review article | Q7318358 |
scholarly article | Q13442814 |
P356 | DOI | 10.1080/10409230701597741 |
P8608 | Fatcat ID | release_5l5arlrimvccll3wpx7myyeoka |
P698 | PubMed publication ID | 17917871 |
P5875 | ResearchGate publication ID | 5926912 |
P2093 | author name string | Floyd E Romesberg | |
Ryan T Cirz | |||
P2860 | cites work | Inhibition of mutation and combating the evolution of antibiotic resistance | Q21146100 |
Induction of Mutations in a Bacterial Virus | Q24519350 | ||
SOS-induced DNA polymerases enhance long-term survival and evolutionary fitness | Q24530758 | ||
Evolvability is a selectable trait | Q24564104 | ||
All three SOS-inducible DNA polymerases (Pol II, Pol IV and Pol V) are involved in induced mutagenesis | Q24597093 | ||
Multiple pathways for SOS-induced mutagenesis in Escherichia coli: an overexpression of dinB/dinP results in strongly enhancing mutagenesis in the absence of any exogenous treatment to damage DNA | Q24628966 | ||
Adaptive amplification and point mutation are independent mechanisms: evidence for various stress-inducible mutation mechanisms | Q24796250 | ||
An SOS-regulated operon involved in damage-inducible mutagenesis in Caulobacter crescentus | Q24800880 | ||
Dispersal and regulation of an adaptive mutagenesis cassette in the bacteria domain | Q25257029 | ||
In silico analysis reveals substantial variability in the gene contents of the gamma proteobacteria LexA-regulon | Q28186056 | ||
Transcriptional Adaptation of Mycobacterium tuberculosis within Macrophages: Insights into the Phagosomal Environment | Q28487339 | ||
Genetic composition of the Bacillus subtilis SOS system | Q29346682 | ||
Role of Pseudomonas aeruginosa dinB-encoded DNA polymerase IV in mutagenesis | Q29346764 | ||
Defining the Pseudomonas aeruginosa SOS response and its role in the global response to the antibiotic ciprofloxacin | Q29346807 | ||
Involvement of multiple genetic loci in Staphylococcus aureus teicoplanin resistance. | Q31814182 | ||
Isolation and characterization of Staphylococcus aureus starvation-induced, stationary-phase mutants defective in survival or recovery | Q31958466 | ||
On the mechanism of gene amplification induced under stress in Escherichia coli | Q33239373 | ||
Mechanisms of mutation in nondividing cells. Insights from the study of adaptive mutation in Escherichia coli | Q33692291 | ||
Evolution of evolvability. | Q33692297 | ||
Mechanisms of genome-wide hypermutation in stationary phase | Q33692333 | ||
cAMP-dependent SOS induction and mutagenesis in resting bacterial populations. | Q33719467 | ||
Role of the dinB gene product in spontaneous mutation in Escherichia coli with an impaired replicative polymerase | Q33792439 | ||
The SOS response regulates adaptive mutation | Q33903483 | ||
SOS mutator DNA polymerase IV functions in adaptive mutation and not adaptive amplification | Q33953638 | ||
Teicoplanin stress-selected mutations increasing sigma(B) activity in Staphylococcus aureus | Q33982248 | ||
Amplification-mutagenesis: evidence that "directed" adaptive mutation and general hypermutability result from growth with a selected gene amplification | Q34012598 | ||
Competitive processivity-clamp usage by DNA polymerases during DNA replication and repair. | Q34053089 | ||
Changes in energy metabolism of Mycobacterium tuberculosis in mouse lung and under in vitro conditions affecting aerobic respiration | Q34081424 | ||
Error-prone repair DNA polymerases in prokaryotes and eukaryotes | Q34131455 | ||
An elevated mutation frequency favors development of vancomycin resistance in Staphylococcus aureus | Q34142416 | ||
hREV3 is essential for error-prone translesion synthesis past UV or benzo[a]pyrene diol epoxide-induced DNA lesions in human fibroblasts | Q34161894 | ||
DnaE2 polymerase contributes to in vivo survival and the emergence of drug resistance in Mycobacterium tuberculosis | Q34191816 | ||
Microbial Competition: Escherichia coli Mutants That Take Over Stationary Phase Cultures | Q34305902 | ||
Adaptive, or stationary-phase, mutagenesis, a component of bacterial differentiation in Bacillus subtilis | Q34436201 | ||
A molecular target for suppression of the evolution of antibiotic resistance: inhibition of the Escherichia coli RecA protein by N(6)-(1-naphthyl)-ADP. | Q34443803 | ||
Roles of E. coli double-strand-break-repair proteins in stress-induced mutation | Q34470485 | ||
High frequency of hypermutable Pseudomonas aeruginosa in cystic fibrosis lung infection. | Q34508854 | ||
Amplification of lac cannot account for adaptive mutation to Lac+ in Escherichia coli. | Q34599104 | ||
Specialized DNA polymerases, cellular survival, and the genesis of mutations | Q34662609 | ||
Involvement of Y-family DNA polymerases in mutagenesis caused by oxidized nucleotides in Escherichia coli | Q34697729 | ||
The prevalence and mechanisms of vancomycin resistance in Staphylococcus aureus. | Q34762753 | ||
Translesion DNA synthesis in eukaryotes: a one- or two-polymerase affair | Q34770150 | ||
Roles of YqjH and YqjW, homologs of the Escherichia coli UmuC/DinB or Y superfamily of DNA polymerases, in stationary-phase mutagenesis and UV-induced mutagenesis of Bacillus subtilis | Q34810207 | ||
Highly mutagenic replication by DNA polymerase V (UmuC) provides a mechanistic basis for SOS untargeted mutagenesis | Q34964296 | ||
Mismatch repair protein MutL becomes limiting during stationary-phase mutation | Q35190848 | ||
Contacts between DNA gyrase and its binding site on DNA: features of symmetry and asymmetry revealed by protection from nucleases | Q35319882 | ||
Natural selection and the emergence of a mutation phenotype: an update of the evolutionary synthesis considering mechanisms that affect genome variation | Q35550595 | ||
UmuD'(2)C is an error-prone DNA polymerase, Escherichia coli pol V. | Q35588920 | ||
Role of RecA protein in untargeted UV mutagenesis of bacteriophage lambda: evidence for the requirement for the dinB gene | Q35608675 | ||
Negative regulation of mutS and mutH repair gene expression by the Hfq and RpoS global regulators of Escherichia coli K-12. | Q35633546 | ||
Complete and SOS-mediated response of Staphylococcus aureus to the antibiotic ciprofloxacin | Q35634868 | ||
Activity of quinolones in the Ames Salmonella TA102 mutagenicity test and other bacterial genotoxicity assays | Q35808386 | ||
Adaptive mutation and amplification in Escherichia coli: two pathways of genome adaptation under stress | Q35811218 | ||
Ciprofloxacin-induced, low-level resistance to structurally unrelated antibiotics in Pseudomonas aeruginosa and methicillin-resistant Staphylococcus aureus | Q35816058 | ||
Adaptive mutation: how growth under selection stimulates Lac(+) reversion by increasing target copy number | Q35893213 | ||
On the nature of Mycobacterium tuberculosis-latent bacilli | Q35968192 | ||
Mutation rate and evolution of fluoroquinolone resistance in Escherichia coli isolates from patients with urinary tract infections. | Q36048167 | ||
Mutagenesis and stress responses induced in Escherichia coli by hydrogen peroxide | Q36239906 | ||
Hypermutable bacteria isolated from humans--a critical analysis | Q36583214 | ||
Adaptive mutation: General mutagenesis is not a programmed response to stress but results from rare coamplification of dinB with lac | Q36689838 | ||
Regulation of bacterial RecA protein function | Q36761497 | ||
Stationary-phase mutation in the bacterial chromosome: recombination protein and DNA polymerase IV dependence | Q37096423 | ||
PBP3 inhibition elicits adaptive responses in Pseudomonas aeruginosa. | Q38309969 | ||
Error-prone polymerase, DNA polymerase IV, is responsible for transient hypermutation during adaptive mutation in Escherichia coli. | Q39753960 | ||
Ultraviolet-induced mutation and DNA repair | Q39984442 | ||
Involvement of error-prone DNA polymerase IV in stationary-phase mutagenesis in Pseudomonas putida | Q40763644 | ||
Molecular mechanisms of induced mutagenesis. Replication in vivo of bacteriophage phiX174 single-stranded, ultraviolet light-irradiated DNA in intact and irradiated host cells | Q40849401 | ||
SOS-independent induction of dinB transcription by beta-lactam-mediated inhibition of cell wall synthesis in Escherichia coli. | Q40943537 | ||
The role of umuC gene product in mutagenesis by simple alkylating agents | Q41613925 | ||
Characterization of the global transcriptional responses to different types of DNA damage and disruption of replication in Bacillus subtilis | Q41960084 | ||
Quinolone resistance in Helicobacter pylori isolates in Germany | Q42112320 | ||
Rebuttal: growth under selection stimulates Lac(+) reversion (Roth and Andersson). | Q42793523 | ||
Ciprofloxacin and trimethoprim cause phage induction and virulence modulation in Staphylococcus aureus | Q43181776 | ||
Induction and inhibition of ciprofloxacin resistance-conferring mutations in hypermutator bacteria | Q43181792 | ||
beta-lactam antibiotics induce the SOS response and horizontal transfer of virulence factors in Staphylococcus aureus | Q43259790 | ||
Induction of SOS genes in Escherichia coli and mutagenesis in Salmonella typhimurium by fluoroquinolones | Q43680076 | ||
The role of the SOS response in bacteria exposed to zidovudine or trimethoprim | Q44084572 | ||
Genetics of mutagenesis in E. coli: various combinations of translesion polymerases (Pol II, IV and V) deal with lesion/sequence context diversity | Q44267628 | ||
Stress-induced mutagenesis in bacteria | Q44459277 | ||
SOS response induction by beta-lactams and bacterial defense against antibiotic lethality | Q45016731 | ||
Ultraviolet reactivation and ultraviolet mutagenesis of λ in different genetic systems | Q45245860 | ||
RecA acts in trans to allow replication of damaged DNA by DNA polymerase V. | Q46457518 | ||
Effect of subinhibitory concentrations of ciprofloxacin on Mycobacterium fortuitum mutation rates | Q46546957 | ||
The mechanism of inhibition of topoisomerase IV by quinolone antibacterials | Q46829877 | ||
Effect of subinhibitory concentrations of antibiotics on mutation frequency in Streptococcus pneumoniae | Q46986600 | ||
Investigating the role of the little finger domain of Y-family DNA polymerases in low fidelity synthesis and translesion replication. | Q47448190 | ||
Snapshots of replication through an abasic lesion; structural basis for base substitutions and frameshifts | Q47946960 | ||
Mechanism of quinolone mutagenicity in bacteria | Q50187884 | ||
[Natural selection]. | Q54359183 | ||
A sliding-clamp toolbelt binds high- and low-fidelity DNA polymerases simultaneously. | Q54478815 | ||
A switch from high-fidelity to error-prone DNA double-strand break repair underlies stress-induced mutation. | Q54478818 | ||
IncN plasmids mediate UV resistance and error-prone repair in Pseudomonas aeruginosa PAO. | Q54644118 | ||
Induction of the SOS gene (umuC) by 4-quinolone antibacterial drugs | Q54683533 | ||
The two-step model of bacterial UV mutagenesis. | Q54796394 | ||
P433 | issue | 5 | |
P304 | page(s) | 341-354 | |
P577 | publication date | 2007-09-01 | |
P1433 | published in | Critical Reviews in Biochemistry and Molecular Biology | Q5186661 |
P1476 | title | Controlling mutation: intervening in evolution as a therapeutic strategy | |
P478 | volume | 42 |
Q60949764 | Advancement of the 5-Amino-1-(Carbamoylmethyl)-1H-1,2,3-Triazole-4-Carboxamide Scaffold to Disarm the Bacterial SOS Response |
Q39448750 | An experimental evaluation of drug-induced mutational meltdown as an antiviral treatment strategy |
Q36667392 | Antibiotic resistance acquired through a DNA damage-inducible response in Acinetobacter baumannii |
Q28547150 | Atypical Role for PhoU in Mutagenic Break Repair under Stress in Escherichia coli |
Q91063368 | Bacterial persistence promotes the evolution of antibiotic resistance by increasing survival and mutation rates |
Q38012391 | Bacterial stress responses as determinants of antimicrobial resistance |
Q30235064 | Biology of Acinetobacter baumannii: Pathogenesis, Antibiotic Resistance Mechanisms, and Prospective Treatment Options |
Q48207182 | DNA polymerase IV primarily operates outside of DNA replication forks in Escherichia coli |
Q37173424 | DinB upregulation is the sole role of the SOS response in stress-induced mutagenesis in Escherichia coli |
Q37876027 | Ecology and evolution as targets: the need for novel eco-evo drugs and strategies to fight antibiotic resistance |
Q54372713 | Effect of recA inactivation on mutagenesis of Escherichia coli exposed to sublethal concentrations of antimicrobials. |
Q39797746 | Genome-wide stochastic adaptive DNA amplification at direct and inverted DNA repeats in the parasite Leishmania |
Q48350953 | Inhibitors of LexA Autoproteolysis and the Bacterial SOS Response Discovered by an Academic-Industry Partnership. |
Q30479751 | Inhibitors of RecA activity discovered by high-throughput screening: cell-permeable small molecules attenuate the SOS response in Escherichia coli |
Q38114534 | Microbial persistence and the road to drug resistance |
Q35864947 | MsDpo4-a DinB Homolog from Mycobacterium smegmatis-Is an Error-Prone DNA Polymerase That Can Promote G:T and T:G Mismatches |
Q37512348 | Multiple strategies for translesion synthesis in bacteria |
Q33373675 | Mutability and importance of a hypermutable cell subpopulation that produces stress-induced mutants in Escherichia coli |
Q35869358 | Mutation as a stress response and the regulation of evolvability |
Q33395486 | Mycobacterium tuberculosis interactome analysis unravels potential pathways to drug resistance |
Q36766892 | Programmed genetic instability: a tumor-permissive mechanism for maintaining the evolvability of higher species through methylation-dependent mutation of DNA repair genes in the male germ line |
Q43080975 | RNA-Mediated cis Regulation in Acinetobacter baumannii Modulates Stress-Induced Phenotypic Variation |
Q37568922 | Role of reactive oxygen species in antibiotic action and resistance. |
Q34119281 | Separate DNA Pol II- and Pol IV-dependent pathways of stress-induced mutation during double-strand-break repair in Escherichia coli are controlled by RpoS. |
Q28476758 | Separation of recombination and SOS response in Escherichia coli RecA suggests LexA interaction sites |
Q39867010 | Specificity determinants for autoproteolysis of LexA, a key regulator of bacterial SOS mutagenesis. |
Q40459862 | Stress-Induced Mutagenesis. |
Q51052567 | Stress-induced mutagenesis and complex adaptation. |
Q43633453 | Suramin is a potent and selective inhibitor of Mycobacterium tuberculosis RecA protein and the SOS response: RecA as a potential target for antibacterial drug discovery. |
Q93185974 | The SOS system: A complex and tightly regulated response to DNA damage |
Q39326888 | The Small RNA GcvB Promotes Mutagenic Break Repair by Opposing the Membrane Stress Response |
Q41050631 | The Transient Multidrug Resistance Phenotype of Salmonella enterica Swarming Cells Is Abolished by Sub-inhibitory Concentrations of Antimicrobial Compounds. |
Q34285727 | The evolution of stress-induced hypermutation in asexual populations |
Q34017416 | The sigma(E) stress response is required for stress-induced mutation and amplification in Escherichia coli |
Q37976150 | What limits the efficiency of double-strand break-dependent stress-induced mutation in Escherichia coli? |