| scholarly article | Q13442814 |
| P50 | author | Changwei Shao | Q55098289 |
| Lianyun Li | Q57308436 | ||
| Min Wu | Q61190852 | ||
| P2093 | author name string | Bo Yang | |
| Xiang Wang | |||
| Xiang-Dong Fu | |||
| Yuan Zhu | |||
| Gang Wei | |||
| Pin-Ji Lei | |||
| Kai Huang | |||
| Kun Zhu | |||
| Lin-Gao Ju | |||
| P2860 | cites work | BTB domain-containing speckle-type POZ protein (SPOP) serves as an adaptor of Daxx for ubiquitination by Cul3-based ubiquitin ligase. | Q53633362 |
| The mRNA expression of SETD2 in human breast cancer: correlation with clinico-pathological parameters | Q21260386 | ||
| Tumor-suppressor role for the SPOP ubiquitin ligase in signal-dependent proteolysis of the oncogenic co-activator SRC-3/AIB1 | Q24303428 | ||
| The Iws1:Spt6:CTD complex controls cotranscriptional mRNA biosynthesis and HYPB/Setd2-mediated histone H3K36 methylation | Q24316355 | ||
| Molecular regulation of H3K4 trimethylation by Wdr82, a component of human Set1/COMPASS | Q24320097 | ||
| Exome sequencing identifies frequent mutation of the SWI/SNF complex gene PBRM1 in renal carcinoma | Q24337664 | ||
| The histone mark H3K36me3 regulates human DNA mismatch repair through its interaction with MutSα | Q24339204 | ||
| SPOP promotes tumorigenesis by acting as a key regulatory hub in kidney cancer | Q24339492 | ||
| Stable X chromosome inactivation involves the PRC1 Polycomb complex and requires histone MACROH2A1 and the CULLIN3/SPOP ubiquitin E3 ligase | Q24529056 | ||
| The genetic basis of early T-cell precursor acute lymphoblastic leukaemia | Q24601515 | ||
| Exome sequencing identifies recurrent SPOP, FOXA1 and MED12 mutations in prostate cancer | Q24616117 | ||
| Structures of SPOP-Substrate Complexes: Insights into Molecular Architectures of BTB-Cul3 Ubiquitin Ligases | Q27657740 | ||
| Comprehensive molecular characterization of clear cell renal cell carcinoma | Q27852374 | ||
| Set2 methylation of histone H3 lysine 36 suppresses histone exchange on transcribed genes | Q27929868 | ||
| Cotranscriptional set2 methylation of histone H3 lysine 36 recruits a repressive Rpd3 complex | Q27935315 | ||
| Identification of a novel nuclear speckle-type protein, SPOP | Q28118699 | ||
| SINK Is a p65-interacting Negative Regulator of NF-κB-dependent Transcription | Q28203033 | ||
| Dynamic histone H3 methylation during gene induction: HYPB/Setd2 mediates all H3K36 trimethylation | Q28593817 | ||
| Systematic sequencing of renal carcinoma reveals inactivation of histone modifying genes | Q29615757 | ||
| A novel domain in Set2 mediates RNA polymerase II interaction and couples histone H3 K36 methylation with transcript elongation | Q30448402 | ||
| The target of the NSD family of histone lysine methyltransferases depends on the nature of the substrate | Q33553524 | ||
| Regulation of alternative splicing by histone modifications | Q34035543 | ||
| The genetic basis of kidney cancer: a metabolic disease | Q34087148 | ||
| Prostate cancer. Ubiquitylome analysis identifies dysregulation of effector substrates in SPOP-mutant prostate cancer | Q34637940 | ||
| The Brd4 extraterminal domain confers transcription activation independent of pTEFb by recruiting multiple proteins, including NSD3. | Q35096586 | ||
| Pre-mRNA splicing is a determinant of histone H3K36 methylation | Q35170883 | ||
| BS69/ZMYND11 reads and connects histone H3.3 lysine 36 trimethylation-decorated chromatin to regulated pre-mRNA processing | Q35186308 | ||
| SETD2-dependent histone H3K36 trimethylation is required for homologous recombination repair and genome stability | Q35188529 | ||
| Identification of functional cooperative mutations of SETD2 in human acute leukemia | Q35632973 | ||
| RNA polymerase II carboxyl-terminal domain phosphorylation regulates protein stability of the Set2 methyltransferase and histone H3 di- and trimethylation at lysine 36 | Q35728265 | ||
| Prostate cancer-associated mutations in speckle-type POZ protein (SPOP) regulate steroid receptor coactivator 3 protein turnover | Q36799180 | ||
| Analysis of Drosophila segmentation network identifies a JNK pathway factor overexpressed in kidney cancer. | Q37373844 | ||
| Variation in chromatin accessibility in human kidney cancer links H3K36 methyltransferase loss with widespread RNA processing defects | Q37550216 | ||
| Understanding the language of Lys36 methylation at histone H3. | Q37672319 | ||
| Epigenetic regulation in RCC: opportunities for therapeutic intervention? | Q37976256 | ||
| Position-dependent alternative splicing activity revealed by global profiling of alternative splicing events regulated by PTB. | Q38341668 | ||
| SETD2 is required for DNA double-strand break repair and activation of the p53-mediated checkpoint. | Q38993472 | ||
| Trimethylation of histone H3 lysine 36 by human methyltransferase PRDM9 protein | Q39007308 | ||
| Transcriptionally active chromatin recruits homologous recombination at DNA double-strand breaks. | Q39012832 | ||
| Androgen receptor is the key transcriptional mediator of the tumor suppressor SPOP in prostate cancer. | Q42153553 | ||
| Targeted exome sequencing in clear cell renal cell carcinoma tumors suggests aberrant chromatin regulation as a crucial step in ccRCC development. | Q45880509 | ||
| A hedgehog-induced BTB protein modulates hedgehog signaling by degrading Ci/Gli transcription factor. | Q47071852 | ||
| P275 | copyright license | Creative Commons Attribution-NonCommercial 4.0 International | Q34179348 |
| P6216 | copyright status | copyrighted | Q50423863 |
| P433 | issue | 1 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | alternative mRNA splicing, via spliceosome | Q21114084 |
| P1104 | number of pages | 14 | |
| P304 | page(s) | 92-105 | |
| P577 | publication date | 2016-09-09 | |
| P1433 | published in | Nucleic Acids Research | Q135122 |
| P1476 | title | SPOP-containing complex regulates SETD2 stability and H3K36me3-coupled alternative splicing | |
| P478 | volume | 45 |
| Q47222829 | Dysregulation of cotranscriptional alternative splicing underlies CHARGE syndrome |
| Q38649645 | Epigenomic analysis in a cell-based model reveals the roles of H3K9me3 in breast cancer transformation. |
| Q47413250 | Functional analysis of Cullin 3 E3 ligases in tumorigenesis. |
| Q57116830 | Functional roles of Speckle-Type Poz (SPOP) Protein in Genomic stability |
| Q96126545 | GLIS2 promotes colorectal cancer through repressing enhancer activation |
| Q64254974 | Histone demethylase KDM3A is required for enhancer activation of hippo target genes in colorectal cancer |
| Q47927828 | Histone methyltransferase SETD2 modulates alternative splicing to inhibit intestinal tumorigenesis |
| Q47133300 | Independence between pre-mRNA splicing and DNA methylation in an isogenic minigene resource |
| Q92977938 | SETD2 mutation in renal clear cell carcinoma suppress autophagy via regulation of ATG12 |
| Q57472357 | SETD2-dependent H3K36me3 plays a critical role in epigenetic regulation of the HPV31 life cycle |
| Q58772309 | SPOP promotes transcriptional expression of DNA repair and replication factors to prevent replication stress and genomic instability |
| Q91598898 | SPOP suppresses prostate cancer through regulation of CYCLIN E1 stability |
| Q39225775 | Shaping the cellular landscape with Set2/SETD2 methylation |
| Q94474488 | The diverse roles of SPOP in prostate cancer and kidney cancer |
| Q64101715 | The hyper-activation of transcriptional enhancers in breast cancer |
| Q58700177 | Ubiquitin Regulation: The Histone Modifying Enzyme's Story |
| Q39268171 | Writing, erasing and reading histone lysine methylations. |
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