scholarly article | Q13442814 |
P6179 | Dimensions Publication ID | 1001744545 |
P356 | DOI | 10.1038/SREP28399 |
P932 | PMC publication ID | 4914971 |
P698 | PubMed publication ID | 27323684 |
P2093 | author name string | Lei Wang | |
Xia Wang | |||
Yuan Zhang | |||
Jie Du | |||
Hui-Hua Li | |||
Hong-Xia Wang | |||
Yun-Long Xia | |||
Hai-Lian Bi | |||
Tian-Peng Zhang | |||
Xu-Min Guan | |||
P2860 | cites work | Identification of CHIP, a novel tetratricopeptide repeat-containing protein that interacts with heat shock proteins and negatively regulates chaperone functions | Q22010047 |
CHIP-dependent termination of MEKK2 regulates temporal ERK activation required for proper hyperosmotic response | Q24336920 | ||
Akt and CHIP coregulate tau degradation through coordinated interactions | Q24656202 | ||
Minimum information about a microarray experiment (MIAME)-toward standards for microarray data | Q27860569 | ||
ErbB2 degradation mediated by the co-chaperone protein CHIP | Q28208008 | ||
Specific recruitment of SH-PTP1 to the erythropoietin receptor causes inactivation of JAK2 and termination of proliferative signals | Q28236222 | ||
Mitogen-activated protein kinase signaling in the heart: angels versus demons in a heart-breaking tale | Q28296128 | ||
Signal transducer and activator of transcription 3 in the heart transduces not only a hypertrophic signal but a protective signal against doxorubicin-induced cardiomyopathy | Q28343848 | ||
Comparative cardiac toxicity of anthracyclines in vitro and in vivo in the mouse | Q28487998 | ||
The Hsc70 co-chaperone CHIP targets immature CFTR for proteasomal degradation | Q29616141 | ||
A mouse model for juvenile doxorubicin-induced cardiac dysfunction | Q33572418 | ||
Doxorubicin selectively inhibits muscle gene expression in cardiac muscle cells in vivo and in vitro | Q33614685 | ||
C-terminus of heat shock protein 70-interacting protein facilitates degradation of apoptosis signal-regulating kinase 1 and inhibits apoptosis signal-regulating kinase 1-dependent apoptosis | Q33899695 | ||
Adriamycin-induced heart failure: mechanism and modulation | Q33963576 | ||
Roles of the SHP-1 tyrosine phosphatase in the negative regulation of cell signalling | Q34038099 | ||
Oxidative Stress and Inflammation in Heart Disease: Do Antioxidants Have a Role in Treatment and/or Prevention? | Q35167634 | ||
The high-affinity HSP90-CHIP complex recognizes and selectively degrades phosphorylated tau client proteins | Q35629026 | ||
CHIP activates HSF1 and confers protection against apoptosis and cellular stress | Q36266926 | ||
Ubiquitin-mediated regulation of JAK-STAT signaling in embryonic stem cells | Q36895354 | ||
CHIP represses myocardin-induced smooth muscle cell differentiation via ubiquitin-mediated proteasomal degradation | Q37156982 | ||
Dissection of signal transduction pathways as a tool for the development of targeted therapies of hepatocellular carcinoma | Q37161303 | ||
Overexpression of Hsp20 prevents endotoxin-induced myocardial dysfunction and apoptosis via inhibition of NF-kappaB activation | Q37353112 | ||
The protective role of manganese superoxide dismutase against adriamycin-induced acute cardiac toxicity in transgenic mice | Q37359529 | ||
Doxorubicin-induced cardiomyopathy: from molecular mechanisms to therapeutic strategies | Q37998724 | ||
JAK2 the future: therapeutic strategies for JAK-dependent malignancies | Q38045061 | ||
Review and meta-analysis of incidence and clinical predictors of anthracycline cardiotoxicity | Q38146049 | ||
IL-6 activated JAK/STAT3 pathway and sensitivity to Hsp90 inhibitors in multiple myeloma | Q38204802 | ||
Promotion of CHIP-mediated p53 degradation protects the heart from ischemic injury | Q39291547 | ||
Atrogin-1/muscle atrophy F-box inhibits calcineurin-dependent cardiac hypertrophy by participating in an SCF ubiquitin ligase complex | Q40177208 | ||
Role of host protein tyrosine phosphatase SHP-1 in Leishmania donovani-induced inhibition of nitric oxide production | Q40216863 | ||
Loss of SHP1 enhances JAK3/STAT3 signaling and decreases proteosome degradation of JAK3 and NPM-ALK in ALK+ anaplastic large-cell lymphoma | Q40258085 | ||
The chaperone-associated ubiquitin ligase CHIP is able to target p53 for proteasomal degradation | Q40418287 | ||
Pifithrin-alpha protects against doxorubicin-induced apoptosis and acute cardiotoxicity in mice | Q40591772 | ||
Gene expression profiling identifies the novel role of immunoproteasome in doxorubicin-induced cardiotoxicity | Q41036599 | ||
Role of SHP-1, Kv.1.2, and cGMP in nitric oxide-induced ERK1/2 MAP kinase dephosphorylation in rat vascular smooth muscle cells | Q42480178 | ||
Heat shock protein 20 interacting with phosphorylated Akt reduces doxorubicin-triggered oxidative stress and cardiotoxicity | Q42585599 | ||
Hypothermia attenuates ischemia/reperfusion-induced endothelial cell apoptosis via alterations in apoptotic pathways and JNK signaling | Q43819838 | ||
Requirement for a hsp90 chaperone-dependent MEK1/2-ERK pathway for B cell antigen receptor-induced cyclin D2 expression in mature B lymphocytes | Q43874043 | ||
Deletion of the inducible 70-kDa heat shock protein genes in mice impairs cardiac contractile function and calcium handling associated with hypertrophy. | Q45972468 | ||
Targeted disruption of p53 attenuates doxorubicin-induced cardiac toxicity in mice | Q46597560 | ||
Phosphatase SHP-1 promotes TLR- and RIG-I-activated production of type I interferon by inhibiting the kinase IRAK1. | Q46661965 | ||
The antiproliferative drug doxorubicin inhibits liver fibrosis in bile duct-ligated rats and can be selectively delivered to hepatic stellate cells in vivo. | Q46913710 | ||
Preventive effect of erythropoietin on cardiac dysfunction in doxorubicin-induced cardiomyopathy. | Q46921630 | ||
Vigilance of hemodynamic changes immediately after transferring patients is crucial. | Q48030626 | ||
Induction of MAPK phosphatase-1 by hypothermia inhibits TNF-alpha-induced endothelial barrier dysfunction and apoptosis | Q48446106 | ||
Granulocyte colony-stimulating factor improves left ventricular function of doxorubicin-induced cardiomyopathy. | Q50694953 | ||
CHIP enhances angiogenesis and restores cardiac function after infarction in transgenic mice. | Q51532758 | ||
Carboxyl terminus of heat shock protein 70-interacting protein inhibits angiotensin II-induced cardiac remodeling. | Q51539199 | ||
CHIP, a cochaperone/ubiquitin ligase that regulates protein quality control, is required for maximal cardioprotection after myocardial infarction in mice. | Q51594198 | ||
Fetal alcohol exposure impairs Hedgehog cholesterol modification and signaling. | Q51996552 | ||
Microarray and Co-expression Network Analysis of Genes Associated with Acute Doxorubicin Cardiomyopathy in Mice. | Q53641268 | ||
Inhibition of Toll-like receptor 2 reduces cardiac fibrosis by attenuating macrophage-mediated inflammation. | Q54562352 | ||
Is malonaldehyde a valuable indicator of lipid peroxidation? | Q68051991 | ||
Suppression of doxorubicin cardiotoxicity by overexpression of catalase in the heart of transgenic mice | Q71119918 | ||
Anthracycline-induced cardiotoxicity | Q82298914 | ||
P275 | copyright license | Creative Commons Attribution 4.0 International | Q20007257 |
P6216 | copyright status | copyrighted | Q50423863 |
P407 | language of work or name | English | Q1860 |
P304 | page(s) | 28399 | |
P577 | publication date | 2016-06-21 | |
P1433 | published in | Scientific Reports | Q2261792 |
P1476 | title | Protection against doxorubicin-induced myocardial dysfunction in mice by cardiac-specific expression of carboxyl terminus of hsp70-interacting protein | |
P478 | volume | 6 |
Q33648557 | Angiotensin-converting enzyme 2 overexpression protects against doxorubicin-induced cardiomyopathy by multiple mechanisms in rats |
Q55265082 | Cardiomyocyte-specific disruption of Cathepsin K protects against doxorubicin-induced cardiotoxicity. |
Q58077030 | Emergence, Development, and Future of Cardio-Oncology in China |
Q92608163 | Ethanol exposure in prenatal and early postnatal induced cardiac injury in rats: involvement of oxidative stress, Hsp70, ERK 1/2, JNK, and apoptosis in a 3-month follow-up study |
Q92757459 | Extracorporeal shock waves protect cardiomyocytes from doxorubicin-induced cardiomyopathy by upregulating survivin via the integrin-ILK-Akt-Sp1/p53 axis |
Q52717628 | Inhibition of TRPA1 Attenuates Doxorubicin-Induced Acute Cardiotoxicity by Suppressing Oxidative Stress, the Inflammatory Response, and Endoplasmic Reticulum Stress. |
Q58737121 | Interleukin-12p35 Knock Out Aggravates Doxorubicin-Induced Cardiac Injury and Dysfunction by Aggravating the Inflammatory Response, Oxidative Stress, Apoptosis and Autophagy in Mice |
Q40954179 | Mechanistic clues to the protective effect of chrysin against doxorubicin-induced cardiomyopathy: Plausible roles of p53, MAPK and AKT pathways |
Q47101716 | Oral administration of Ginsenoside Rg1 prevents cardiac toxicity induced by doxorubicin in mice through anti-apoptosis |
Q42678476 | Signalling mechanisms underlying doxorubicin and Nox2 NADPH oxidase-induced cardiomyopathy: involvement of mitofusin-2. |
Q99710920 | Sodium nitrate co-supplementation does not exacerbate low dose metronomic doxorubicin-induced cachexia in healthy mice |
Q58617447 | The increased expression of the inducible Hsp70 (HSP70A1A) in serum of patients with heart failure and its protective effect against the cardiotoxic agent doxorubicin |
Q37658559 | UVRAG Deficiency Exacerbates Doxorubicin-Induced Cardiotoxicity |
Q64249100 | Ursolic acid prevents doxorubicin-induced cardiac toxicity in mice through eNOS activation and inhibition of eNOS uncoupling |
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