scholarly article | Q13442814 |
P50 | author | Lilach Hadany | Q90025218 |
P2093 | author name string | P J Hastings | |
Susan M Rosenberg | |||
Caleb Gonzalez | |||
Mellanie Price | |||
Rebecca G Ponder | |||
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Genome-wide hypermutation in a subpopulation of stationary-phase cells underlies recombination-dependent adaptive mutation | Q33886793 | ||
The SOS response regulates adaptive mutation | Q33903483 | ||
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SOS mutator DNA polymerase IV functions in adaptive mutation and not adaptive amplification | Q33953638 | ||
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A ruby in the rubbish: beneficial mutations, deleterious mutations and the evolution of sex | Q33963090 | ||
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Two enzymes, both of which process recombination intermediates, have opposite effects on adaptive mutation in Escherichia coli. | Q33966542 | ||
Opposing roles of the holliday junction processing systems of Escherichia coli in recombination-dependent adaptive mutation | Q33966750 | ||
Error‐prone DNA polymerase IV is controlled by the stress‐response sigma factor, RpoS, in Escherichia coli | Q34049897 | ||
Down-regulation of Rad51 and decreased homologous recombination in hypoxic cancer cells | Q34348322 | ||
Adaptive, or stationary-phase, mutagenesis, a component of bacterial differentiation in Bacillus subtilis | Q34436201 | ||
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Transient and heritable mutators in adaptive evolution in the lab and in nature | Q34603730 | ||
Some features of the mutability of bacteria during nonlethal selection | Q34608581 | ||
Evidence that stationary-phase hypermutation in the Escherichia coli chromosome is promoted by recombination | Q34609122 | ||
Regulating general mutation rates: examination of the hypermutable state model for Cairnsian adaptive mutation. | Q34617476 | ||
General stress response regulator RpoS in adaptive mutation and amplification in Escherichia coli | Q34643555 | ||
Activation of a LTR-retrotransposon by telomere erosion | Q34791258 | ||
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Compounds Which Serve as the Sole Source of Carbon or Nitrogen for Salmonella typhimurium LT-2 | Q35157309 | ||
Survival versus maintenance of genetic stability: a conflict of priorities during stress | Q35811210 | ||
Evidence for mutation showers | Q35850189 | ||
Mutation as a stress response and the regulation of evolvability | Q35869358 | ||
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Environmental stress and lesion-bypass DNA polymerases | Q36486062 | ||
A genome-wide view of the spectrum of spontaneous mutations in yeast. | Q36756806 | ||
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H-NS and RpoS regulate emergence of Lac Ara+ mutants of Escherichia coli MCS2. | Q38344584 | ||
Repair of DNA damage induced by bile salts in Salmonella enterica | Q38583344 | ||
The SOS system | Q39496380 | ||
Involvement of sigma(S) in starvation-induced transposition of Pseudomonas putida transposon Tn4652. | Q39504849 | ||
Different spectra of stationary-phase mutations in early-arising versus late-arising mutants of Pseudomonas putida: involvement of the DNA repair enzyme MutY and the stationary-phase sigma factor RpoS. | Q39680847 | ||
Decreased Expression of the DNA Mismatch Repair Gene Mlh1 under Hypoxic Stress in Mammalian Cells | Q39745939 | ||
Non-homologous end joining as an important mutagenic process in cell cycle-arrested cells | Q39756406 | ||
Redundant transfer of F' plasmids occurs between Escherichia coli cells during nonlethal selections | Q39835578 | ||
Co-repression of mismatch repair gene expression by hypoxia in cancer cells: role of the Myc/Max network | Q40175307 | ||
Repression of RAD51 gene expression by E2F4/p130 complexes in hypoxia | Q40227628 | ||
Purification and characterization of the in vitro activity of I-Sce I, a novel and highly specific endonuclease encoded by a group I intron | Q40515080 | ||
Involvement of error-prone DNA polymerase IV in stationary-phase mutagenesis in Pseudomonas putida | Q40763644 | ||
Adaptive reversion of a frameshift mutation in Escherichia coli by simple base deletions in homopolymeric runs | Q41572901 | ||
Roles of E. coli DNA polymerases IV and V in lesion-targeted and untargeted SOS mutagenesis | Q41734679 | ||
Recombination-dependent mutation in Escherichia coli occurs in stationary phase | Q42573518 | ||
The TGV transgenic vectors for single-copy gene expression from the Escherichia coli chromosome | Q43694249 | ||
Stress-induced mutagenesis in bacteria | Q44459277 | ||
Lessons from 50 years of SOS DNA-damage-induced mutagenesis. | Q46014624 | ||
Hypoxia-induced down-regulation of BRCA1 expression by E2Fs. | Q47673689 | ||
Genetic instability: the dark side of the hypoxic response. | Q47776496 | ||
HIF-1alpha induces genetic instability by transcriptionally downregulating MutSalpha expression. | Q47829016 | ||
RecG helicase promotes DNA double-strand break repair | Q47864494 | ||
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P275 | copyright license | Creative Commons Attribution 4.0 International | Q20007257 |
P6216 | copyright status | copyrighted | Q50423863 |
P433 | issue | 10 | |
P921 | main subject | Escherichia coli | Q25419 |
P304 | page(s) | e1000208 | |
P577 | publication date | 2008-10-03 | |
P1433 | published in | PLOS Genetics | Q1893441 |
P1476 | title | Mutability and importance of a hypermutable cell subpopulation that produces stress-induced mutants in Escherichia coli | |
P478 | volume | 4 |
Q27677020 | A strategically located serine residue is critical for the mutator activity of DNA polymerase IV from Escherichia coli |
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Q34090613 | Clustered and genome-wide transient mutagenesis in human cancers: Hypermutation without permanent mutators or loss of fitness |
Q35002361 | Damage-induced localized hypermutability. |
Q37173424 | DinB upregulation is the sole role of the SOS response in stress-induced mutagenesis in Escherichia coli |
Q41855236 | Elevated mutagenesis does not explain the increased frequency of antibiotic resistant mutants in starved aging colonies |
Q86095467 | Evaluating evolutionary models of stress-induced mutagenesis in bacteria |
Q46078838 | Evaluation of the roles of Pol zeta and NHEJ in starvation-associated spontaneous mutagenesis in the yeast Saccharomyces cerevisiae |
Q64259606 | Evolution of Resistance in Cancer: A Cell Cycle Perspective |
Q34013646 | Global chromosomal structural instability in a subpopulation of starving Escherichia coli cells |
Q37922538 | Hypermutation and stress adaptation in bacteria |
Q95300387 | Hypermutation in single-stranded DNA |
Q33417637 | Life, death, differentiation, and the multicellularity of bacteria |
Q38053191 | Mechanisms and selection of evolvability: experimental evidence. |
Q34037238 | Mutational clusters generated by non-processive polymerases: A case study using DNA polymerase betain vitro |
Q37678002 | Mutators and hypermutability in bacteria: the Escherichia coli paradigm |
Q47558874 | Oxygen and RNA in stress-induced mutation. |
Q38820376 | Phenotypic heterogeneity in a bacteriophage population only appears as stress-induced mutagenesis. |
Q39005916 | Population Heterogeneity in Mutation Rate Increases the Frequency of Higher-Order Mutants and Reduces Long-Term Mutational Load. |
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Q42324867 | Role of Ribonucleotide Reductase in Bacillus subtilis Stress-Associated Mutagenesis |
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Q40459862 | Stress-Induced Mutagenesis. |
Q64389723 | Stress-Induced Mutagenesis: Implications in Cancer and Drug Resistance |
Q47655848 | Stress-induced cellular adaptive strategies: ancient evolutionarily conserved programs as new anticancer therapeutic targets. |
Q37976153 | Stress-induced modulators of repeat instability and genome evolution |
Q36496909 | Stress-induced mutation via DNA breaks in Escherichia coli: a molecular mechanism with implications for evolution and medicine |
Q42776660 | The TCA cycle is not required for selection or survival of multidrug-resistant Salmonella |
Q34017416 | The sigma(E) stress response is required for stress-induced mutation and amplification in Escherichia coli |
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