scholarly article | Q13442814 |
P2093 | author name string | Beate Lanske | |
M Shawkat Razzaque | |||
Mutsuko Ohnishi | |||
Teruyo Nakatani | |||
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Klotho, a gene related to a syndrome resembling human premature aging, functions in a negative regulatory circuit of vitamin D endocrine system | Q28207490 | ||
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New aspect of renal phosphate reabsorption: the type IIc sodium-dependent phosphate transporter. | Q36905432 | ||
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Does Fgf23-klotho activity influence vascular and soft tissue calcification through regulating mineral ion metabolism? | Q37182735 | ||
Inactivation of klotho function induces hyperphosphatemia even in presence of high serum fibroblast growth factor 23 levels in a genetically engineered hypophosphatemic (Hyp) mouse model | Q37415834 | ||
The beta-glucuronidase klotho hydrolyzes and activates the TRPV5 channel | Q40359636 | ||
Premature aging-like phenotype in fibroblast growth factor 23 null mice is a vitamin D-mediated process | Q42736434 | ||
Does FGF23 toxicity influence the outcome of chronic kidney disease? | Q43078080 | ||
Mineral metabolism, mortality, and morbidity in maintenance hemodialysis | Q50144395 | ||
Hypervitaminosis D and premature aging: lessons learned from Fgf23 and Klotho mutant mice | Q56767950 | ||
Collagen-binding heat shock protein (HSP) 47 expression in anti-thymocyte serum (ATS)-induced glomerulonephritis. | Q64999385 | ||
Immunolocalization of collagen and collagen-binding heat shock protein 47 in fibrotic lung diseases | Q77747025 | ||
Life-long suppression of growth hormone-insulin-like growth factor I activity in genetically altered rats could prevent age-related renal damage | Q80228977 | ||
FGF-23, vitamin D and calcification: the unholy triad | Q80394650 | ||
Genetic suppression of GH-IGF-1 activity, combined with lifelong caloric restriction, prevents age-related renal damage and prolongs the life span in rats | Q81147498 | ||
P433 | issue | 6 | |
P304 | page(s) | 583-590 | |
P577 | publication date | 2009-09-21 | |
P1433 | published in | Circulation: Cardiovascular Genetics | Q15816430 |
P1476 | title | In vivo genetic evidence for suppressing vascular and soft-tissue calcification through the reduction of serum phosphate levels, even in the presence of high serum calcium and 1,25-dihydroxyvitamin d levels | |
P478 | volume | 2 |
Q42727584 | A phosphate-centric paradigm for pathophysiology and therapy of chronic kidney disease |
Q37062520 | Accelerated ageing and renal dysfunction links lower socioeconomic status and dietary phosphate intake |
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Q37498019 | Can salivary phosphate levels be an early biomarker to monitor the evolvement of obesity? |
Q37853829 | Cell biological and physicochemical aspects of arterial calcification |
Q35003219 | Dietary and genetic evidence for enhancing glucose metabolism and reducing obesity by inhibiting klotho functions |
Q34072221 | Dietary and genetic evidence for phosphate toxicity accelerating mammalian aging |
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Q34788510 | Genetic ablation of sfrp4 in mice does not affect serum phosphate homeostasis. |
Q36992835 | Genetic induction of phosphate toxicity significantly reduces the survival of hypercholesterolemic obese mice |
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Q90721090 | In Vivo Toxicity and In Vitro Solubility Assessment of Pre-Treated Struvite as A Potential Alternative Phosphorus Source in Animal Feed |
Q28535055 | Inadequate awareness among chronic kidney disease patients regarding food and drinks containing artificially added phosphate |
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Q37606037 | Klotho deficiency disrupts hematopoietic stem cell development and erythropoiesis |
Q38115124 | Klotho, phosphate and FGF-23 in ageing and disturbed mineral metabolism |
Q64231913 | Lack of Awareness of Dietary Sources of Phosphorus Is a Clinical Concern |
Q34119083 | Lack of awareness among future medical professionals about the risk of consuming hidden phosphate-containing processed food and drinks |
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Q53357907 | Spironolactone ameliorates arterial medial calcification in uremic rats: the role of mineralocorticoid receptor signaling in vascular calcification. |
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