scholarly article | Q13442814 |
P6179 | Dimensions Publication ID | 1031512000 |
P356 | DOI | 10.1186/1742-4690-9-89 |
P2888 | exact match | https://scigraph.springernature.com/pub.10.1186/1742-4690-9-89 |
P8608 | Fatcat ID | release_z6g2hsscdzfsvbcnpcjewua56m |
P932 | PMC publication ID | 3496648 |
P698 | PubMed publication ID | 23110705 |
P5875 | ResearchGate publication ID | 232740526 |
P50 | author | Barton Haynes | Q4865631 |
Andrew James McMichael | Q21165187 | ||
Alan S. Perelson | Q55719625 | ||
Guido Ferrari | Q56481169 | ||
M Anthony Moody | Q73297881 | ||
George M. Shaw | Q78514357 | ||
Josh A Eudailey | Q88147253 | ||
Nilu Goonetilleke | Q100456048 | ||
Bhavna Hora | Q114512865 | ||
Fangping Cai | Q114512866 | ||
Hongshuo Song | Q114722126 | ||
Mark S Drinker | Q117248643 | ||
Joy Pickeral | Q125314693 | ||
Jeffrey W Pavlicek | Q125314735 | ||
Beatrice H. Hahn | Q55293537 | ||
Tanmoy Bhattacharya | Q55414889 | ||
P2093 | author name string | Hui Li | |
Feng Gao | |||
Shilpa S Iyer | |||
Michael K P Liu | |||
Julie M Decker | |||
Katharine J Bar | |||
Anna Berg | |||
P2860 | cites work | Variable fitness impact of HIV-1 escape mutations to cytotoxic T lymphocyte (CTL) response | Q21131580 |
Impaired fitness of human immunodeficiency virus type 1 variants with high-level resistance to protease inhibitors | Q28379411 | ||
Recombination rate and selection strength in HIV intra-patient evolution | Q28472689 | ||
HIV population dynamics in vivo: implications for genetic variation, pathogenesis, and therapy | Q29616404 | ||
Lower in vivo mutation rate of human immunodeficiency virus type 1 than that predicted from the fidelity of purified reverse transcriptase | Q29619994 | ||
Replicative fitness of historical and recent HIV-1 isolates suggests HIV-1 attenuation over time. | Q30351457 | ||
Unselected mutations in the human immunodeficiency virus type 1 genome are mostly nonsynonymous and often deleterious | Q31047702 | ||
Transmission of HIV-1 CTL escape variants provides HLA-mismatched recipients with a survival advantage | Q33325923 | ||
A dual infection/competition assay shows a correlation between ex vivo human immunodeficiency virus type 1 fitness and disease progression | Q33604871 | ||
Comparative fitness of multi-dideoxynucleoside-resistant human immunodeficiency virus type 1 (HIV-1) in an In vitro competitive HIV-1 replication assay | Q33814927 | ||
Dynamics and timing of in vivo mutations at Gag residue 242 during primary HIV-1 subtype C infection | Q33907924 | ||
Recombination in HIV-1 | Q57998149 | ||
High replication fitness and transmission efficiency of HIV-1 subtype C from India: Implications for subtype C predominance | Q83212088 | ||
Modeling and estimation of replication fitness of human immunodeficiency virus type 1 in vitro experiments by using a growth competition assay | Q34434646 | ||
Human immunodeficiency virus type 1 protease inhibitor drug-resistant mutants give discordant results when compared in single-cycle and multiple-cycle fitness assays | Q34489422 | ||
Fitness cost of escape mutations in p24 Gag in association with control of human immunodeficiency virus type 1 | Q34545760 | ||
Fitness comparison of thymidine analog resistance pathways in human immunodeficiency virus type 1. | Q34717892 | ||
Analysis of low-frequency mutations associated with drug resistance to raltegravir before antiretroviral treatment | Q34737411 | ||
Estimate of effective recombination rate and average selection coefficient for HIV in chronic infection | Q34804767 | ||
Replicative fitness costs of nonnucleoside reverse transcriptase inhibitor drug resistance mutations on HIV subtype C. | Q34933329 | ||
Fitness of drug resistant HIV-1: methodology and clinical implications | Q35046895 | ||
Evolution of drug-resistant viral populations during interruption of antiretroviral therapy | Q35077423 | ||
Fitness costs and diversity of the cytotoxic T lymphocyte (CTL) response determine the rate of CTL escape during acute and chronic phases of HIV infection | Q35274776 | ||
Compensatory mutation partially restores fitness and delays reversion of escape mutation within the immunodominant HLA-B*5703-restricted Gag epitope in chronic human immunodeficiency virus type 1 infection | Q35947590 | ||
Early low-titer neutralizing antibodies impede HIV-1 replication and select for virus escape | Q36000736 | ||
Escape and compensation from early HLA-B57-mediated cytotoxic T-lymphocyte pressure on human immunodeficiency virus type 1 Gag alter capsid interactions with cyclophilin A. | Q36315572 | ||
Clustered mutations in HIV-1 gag are consistently required for escape from HLA-B27-restricted cytotoxic T lymphocyte responses. | Q36376216 | ||
Transmission of HIV-1 Gag immune escape mutations is associated with reduced viral load in linked recipients | Q36639954 | ||
Quantitation of human immunodeficiency virus type 1 infection kinetics | Q36686530 | ||
CCR5- and CXCR4-tropic subtype C human immunodeficiency virus type 1 isolates have a lower level of pathogenic fitness than other dominant group M subtypes: implications for the epidemic | Q37191866 | ||
Genetic identity, biological phenotype, and evolutionary pathways of transmitted/founder viruses in acute and early HIV-1 infection | Q37273158 | ||
The first T cell response to transmitted/founder virus contributes to the control of acute viremia in HIV-1 infection | Q37273195 | ||
Evolution of HLA-B*5703 HIV-1 escape mutations in HLA-B*5703-positive individuals and their transmission recipients | Q37273283 | ||
Maternal transmission of human immunodeficiency virus escape mutations subverts HLA-B57 immunodominance but facilitates viral control in the haploidentical infant | Q37333818 | ||
Dynamics of HIV-1 recombination in its natural target cells | Q37356815 | ||
Primary infection by a human immunodeficiency virus with atypical coreceptor tropism | Q38631923 | ||
Changes in human immunodeficiency virus type 1 fitness and genetic diversity during disease progression | Q39534825 | ||
Estimating relative fitness in viral competition experiments. | Q39597718 | ||
Fitness disadvantage of transitional intermediates contributes to dynamic change in the infecting-virus population during coreceptor switch in R5 simian/human immunodeficiency virus-infected macaques | Q41230660 | ||
High rate of genetic recombination in murine leukemia virus: implications for influencing proviral ploidy | Q41496579 | ||
Factors underlying spontaneous inactivation and susceptibility to neutralization of human immunodeficiency virus | Q41611077 | ||
HLA-B57/B*5801 human immunodeficiency virus type 1 elite controllers select for rare gag variants associated with reduced viral replication capacity and strong cytotoxic T-lymphocyte [corrected] recognition | Q41966337 | ||
Fitness of human immunodeficiency virus type 1 protease inhibitor-selected single mutants | Q45740545 | ||
HIV evolution: CTL escape mutation and reversion after transmission | Q47211742 | ||
Detection of minor drug-resistant populations by parallel allele-specific sequencing. | Q54563995 | ||
P921 | main subject | HIV | Q15787 |
evasion of host immune response | Q1660157 | ||
HIV transmission | Q49029021 | ||
P304 | page(s) | 89 | |
P577 | publication date | 2012-10-30 | |
P1433 | published in | Retrovirology | Q15793508 |
P1476 | title | Impact of immune escape mutations on HIV-1 fitness in the context of the cognate transmitted/founder genome | |
P478 | volume | 9 |
Q41919990 | A strongly selected mutation in the HIV-1 genome is independent of T cell responses and neutralizing antibodies. |
Q34057942 | Analysis of HLA A*02 association with vaccine efficacy in the RV144 HIV-1 vaccine trial |
Q37272098 | Balance between transmitted HLA preadapted and nonassociated polymorphisms is a major determinant of HIV-1 disease progression. |
Q34114798 | Cooperation of B cell lineages in induction of HIV-1-broadly neutralizing antibodies |
Q34308788 | Early immune adaptation in HIV-1 revealed by population-level approaches |
Q35247825 | Extensive recombination due to heteroduplexes generates large amounts of artificial gene fragments during PCR |
Q37303016 | Factors affecting relative fitness measurements in pairwise competition assays of human immunodeficiency viruses |
Q36086336 | Fitness-Balanced Escape Determines Resolution of Dynamic Founder Virus Escape Processes in HIV-1 Infection. |
Q28487168 | HIV-1 Env C2-V4 diversification in a slow-progressor infant reveals a flat but rugged fitness landscape |
Q36827382 | HIV-1 conserved-element vaccines: relationship between sequence conservation and replicative capacity |
Q39080439 | Immune camouflage: relevance to vaccines and human immunology |
Q61811049 | Interaction of the Host and Viral Genome and Their Influence on HIV Disease |
Q50300594 | Kinetics of HIV-Specific CTL Responses Plays a Minimal Role in Determining HIV Escape Dynamics. |
Q92486763 | Modelling and in vitro testing of the HIV-1 Nef fitness landscape |
Q41213664 | Pairwise growth competition assay for determining the replication fitness of human immunodeficiency viruses |
Q34686014 | Preexisting compensatory amino acids compromise fitness costs of a HIV-1 T cell escape mutation |
Q35875253 | Recombination Enhances HIV-1 Envelope Diversity by Facilitating the Survival of Latent Genomic Fragments in the Plasma Virus Population. |
Q37532983 | Relative resistance of HIV-1 founder viruses to control by interferon-alpha |
Q42208022 | Reliable reconstruction of HIV-1 whole genome haplotypes reveals clonal interference and genetic hitchhiking among immune escape variants. |
Q37612507 | Resistance to type 1 interferons is a major determinant of HIV-1 transmission fitness |
Q33910862 | Reversion and T cell escape mutations compensate the fitness loss of a CD8+ T cell escape mutant in their cognate transmitted/founder virus |
Q36656789 | Short Communication: A Recombinant Variant with Increased Envelope Entry Efficiency Emerged During Early Infection of an HIV-1 Subtype C Dual Infected Rapid Progressor |
Q38640293 | T cell epitope redundancy: cross-conservation of the TCR face between pathogens and self and its implications for vaccines and autoimmunity |
Q100456050 | The HIV-1 latent reservoir is largely sensitive to circulating T cells |
Q41493589 | The host HLA-A*02 allele is associated with the response to pegylated interferon and ribavirin in patients with chronic hepatitis C virus infection |
Q35377813 | The route of HIV escape from immune response targeting multiple sites is determined by the cost-benefit tradeoff of escape mutations |
Q37547702 | The selection of low envelope glycoprotein reactivity to soluble CD4 and cold during simian-human immunodeficiency virus infection of rhesus macaques. |
Q37464540 | Transmission of Multiple HIV-1 Subtype C Transmitted/founder Viruses into the Same Recipients Was not Determined by Modest Phenotypic Differences |
Q26861425 | Using nonhuman primates to model HIV transmission |
Q26798040 | What do mathematical models tell us about killing rates during HIV-1 infection? |
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