| scholarly article | Q13442814 |
| P356 | DOI | 10.1016/S1097-2765(00)80469-4 |
| P698 | PubMed publication ID | 10230394 |
| P50 | author | Stanley J. Korsmeyer | Q138795 |
| Matthias Wilm | Q63409908 | ||
| P2093 | author name string | J D Scott | |
| H Harada | |||
| M Mann | |||
| S S Taylor | |||
| L J Huang | |||
| B Becknell | |||
| P2860 | cites work | Bid, a Bcl2 Interacting Protein, Mediates Cytochrome c Release from Mitochondria in Response to Activation of Cell Surface Death Receptors | Q24310559 |
| Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis | Q24310597 | ||
| Dimerization properties of human BAD. Identification of a BH-3 domain and analysis of its binding to mutant BCL-2 and BCL-XL proteins | Q24310631 | ||
| Cytochrome c and dATP-Dependent Formation of Apaf-1/Caspase-9 Complex Initiates an Apoptotic Protease Cascade | Q24311006 | ||
| Bad, a heterodimeric partner for Bcl-XL and Bcl-2, displaces Bax and promotes cell death | Q24311298 | ||
| Bcl-xL Regulates the Membrane Potential and Volume Homeostasis of Mitochondria | Q24311858 | ||
| Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programmed cell death | Q24312211 | ||
| BID: a novel BH3 domain-only death agonist | Q24320091 | ||
| Bcl-2 targets the protein kinase Raf-1 to mitochondria | Q24321753 | ||
| harakiri, a novel regulator of cell death, encodes a protein that activates apoptosis and interacts selectively with survival-promoting proteins Bcl-2 and Bcl-X(L) | Q24532147 | ||
| Bim: a novel member of the Bcl-2 family that promotes apoptosis | Q24532974 | ||
| Enforced dimerization of BAX results in its translocation, mitochondrial dysfunction and apoptosis | Q24533270 | ||
| Movement of Bax from the Cytosol to Mitochondria during Apoptosis | Q24677881 | ||
| Solution structure of the proapoptotic molecule BID: a structural basis for apoptotic agonists and antagonists | Q27617723 | ||
| Structure of Bcl-xL-Bak peptide complex: recognition between regulators of apoptosis | Q27734734 | ||
| Akt Phosphorylation of BAD Couples Survival Signals to the Cell-Intrinsic Death Machinery | Q27860586 | ||
| Signaling Through Scaffold, Anchoring, and Adaptor Proteins | Q28131792 | ||
| BH3 Domain of BAD Is Required for Heterodimerization with BCL-XL and Pro-apoptotic Activity | Q28249553 | ||
| Interleukin-3-Induced Phosphorylation of BAD Through the Protein Kinase Akt | Q28255373 | ||
| The C. elegans Protein EGL-1 Is Required for Programmed Cell Death and Interacts with the Bcl-2–like Protein CED-9 | Q28272159 | ||
| The Kit receptor promotes cell survival via activation of PI 3-kinase and subsequent Akt-mediated phosphorylation of Bad on Ser136 | Q28275577 | ||
| Characterization of S-AKAP84, a novel developmentally regulated A kinase anchor protein of male germ cells | Q28285035 | ||
| Serine phosphorylation of death agonist BAD in response to survival factor results in binding to 14-3-3 not BCL-X(L) | Q28297419 | ||
| Localization of protein kinases by anchoring proteins: a theme in signal transduction | Q28299903 | ||
| Identification of a novel protein kinase A anchoring protein that binds both type I and type II regulatory subunits | Q28305592 | ||
| Caspase cleaved BID targets mitochondria and is required for cytochrome c release, while BCL-XL prevents this release but not tumor necrosis factor-R1/Fas death | Q28586301 | ||
| PD 098059 is a specific inhibitor of the activation of mitogen-activated protein kinase kinase in vitro and in vivo | Q28609356 | ||
| Role of translocation in the activation and function of protein kinase B | Q28609902 | ||
| The protein kinase encoded by the Akt proto-oncogene is a target of the PDGF-activated phosphatidylinositol 3-kinase | Q29618387 | ||
| Bcl-2 is an inner mitochondrial membrane protein that blocks programmed cell death | Q29620000 | ||
| Social controls on cell survival and cell death | Q34372181 | ||
| Dissociation of cytokine-induced phosphorylation of Bad and activation of PKB/akt: Involvement of MEK upstream of Bad phosphorylation | Q36490362 | ||
| Interleukin 3-dependent survival by the Akt protein kinase | Q36593239 | ||
| Anchoring of protein kinase A facilitates hormone-mediated insulin secretion | Q36834213 | ||
| BAX-induced cell death may not require interleukin 1 beta-converting enzyme-like proteases | Q37044625 | ||
| Etoposide and cisplatin induced apoptosis in activated RAW 264.7 macrophages is attenuated by cAMP-induced gene expression | Q38334834 | ||
| Bad Is a BH3 Domain-Containing Protein That Forms an Inactivating Dimer with Bcl-XL | Q40024055 | ||
| Cytokine receptor signalling | Q40416594 | ||
| On target with a new mechanism for the regulation of protein phosphorylation | Q40855882 | ||
| Mitochondrial control of apoptosis | Q41339880 | ||
| PI3K: downstream AKTion blocks apoptosis | Q41351876 | ||
| Protein kinase A anchoring | Q41505004 | ||
| Characterization of the signaling interactions that promote the survival and growth of developing retinal ganglion cells in culture | Q46084557 | ||
| Anchoring of protein kinase A is required for modulation of AMPA/kainate receptors on hippocampal neurons | Q48137676 | ||
| The Overexpression of Bax Produces Cell Death upon Induction of the Mitochondrial Permeability Transition | Q74358996 | ||
| P433 | issue | 4 | |
| P1104 | number of pages | 10 | |
| P304 | page(s) | 413-422 | |
| P577 | publication date | 1999-04-01 | |
| P1433 | published in | Molecular Cell | Q3319468 |
| P1476 | title | Phosphorylation and inactivation of BAD by mitochondria-anchored protein kinase A | |
| P478 | volume | 3 |
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