| scholarly article | Q13442814 |
| P2093 | author name string | Susan M Rosenberg | |
| Chandan Shee | |||
| Janet L Gibson | |||
| P2860 | cites work | Environmental stress and lesion-bypass DNA polymerases | Q36486062 |
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| Mutations in clusters and showers | Q35850075 | ||
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| Clustered mutations in yeast and in human cancers can arise from damaged long single-strand DNA regions. | Q35992079 | ||
| Homologous recombination and its regulation | Q36106937 | ||
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| P433 | issue | 4 | |
| P921 | main subject | Escherichia coli | Q25419 |
| P304 | page(s) | 714-721 | |
| P577 | publication date | 2012-10-04 | |
| P1433 | published in | Cell Reports | Q5058165 |
| P1476 | title | Two mechanisms produce mutation hotspots at DNA breaks in Escherichia coli | |
| P478 | volume | 2 |
| Q34590485 | A formal perturbation equation between genotype and phenotype determines the Evolutionary Action of protein-coding variations on fitness |
| Q35189852 | An underlying mechanism for the increased mutagenesis of lagging-strand genes in Bacillus subtilis |
| Q33602374 | Ancient genes establish stress-induced mutation as a hallmark of cancer |
| Q28547150 | Atypical Role for PhoU in Mutagenic Break Repair under Stress in Escherichia coli |
| Q35796604 | Cationic Peptides Facilitate Iron-induced Mutagenesis in Bacteria |
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| Q52576549 | Discovery of Lineage-Specific Genome Change in Rice Through Analysis of Resequencing Data. |
| Q35028595 | Engineered proteins detect spontaneous DNA breakage in human and bacterial cells |
| Q37138672 | Enhancement of microhomology-mediated genomic rearrangements by transient loss of mouse Bloom syndrome helicase |
| Q38108358 | Evolutionary dynamics and information hierarchies in biological systems |
| Q64388893 | Fluorescent fusions of the N protein of phage Mu label DNA damage in living cells |
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| Q58595432 | NGS sequencing reveals that many of the genetic variations in transgenic rice plants match the variations found in natural rice population |
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| Q39326888 | The Small RNA GcvB Promotes Mutagenic Break Repair by Opposing the Membrane Stress Response |
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