HilD, HilC, and RtsA Form Homodimers and Heterodimers to Regulate Expression of the Salmonella Pathogenicity Island I Type III Secretion System

scientific article published on 10 February 2020

HilD, HilC, and RtsA Form Homodimers and Heterodimers to Regulate Expression of the Salmonella Pathogenicity Island I Type III Secretion System is …
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scholarly articleQ13442814

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P356DOI10.1128/JB.00012-20
P932PMC publication ID7148127
P698PubMed publication ID32041797

P50authorJames M SlauchQ54142755
P2093author name stringMarinos Kalafatis
Koh-Eun Narm
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FliZ Regulates Expression of theSalmonellaPathogenicity Island 1 Invasion Locus by Controlling HilD Protein Activity inSalmonella entericaSerovar TyphimuriumQ42162171
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HilE Regulates HilD by Blocking DNA Binding in Salmonella enterica serovar TyphimuriumQ49597228
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AraC/XylS family members, HilD and HilC, directly activate virulence gene expression independently of HilA in Salmonella typhimurium.Q50105717
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Two AraC/XylS family members can independently counteract the effect of repressing sequences upstream of the hilA promoterQ50126541
P577publication date2020-02-10
P1433published inJournal of BacteriologyQ478419
P1476titleHilD, HilC, and RtsA Form Homodimers and Heterodimers to Regulate Expression of the Salmonella Pathogenicity Island I Type III Secretion System

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