from the induction of the SOS system in the absence of mutagenic treatment | Q70352825 | ||
Influence of RecA protein on induced mutagenesis | Q71627364 | ||
Mechanisms of antimicrobial resistance in bacteria | Q79823685 | ||
Study of involvement of ImuB and DnaE2 in stationary-phase mutagenesis in Pseudomonas putida | Q79854682 | ||
Transient and heritable mutators in adaptive evolution in the lab and in nature | Q34603730 | ||
General stress response regulator RpoS in adaptive mutation and amplification in Escherichia coli | Q34643555 | ||
Roles of YqjH and YqjW, homologs of the Escherichia coli UmuC/DinB or Y superfamily of DNA polymerases, in stationary-phase mutagenesis and UV-induced mutagenesis of Bacillus subtilis | Q34810207 | ||
Role of DNA polymerase IV in Escherichia coli SOS mutator activity | Q35130259 | ||
beta-Lactam resistance in gram-negative bacteria: global trends and clinical impact | Q35535598 | ||
UmuD'(2)C is an error-prone DNA polymerase, Escherichia coli pol V. | Q35588920 | ||
Nonadaptive mutations occur on the F' episome during adaptive mutation conditions in Escherichia coli | Q35620439 | ||
Mutation as a stress response and the regulation of evolvability | Q35869358 | ||
Trading places: how do DNA polymerases switch during translesion DNA synthesis? | Q36139683 | ||
Overlap between ampC and frd operons on the Escherichia coli chromosome | Q36282061 | ||
Cleavage of the Escherichia coli lexA protein by the recA protease | Q36392308 | ||
Environmental stress and lesion-bypass DNA polymerases | Q36486062 | ||
Dominant mutations (lex) in Escherichia coli K-12 which affect radiation sensitivity and frequency of ultraviolet lght-induced mutations | Q36834871 | ||
Rescue of stalled replication forks by RecG: simultaneous translocation on the leading and lagging strand templates supports an active DNA unwinding model of fork reversal and Holliday junction formation | Q37092701 | ||
Stationary-phase mutation in the bacterial chromosome: recombination protein and DNA polymerase IV dependence | Q37096423 | ||
DinB upregulation is the sole role of the SOS response in stress-induced mutagenesis in Escherichia coli | Q37173424 | ||
AmpC beta-lactamases | Q37366249 | ||
Bacterial cell wall recycling provides cytosolic muropeptides as effectors for beta-lactamase induction | Q37636934 | ||
H-NS and RpoS regulate emergence of Lac Ara+ mutants of Escherichia coli MCS2. | Q38344584 | ||
Repair of DNA damage induced by bile salts in Salmonella enterica | Q38583344 | ||
Inactivation of the ampD gene in Pseudomonas aeruginosa leads to moderate-basal-level and hyperinducible AmpC beta-lactamase expression | Q39473061 | ||
Involvement of sigma(S) in starvation-induced transposition of Pseudomonas putida transposon Tn4652. | Q39504849 | ||
Genetic antagonism and hypermutability in Mycobacterium smegmatis | Q39538622 | ||
Adaptive mutations produce resistance to ciprofloxacin. | Q39784978 | ||
Molecular genetic analysis of cephalosporinase production and its role in beta-lactam resistance in clinical isolates of Enterobacter cloacae | Q39827699 | ||
Genetic analysis of the recG locus of Escherichia coli K-12 and of its role in recombination and DNA repair | Q39938960 | ||
Inactivation of the ampD gene causes semiconstitutive overproduction of the inducible Citrobacter freundii beta-lactamase | Q39956594 | ||
Involvement of error-prone DNA polymerase IV in stationary-phase mutagenesis in Pseudomonas putida | Q40763644 | ||
Isolation and characterization of mutants of Escherichia coli deficient in induction of mutations by ultraviolet light | Q40850491 | ||
SOS-independent induction of dinB transcription by beta-lactam-mediated inhibition of cell wall synthesis in Escherichia coli. | Q40943537 | ||
Adaptive reversion of a frameshift mutation in Escherichia coli by simple base deletions in homopolymeric runs | Q41572901 | ||
Signalling proteins in enterobacterial AmpC beta-lactamase regulation | Q42497655 | ||
P433 | issue | 19 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | Escherichia coli | Q25419 |
antibiotic resistance | Q380775 | ||
P304 | page(s) | 5881-5889 | |
P577 | publication date | 2009-07-31 | |
P1433 | published in | Journal of Bacteriology | Q478419 |
P1476 | title | Stress-induced beta-lactam antibiotic resistance mutation and sequences of stationary-phase mutations in the Escherichia coli chromosome | |
P478 | volume | 191 |
Q28547796 | A Genetic Selection for dinB Mutants Reveals an Interaction between DNA Polymerase IV and the Replicative Polymerase That Is Required for Translesion Synthesis |
Q27677020 | A strategically located serine residue is critical for the mutator activity of DNA polymerase IV from Escherichia coli |
Q36667392 | Antibiotic resistance acquired through a DNA damage-inducible response in Acinetobacter baumannii |
Q27008535 | Bacterial Responses and Genome Instability Induced by Subinhibitory Concentrations of Antibiotics |
Q38012391 | Bacterial stress responses as determinants of antimicrobial resistance |
Q33594339 | Competition of Escherichia coli DNA polymerases I, II and III with DNA Pol IV in stressed cells |
Q36878608 | Competitive fitness during feast and famine: how SOS DNA polymerases influence physiology and evolution in Escherichia coli |
Q37396897 | DNA polymerases are error-prone at RecA-mediated recombination intermediates. |
Q90365037 | Development of amoxicillin resistance in Escherichia coli after exposure to remnants of a non-related phagemid-containing E. coli: an exploratory study |
Q37922538 | Hypermutation and stress adaptation in bacteria |
Q42969505 | Identity and function of a large gene network underlying mutagenic repair of DNA breaks |
Q35170976 | Impact of a stress-inducible switch to mutagenic repair of DNA breaks on mutation in Escherichia coli |
Q34378318 | Implications of stress-induced genetic variation for minimizing multidrug resistance in bacteria |
Q38114534 | Microbial persistence and the road to drug resistance |
Q37512348 | Multiple strategies for translesion synthesis in bacteria |
Q40559189 | Natural selection underlies apparent stress-induced mutagenesis in a bacteriophage infection model. |
Q35858492 | Noncognate DNA damage prevents the formation of the active conformation of the Y-family DNA polymerases DinB and DNA polymerase κ |
Q58997159 | Opposing effects of final population density and stress on Escherichia coli mutation rate |
Q38670044 | Persistent damaged bases in DNA allow mutagenic break repair in Escherichia coli |
Q36936978 | Preferential D-loop extension by a translesion DNA polymerase underlies error-prone recombination |
Q28551034 | Quantifying the Determinants of Evolutionary Dynamics Leading to Drug Resistance |
Q51136979 | Rapid evolution of acetic acid-detoxifying Escherichia coli under phosphate starvation conditions requires activation of the cryptic PhnE permease and induction of translesion synthesis DNA polymerases. |
Q36365259 | Roles of Nucleoid-Associated Proteins in Stress-Induced Mutagenic Break Repair in Starving Escherichia coli |
Q21144990 | SOS response induces persistence to fluoroquinolones in Escherichia coli |
Q57125581 | Seasonal Changes in Antibiotic Resistance Genes in Rivers and Reservoirs in South Korea |
Q34056695 | Selection of dinB alleles suppressing survival loss upon dinB overexpression in Escherichia coli |
Q34119281 | Separate DNA Pol II- and Pol IV-dependent pathways of stress-induced mutation during double-strand-break repair in Escherichia coli are controlled by RpoS. |
Q34539902 | Starvation, together with the SOS response, mediates high biofilm-specific tolerance to the fluoroquinolone ofloxacin. |
Q40459862 | Stress-Induced Mutagenesis. |
Q64389723 | Stress-Induced Mutagenesis: Implications in Cancer and Drug Resistance |
Q38507433 | Stress-induced loss of heterozygosity in Candida: a possible missing link in the ability to evolve |
Q36496909 | Stress-induced mutation via DNA breaks in Escherichia coli: a molecular mechanism with implications for evolution and medicine |
Q43633453 | Suramin is a potent and selective inhibitor of Mycobacterium tuberculosis RecA protein and the SOS response: RecA as a potential target for antibacterial drug discovery. |
Q90493507 | The Magnitude of Candida albicans Stress-Induced Genome Instability Results from an Interaction Between Ploidy and Antifungal Drugs |
Q39326888 | The Small RNA GcvB Promotes Mutagenic Break Repair by Opposing the Membrane Stress Response |
Q41050631 | The Transient Multidrug Resistance Phenotype of Salmonella enterica Swarming Cells Is Abolished by Sub-inhibitory Concentrations of Antimicrobial Compounds. |
Q34017416 | The sigma(E) stress response is required for stress-induced mutation and amplification in Escherichia coli |
Q41445233 | Transcriptional de-repression and Mfd are mutagenic in stressed Bacillus subtilis cells |
Q34509062 | Translesion DNA Synthesis |
Q36713351 | Two mechanisms produce mutation hotspots at DNA breaks in Escherichia coli |
Q64096919 | What is mutation? A chapter in the series: How microbes "jeopardize" the modern synthesis |
Q37976150 | What limits the efficiency of double-strand break-dependent stress-induced mutation in Escherichia coli? |
Q36737307 | β-Lactam antibiotics promote bacterial mutagenesis via an RpoS-mediated reduction in replication fidelity |
Search more.