Does FGF23 toxicity influence the outcome of chronic kidney disease?

scientific article published on 7 November 2008

Does FGF23 toxicity influence the outcome of chronic kidney disease? is …
instance of (P31):
review articleQ7318358
scholarly articleQ13442814
editorialQ871232

External links are
P356DOI10.1093/NDT/GFN620
P932PMC publication ID2721424
P698PubMed publication ID18996835
P5875ResearchGate publication ID23461375

P2093author name stringMohammed Shawkat Razzaque
P2860cites workAutosomal dominant hypophosphataemic rickets is associated with mutations in FGF23Q24290481
FGF-23 is a potent regulator of vitamin D metabolism and phosphate homeostasisQ24319751
Cloning and characterization of FGF23 as a causative factor of tumor-induced osteomalaciaQ24623628
Molecular insights into the klotho-dependent, endocrine mode of action of fibroblast growth factor 19 subfamily membersQ24683254
Role of fibroblast growth factor 23 in health and in chronic kidney diseaseQ28254210
Fibroblast growth factor 23: roles in health and diseaseQ28262842
Klotho converts canonical FGF receptor into a specific receptor for FGF23Q28272505
Chronic kidney disease and the risks of death, cardiovascular events, and hospitalizationQ28283679
Circulating FGF-23 is regulated by 1alpha,25-dihydroxyvitamin D3 and phosphorus in vivoQ28572995
Fibroblast growth factor 23 and mortality among patients undergoing hemodialysisQ33932297
Circulating concentration of FGF-23 increases as renal function declines in patients with chronic kidney disease, but does not change in response to variation in phosphate intake in healthy volunteersQ33974129
Fibroblast growth factor-23 mitigates hyperphosphatemia but accentuates calcitriol deficiency in chronic kidney diseaseQ34557934
Dynamics of parathyroid hormone secretion in health and secondary hyperparathyroidism.Q34702230
Cellular and molecular events leading to renal tubulointerstitial fibrosis.Q34786418
A homozygous missense mutation in human KLOTHO causes severe tumoral calcinosisQ35925637
FGF23 and disorders of phosphate homeostasisQ36111415
Structural and biochemical properties of fibroblast growth factor 23.Q36216956
Hypophosphatemia: an update on its etiology and treatmentQ36272304
Phosphatonins and the regulation of phosphate homeostasisQ36604351
Emerging role of fibroblast growth factor 23 in a bone-kidney axis regulating systemic phosphate homeostasis and extracellular matrix mineralizationQ36848109
The emerging role of the fibroblast growth factor-23-klotho axis in renal regulation of phosphate homeostasisQ36860289
Does renal ageing affect survival?Q36896257
New aspect of renal phosphate reabsorption: the type IIc sodium-dependent phosphate transporter.Q36905432
Can fibroblast growth factor 23 fine-tune therapies for diseases of abnormal mineral ion metabolism?Q36966608
In vivo genetic evidence for klotho-dependent, fibroblast growth factor 23 (Fgf23) -mediated regulation of systemic phosphate homeostasisQ37072412
Does Fgf23-klotho activity influence vascular and soft tissue calcification through regulating mineral ion metabolism?Q37182735
Amelioration of the premature ageing-like features of Fgf-23 knockout mice by genetically restoring the systemic actions of FGF-23Q37418420
Serum fibroblast growth factor-23 levels predict the future refractory hyperparathyroidism in dialysis patients.Q40447584
An FGF23 missense mutation causes familial tumoral calcinosis with hyperphosphatemia.Q40482177
Transgenic mice overexpressing human fibroblast growth factor 23 (R176Q) delineate a putative role for parathyroid hormone in renal phosphate wasting disordersQ40530218
Premature aging-like phenotype in fibroblast growth factor 23 null mice is a vitamin D-mediated processQ42736434
Transgenic mice expressing fibroblast growth factor 23 under the control of the alpha1(I) collagen promoter exhibit growth retardation, osteomalacia, and disturbed phosphate homeostasisQ44779387
Sinoatrial node dysfunction and early unexpected death of mice with a defect of klotho gene expressionQ44810019
FGF-23 in patients with end-stage renal disease on hemodialysisQ44846350
Fibroblast growth factor-23 relationship to dietary phosphate and renal phosphate handling in healthy young men.Q45197408
Dietary phosphorus regulates serum fibroblast growth factor-23 concentrations in healthy men.Q46680438
Severely reduced production of klotho in human chronic renal failure kidneyQ49064415
Mineral metabolism, mortality, and morbidity in maintenance hemodialysisQ50144395
FGF-23 transgenic mice demonstrate hypophosphatemic rickets with reduced expression of sodium phosphate cotransporter type IIa.Q54733425
Hypervitaminosis D and premature aging: lessons learned from Fgf23 and Klotho mutant miceQ56767950
Association of Disorders in Mineral Metabolism with Progression of Chronic Kidney DiseaseQ57772116
The collagen-specific molecular chaperone HSP47: is there a role in fibrosis?Q79433884
FGF-23, vitamin D and calcification: the unholy triadQ80394650
Intravenous calcitriol therapy increases serum concentrations of fibroblast growth factor-23 in dialysis patients with secondary hyperparathyroidismQ81853118
P433issue1
P407language of work or nameEnglishQ1860
P304page(s)4-7
P577publication date2008-11-07
P1433published inNephrology Dialysis TransplantationQ15710302
P1476titleDoes FGF23 toxicity influence the outcome of chronic kidney disease?
P478volume24

Reverse relations

cites work (P2860)
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Q37641052Clinical relevance of FGF-23 in chronic kidney disease
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Q64970703Endocrine regulation of MFS2 by branchless controls phosphate excretion and stone formation in Drosophila renal tubules.
Q39866881FGF-23 and future cardiovascular events in patients with chronic kidney disease before initiation of dialysis treatment
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Q43074642FGF23-induced hypophosphatemia persists in Hyp mice deficient in the WNT coreceptor Lrp6.
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