| scholarly article | Q13442814 |
| P2093 | author name string | Jessica E Cott Chubiz | |
| James M Slauch | |||
| Yekaterina A Golubeva | |||
| Jeremy R Ellermeier | |||
| P2860 | cites work | Structure of Vibrio cholerae ToxT reveals a mechanism for fatty acid regulation of virulence genes | Q27659552 |
| Flip-flop of oleic acid in a phospholipid membrane: rate and mechanism. | Q46024780 | ||
| Fatty acid flip-flop in a model membrane is faster than desorption into the aqueous phase | Q46435469 | ||
| Construction of targeted single copy lac fusions using lambda Red and FLP-mediated site-specific recombination in bacteria | Q48298401 | ||
| An externally modulated, noise-driven switch for the regulation of SPI1 in Salmonella enterica serovar Typhimurium. | Q50044875 | ||
| One-step inactivation of chromosomal genes in Escherichia coli K-12 using PCR products | Q27860842 | ||
| Identification of the major intestinal fatty acid transport protein | Q28145772 | ||
| Detection of host-derived sphingosine by Pseudomonas aeruginosa is important for survival in the murine lung | Q28492560 | ||
| The Salmonella SPI1 type three secretion system responds to periplasmic disulfide bond status via the flagellar apparatus and the RcsCDB system | Q28492915 | ||
| FadD is required for utilization of endogenous fatty acids released from membrane lipids | Q28743102 | ||
| An efficient recombination system for chromosome engineering in Escherichia coli | Q29615038 | ||
| Gene disruption in Escherichia coli: TcR and KmR cassettes with the option of Flp-catalyzed excision of the antibiotic-resistance determinant | Q29615039 | ||
| DNA-binding activities of the HilC and HilD virulence regulatory proteins of Salmonella enterica serovar Typhimurium | Q30309622 | ||
| Perturbation of the small intestine microbial ecology by streptomycin alters pathology in a Salmonella enterica serovar typhimurium murine model of infection | Q33443646 | ||
| The role of coupled positive feedback in the expression of the SPI1 type three secretion system in Salmonella | Q33649749 | ||
| N-terminal residues of the Vibrio cholerae virulence regulatory protein ToxT involved in dimerization and modulation by fatty acids | Q33932535 | ||
| Differential regulation of Salmonella typhimurium type III secreted proteins by pathogenicity island 1 (SPI-1)-encoded transcriptional activators InvF and hilA. | Q34001486 | ||
| Salmonella interactions with host cells: type III secretion at work | Q34425135 | ||
| Integration of a complex regulatory cascade involving the SirA/BarA and Csr global regulatory systems that controls expression of the Salmonella SPI-1 and SPI-2 virulence regulons through HilD | Q35049823 | ||
| Compounds Which Serve as the Sole Source of Carbon or Nitrogen for Salmonella typhimurium LT-2 | Q35157309 | ||
| RtsA and RtsB coordinately regulate expression of the invasion and flagellar genes in Salmonella enterica serovar Typhimurium. | Q35662912 | ||
| cis-acting ompF mutations that result in OmpR-dependent constitutive expression | Q36148065 | ||
| The route of enteric infection in normal mice | Q36273394 | ||
| Salmonella typhimurium initiates murine infection by penetrating and destroying the specialized epithelial M cells of the Peyer's patches. | Q36363455 | ||
| Identification of transport proteins involved in free fatty acid efflux in Escherichia coli. | Q36506203 | ||
| The intestinal fatty acid propionate inhibits Salmonella invasion through the post-translational control of HilD | Q36636799 | ||
| A 40 kb chromosomal fragment encoding Salmonella typhimurium invasion genes is absent from the corresponding region of the Escherichia coli K-12 chromosome | Q36707901 | ||
| Formate acts as a diffusible signal to induce Salmonella invasion | Q36747272 | ||
| The Salmonella Spi1 virulence regulatory protein HilD directly activates transcription of the flagellar master operon flhDC. | Q37713164 | ||
| The HilA box and sequences outside it determine the magnitude of HilA-dependent activation of P(prgH) from Salmonella pathogenicity island 1. | Q38298661 | ||
| Butyrate specifically down-regulates salmonella pathogenicity island 1 gene expression. | Q38317128 | ||
| Two-Carbon Compounds and Fatty Acids as Carbon Sources | Q38600992 | ||
| Multiple factors independently regulate hilA and invasion gene expression in Salmonella enterica serovar typhimurium | Q39538569 | ||
| Integrating global regulatory input into the Salmonella pathogenicity island 1 type III secretion system | Q39694438 | ||
| Mechanism for inhibition of Vibrio cholerae ToxT activity by the unsaturated fatty acid components of bile. | Q39768256 | ||
| TolC, but not AcrB, is involved in the invasiveness of multidrug-resistant Salmonella enterica serovar Typhimurium by increasing type III secretion system-1 expression | Q40013537 | ||
| Lipid Digestion and Absorption | Q40148842 | ||
| HilD, HilC and RtsA constitute a feed forward loop that controls expression of the SPI1 type three secretion system regulator hilA in Salmonella enterica serovar Typhimurium. | Q40402350 | ||
| Medium-chain fatty acids decrease colonization and invasion through hilA suppression shortly after infection of chickens with Salmonella enterica serovar Enteritidis | Q40548073 | ||
| ppGpp-dependent stationary phase induction of genes on Salmonella pathogenicity island 1. | Q40552048 | ||
| Intestinal short-chain fatty acids alter Salmonella typhimurium invasion gene expression and virulence through BarA/SirA. | Q40687509 | ||
| Respective roles of culturable and viable-but-nonculturable cells in the heterogeneity of Salmonella enterica serovar typhimurium invasiveness | Q41990605 | ||
| hilA is a novel ompR/toxR family member that activates the expression of Salmonella typhimurium invasion genes | Q42638991 | ||
| Effect of fatty acids and cholesterol present in bile on expression of virulence factors and motility of Vibrio cholerae | Q42800298 | ||
| Unsaturated long chain free fatty acids are input signals of the Salmonella enterica PhoP/PhoQ regulatory system | Q42905654 | ||
| P433 | issue | 1 | |
| P921 | main subject | fatty acid | Q61476 |
| P304 | page(s) | e02170-15 | |
| P577 | publication date | 2015-01-01 | |
| P1433 | published in | mBio | Q15817061 |
| P1476 | title | Intestinal Long-Chain Fatty Acids Act as a Direct Signal To Modulate Expression of the Salmonella Pathogenicity Island 1 Type III Secretion System | |
| P478 | volume | 7 |
| Q37717323 | A new class of inhibitors of the AraC family virulence regulator Vibrio cholerae ToxT. |
| Q57023074 | Contribution of SPI-1 bistability to Salmonella enterica cooperative virulence: insights from single cell analysis |
| Q92886841 | Establishment of an induced memory response in Pseudomonas aeruginosa during infection of a eukaryotic host |
| Q40070115 | Exogenous Polyunsaturated Fatty Acids Impact Membrane Remodeling and Affect Virulence Phenotypes among Pathogenic Vibrio Species |
| Q52559475 | Exogenous fatty acids alter phospholipid composition, membrane permeability, capacity for biofilm formation, and antimicrobial peptide susceptibility in Klebsiella pneumoniae. |
| Q33585337 | Gre factors-mediated control of hilD transcription is essential for the invasion of epithelial cells by Salmonella enterica serovar Typhimurium. |
| Q93106825 | HilD induces expression of a novel Salmonella Typhimurium invasion factor, YobH, through a regulatory cascade involving SprB |
| Q89636193 | HilD, HilC, and RtsA Form Homodimers and Heterodimers to Regulate Expression of the Salmonella Pathogenicity Island I Type III Secretion System |
| Q49597228 | HilE Regulates HilD by Blocking DNA Binding in Salmonella enterica serovar Typhimurium |
| Q57811561 | Host lipid sensing promotes invasion of cells with pathogenic Salmonella |
| Q47916010 | How can naturally occurring fatty acids neutralize Listeria? |
| Q31144688 | In silico clustering of Salmonella global gene expression data reveals novel genes co-regulated with the SPI-1 virulence genes through HilD. |
| Q91580928 | Mechanisms of Salmonella pathogenesis in animal models |
| Q93366073 | Oxygen-dependent regulation of SPI1 type three secretion system by small RNAs in Salmonella enterica serovar Typhimurium |
| Q38639808 | Pan-genome Analyses of the Species Salmonella enterica, and Identification of Genomic Markers Predictive for Species, Subspecies, and Serovar |
| Q92641059 | PhoP-Mediated Repression of the SPI1 Type 3 Secretion System in Salmonella enterica Serovar Typhimurium |
| Q39131951 | Post-transcriptional regulation of type III secretion in plant and animal pathogens. |
| Q61805343 | Production of long-chain free fatty acids from metabolically engineered Rhodobacter sphaeroides heterologously producing periplasmic phospholipase A2 in dodecane-overlaid two-phase culture |
| Q57805501 | Redox, amino acid, and fatty acid metabolism intersect with bacterial virulence in the gut |
| Q90701291 | Salmonella enterica Requires Lipid Metabolism Genes To Replicate in Proinflammatory Macrophages and Mice |
| Q42694341 | Salmonella enterica Serovar Typhimurium Strategies for Host Adaptation |
| Q64095408 | Salmonella invasion is controlled through the secondary structure of the hilD transcript |
| Q33811877 | Signal transduction pathway mediated by the novel regulator LoiA for low oxygen tension induced Salmonella Typhimurium invasion |
| Q48232558 | The Biochemistry of Sensing: Enteric Pathogens Regulate Type III Secretion in Response to Environmental and Host Cues. |
| Q88043965 | The Hcp-like protein HilE inhibits homodimerization and DNA binding of the virulence-associated transcriptional regulator HilD in Salmonella |
| Q64082768 | Using Bacterial Transcriptomics to Investigate Targets of Host-Bacterial Interactions in Caenorhabditis elegans |
Search more.