scholarly article | Q13442814 |
P819 | ADS bibcode | 2014PLoSO...9j2734S |
P356 | DOI | 10.1371/JOURNAL.PONE.0102734 |
P8608 | Fatcat ID | release_5xr7twugdfhnfj5377vmbj7mqu |
P932 | PMC publication ID | 4100905 |
P698 | PubMed publication ID | 25028937 |
P5875 | ResearchGate publication ID | 263972683 |
P50 | author | Andrew James McMichael | Q21165187 |
Tanmoy Bhattacharya | Q55414889 | ||
Alan S. Perelson | Q55719625 | ||
P2093 | author name string | Hui Li | |
Feng Gao | |||
Hongshuo Song | |||
Shilpa S Iyer | |||
Bhavna Hora | |||
Kevin Wiehe | |||
Michael K P Liu | |||
Nilu Goonetilleke | |||
P2860 | cites work | Variable fitness impact of HIV-1 escape mutations to cytotoxic T lymphocyte (CTL) response | Q21131580 |
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Transmission of HIV-1 CTL escape variants provides HLA-mismatched recipients with a survival advantage | Q33325923 | ||
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Transmission of single HIV-1 genomes and dynamics of early immune escape revealed by ultra-deep sequencing | Q33680395 | ||
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Selective escape from CD8+ T-cell responses represents a major driving force of human immunodeficiency virus type 1 (HIV-1) sequence diversity and reveals constraints on HIV-1 evolution | Q34092644 | ||
Cooperation of B cell lineages in induction of HIV-1-broadly neutralizing antibodies | Q34114798 | ||
Early selection in Gag by protective HLA alleles contributes to reduced HIV-1 replication capacity that may be largely compensated for in chronic infection | Q34295972 | ||
Translating HIV sequences into quantitative fitness landscapes predicts viral vulnerabilities for rational immunogen design | Q34334803 | ||
Fitness cost of escape mutations in p24 Gag in association with control of human immunodeficiency virus type 1 | Q34545760 | ||
Progression to AIDS in South Africa is associated with both reverting and compensatory viral mutations | Q34841789 | ||
Rapid reversion of sequence polymorphisms dominates early human immunodeficiency virus type 1 evolution | Q35634318 | ||
Generation of transmitted/founder HIV-1 infectious molecular clones and characterization of their replication capacity in CD4 T lymphocytes and monocyte-derived macrophages | Q35826158 | ||
Compensatory mutation partially restores fitness and delays reversion of escape mutation within the immunodominant HLA-B*5703-restricted Gag epitope in chronic human immunodeficiency virus type 1 infection | Q35947590 | ||
Early low-titer neutralizing antibodies impede HIV-1 replication and select for virus escape | Q36000736 | ||
Escape from the dominant HLA-B27-restricted cytotoxic T-lymphocyte response in Gag is associated with a dramatic reduction in human immunodeficiency virus type 1 replication | Q36315510 | ||
Escape and compensation from early HLA-B57-mediated cytotoxic T-lymphocyte pressure on human immunodeficiency virus type 1 Gag alter capsid interactions with cyclophilin A. | Q36315572 | ||
Impact of immune escape mutations on HIV-1 fitness in the context of the cognate transmitted/founder genome | Q36394656 | ||
Intersubtype differences in the effect of a rare p24 gag mutation on HIV-1 replicative fitness | Q36414578 | ||
Vertical T cell immunodominance and epitope entropy determine HIV-1 escape | Q36497283 | ||
TCR clonotypes modulate the protective effect of HLA class I molecules in HIV-1 infection | Q36513955 | ||
Transmission of HIV-1 Gag immune escape mutations is associated with reduced viral load in linked recipients | Q36639954 | ||
Frequent and variable cytotoxic-T-lymphocyte escape-associated fitness costs in the human immunodeficiency virus type 1 subtype B Gag proteins | Q36759719 | ||
HIV-1 conserved-element vaccines: relationship between sequence conservation and replicative capacity | Q36827382 | ||
Marked epitope- and allele-specific differences in rates of mutation in human immunodeficiency type 1 (HIV-1) Gag, Pol, and Nef cytotoxic T-lymphocyte epitopes in acute/early HIV-1 infection | Q36898637 | ||
HLA-associated alterations in replication capacity of chimeric NL4-3 viruses carrying gag-protease from elite controllers of human immunodeficiency virus type 1. | Q37033250 | ||
HLA-associated clinical progression correlates with epitope reversion rates in early human immunodeficiency virus infection | Q37051649 | ||
Genetic identity, biological phenotype, and evolutionary pathways of transmitted/founder viruses in acute and early HIV-1 infection | Q37273158 | ||
The first T cell response to transmitted/founder virus contributes to the control of acute viremia in HIV-1 infection | Q37273195 | ||
Evolution of HLA-B*5703 HIV-1 escape mutations in HLA-B*5703-positive individuals and their transmission recipients | Q37273283 | ||
Impact of HLA in mother and child on disease progression of pediatric human immunodeficiency virus type 1 infection | Q37356088 | ||
Primary infection by a human immunodeficiency virus with atypical coreceptor tropism | Q38631923 | ||
HLA-B57/B*5801 human immunodeficiency virus type 1 elite controllers select for rare gag variants associated with reduced viral replication capacity and strong cytotoxic T-lymphocyte [corrected] recognition | Q41966337 | ||
Evolution of HIV-1 in an HLA-B*57-positive patient during virologic escape. | Q44082963 | ||
HIV evolution: CTL escape mutation and reversion after transmission | Q47211742 | ||
Detection of minor drug-resistant populations by parallel allele-specific sequencing. | Q54563995 | ||
Evolution and transmission of stable CTL escape mutations in HIV infection | Q56880602 | ||
Viral replication capacity as a correlate of HLA B57/B5801-associated nonprogressive HIV-1 infection | Q80819695 | ||
P275 | copyright license | Creative Commons CC0 License | Q6938433 |
P6216 | copyright status | copyrighted, dedicated to the public domain by copyright holder | Q88088423 |
P433 | issue | 7 | |
P407 | language of work or name | English | Q1860 |
P304 | page(s) | e102734 | |
P577 | publication date | 2014-07-16 | |
P1433 | published in | PLOS One | Q564954 |
P1476 | title | Reversion and T cell escape mutations compensate the fitness loss of a CD8+ T cell escape mutant in their cognate transmitted/founder virus | |
P478 | volume | 9 |
Q41919990 | A strongly selected mutation in the HIV-1 genome is independent of T cell responses and neutralizing antibodies. |
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Q32183405 | Immunologic and Virologic Mechanisms for Partial Protection from Intravenous Challenge by an Integration-Defective SIV Vaccine |
Q42680526 | Patterns and rates of viral evolution in HIV-1 subtype B infected females and males |
Q34686014 | Preexisting compensatory amino acids compromise fitness costs of a HIV-1 T cell escape mutation |
Q37464540 | Transmission of Multiple HIV-1 Subtype C Transmitted/founder Viruses into the Same Recipients Was not Determined by Modest Phenotypic Differences |
Q26798040 | What do mathematical models tell us about killing rates during HIV-1 infection? |
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