scholarly article | Q13442814 |
review article | Q7318358 |
P50 | author | Srikanta Dash | Q96901165 |
Srinivas Chava | Q114734960 | ||
Luis A Balart | Q114734962 | ||
Pauline Ferraris | Q30089820 | ||
Partha K Chandra | Q56477437 | ||
Robert F. Garry | Q66732989 | ||
Yucel Aydin | Q90228663 | ||
Weina Chen | Q96231626 | ||
P2093 | author name string | Tong Wu | |
P2860 | cites work | Complex formation of the nonstructural protein 3 of hepatitis C virus with the p53 tumor suppressor | Q22008046 |
ER stress induces cleavage of membrane-bound ATF6 by the same proteases that process SREBPs | Q24290776 | ||
Two Beclin 1-binding proteins, Atg14L and Rubicon, reciprocally regulate autophagy at different stages | Q24318814 | ||
A receptor for the selective uptake and degradation of proteins by lysosomes | Q24319797 | ||
Signal peptidase complex subunit 1 participates in the assembly of hepatitis C virus through an interaction with E2 and NS2 | Q24322876 | ||
NS3 protein of Hepatitis C virus associates with the tumour suppressor p53 and inhibits its function in an NS3 sequence-dependent manner | Q24337056 | ||
Innate immune recognition of viral infection | Q24498364 | ||
Hepatitis C virus subgenomic replicons induce endoplasmic reticulum stress activating an intracellular signaling pathway | Q24537844 | ||
Activation of chaperone-mediated autophagy during oxidative stress | Q24562065 | ||
Mammalian autophagy: core molecular machinery and signaling regulation | Q24595447 | ||
Identification of three interferon-inducible cellular enzymes that inhibit the replication of hepatitis C virus | Q24658111 | ||
Autophagy in hepatocellular carcinomas: from pathophysiology to therapeutic response | Q26767245 | ||
Chronic hepatitis C virus infection and lipoprotein metabolism | Q26784234 | ||
The different roles of selective autophagic protein degradation in mammalian cells | Q26785503 | ||
Microbial Regulation of p53 Tumor Suppressor | Q26785743 | ||
Decoding cell death signals in liver inflammation | Q26823299 | ||
HCV induces the expression of Rubicon and UVRAG to temporally regulate the maturation of autophagosomes and viral replication | Q35586232 | ||
Chaperone-Mediated Autophagy Targets IFNAR1 for Lysosomal Degradation in Free Fatty Acid Treated HCV Cell Culture | Q35626712 | ||
Apoptosis: the nexus of liver injury and fibrosis. | Q35651666 | ||
Immunomodulatory functions of type I interferons | Q26823904 | ||
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From non-A, non-B hepatitis to hepatitis C virus cure | Q26864968 | ||
Modulation of the unfolded protein response by the human hepatitis B virus | Q26999215 | ||
Role of innate immunity in the development of hepatocellular carcinoma | Q27024780 | ||
Human cytomegalovirus infection activates and regulates the unfolded protein response | Q27469611 | ||
Hepatitis C Virus Genotype 1a Growth and Induction of Autophagy | Q27485449 | ||
Induction of incomplete autophagic response by hepatitis C virus via the unfolded protein response | Q27487012 | ||
HCV Induces Oxidative and ER Stress, and Sensitizes Infected Cells to Apoptosis in SCID/Alb-uPA Mice | Q27487963 | ||
Autophagy, Immunity, and Microbial Adaptations | Q27489075 | ||
The autophagy machinery is required to initiate hepatitis C virus replication | Q27489377 | ||
Eating the enemy within: autophagy in infectious diseases | Q27489453 | ||
Cellular stress response and innate immune signaling: integrating pathways in host defense and inflammation. | Q27692108 | ||
Signal integration in the endoplasmic reticulum unfolded protein response | Q27860577 | ||
Global epidemiology of hepatitis C virus infection | Q27860799 | ||
Autophagy fights disease through cellular self-digestion | Q27860902 | ||
Getting membrane proteins on and off the shuttle bus between the endoplasmic reticulum and the Golgi complex | Q28066509 | ||
IRE1 couples endoplasmic reticulum load to secretory capacity by processing the XBP-1 mRNA | Q28214814 | ||
HCV causes chronic endoplasmic reticulum stress leading to adaptation and interference with the unfolded protein response | Q28477137 | ||
Hepatitis C virus induces the mitochondrial translocation of Parkin and subsequent mitophagy | Q28489090 | ||
Regulated translation initiation controls stress-induced gene expression in mammalian cells | Q28506388 | ||
Regulation of hepatic lipogenesis by the transcription factor XBP1 | Q28507784 | ||
Hepatitis C virus NS5A protein binds TBP and p53, inhibiting their DNA binding and p53 interactions with TBP and ERCC3 | Q28569202 | ||
The unfolded protein response: from stress pathway to homeostatic regulation | Q29547396 | ||
Regulation mechanisms and signaling pathways of autophagy | Q29547416 | ||
Live or let die: the cell's response to p53 | Q29547663 | ||
The role of the Atg1/ULK1 complex in autophagy regulation | Q29614497 | ||
A role for ubiquitin in selective autophagy | Q29614499 | ||
Mediators of endoplasmic reticulum stress-induced apoptosis | Q29615496 | ||
The unfolded protein response: controlling cell fate decisions under ER stress and beyond | Q29615499 | ||
mTOR regulation of autophagy | Q29616301 | ||
Cell death and endoplasmic reticulum stress: disease relevance and therapeutic opportunities | Q29617768 | ||
Epidemiology of viral hepatitis and hepatocellular carcinoma | Q29617918 | ||
Quality control in the endoplasmic reticulum | Q29619929 | ||
The global burden of hepatitis C | Q29620138 | ||
Peptide sequences that target proteins for enhanced degradation during serum withdrawal | Q68328388 | ||
Acetaminophen induces ER dependent signaling in mouse liver | Q79505704 | ||
That which does not kill me makes me stronger: adapting to chronic ER stress | Q81405125 | ||
Hyperhomocysteinemia, endoplasmic reticulum stress, and alcoholic liver injury | Q35799626 | ||
Knockdown of autophagy enhances the innate immune response in hepatitis C virus-infected hepatocytes | Q35910776 | ||
Antigen processing by macroautophagy for MHC presentation | Q35927580 | ||
Chikungunya virus-induced autophagy delays caspase-dependent cell death | Q35946144 | ||
Replication of hepatitis C virus RNA on autophagosomal membranes | Q36002897 | ||
Endoplasmic reticulum stress in liver disease | Q36031838 | ||
Chronic inflammation, immune escape, and oncogenesis in the liver: a unique neighborhood for novel intersections | Q36055039 | ||
Hepatitis C virus upregulates Beclin1 for induction of autophagy and activates mTOR signaling. | Q36172124 | ||
Hepatitis C virus-induced autophagy is independent of the unfolded protein response | Q36276577 | ||
The LMP1 oncogene of EBV activates PERK and the unfolded protein response to drive its own synthesis | Q36445900 | ||
Emerging roles of interferon-stimulated genes in the innate immune response to hepatitis C virus infection | Q36460968 | ||
Autophagy hijacked through viroporin-activated calcium/calmodulin-dependent kinase kinase-β signaling is required for rotavirus replication | Q36483693 | ||
Role of the unfolded protein response, GRP78 and GRP94 in organ homeostasis. | Q36497621 | ||
ER stress: Autophagy induction, inhibition and selection. | Q36777229 | ||
Virus-specific mechanisms of carcinogenesis in hepatitis C virus associated liver cancer | Q36812745 | ||
Modification of intracellular membrane structures for virus replication | Q37139313 | ||
Innate immune modulation by RNA viruses: emerging insights from functional genomics | Q37226509 | ||
The adaptive endoplasmic reticulum stress response to lipotoxicity in progressive human nonalcoholic fatty liver disease | Q37412003 | ||
HCV infection selectively impairs type I but not type III IFN signaling | Q37415444 | ||
Toll-like receptors in control of immunological autophagy. | Q37460134 | ||
The ins and outs of hepatitis C virus entry and assembly | Q37498626 | ||
Autophagic response in the Rabbit Hemorrhagic Disease, an animal model of virally-induced fulminant hepatic failure | Q37580627 | ||
Activity of hexokinase is increased by its interaction with hepatitis C virus protein NS5A. | Q37643752 | ||
Divergent roles of autophagy in virus infection | Q37679218 | ||
Understanding the hepatitis C virus life cycle paves the way for highly effective therapies. | Q37699395 | ||
A new laboratory-based algorithm to predict development of hepatocellular carcinoma in patients with hepatitis C and cirrhosis | Q37710955 | ||
Abnormal structure and expression of p53 gene in human hepatocellular carcinoma | Q37714419 | ||
MHC presentation via autophagy and how viruses escape from it | Q37790648 | ||
Assembly of infectious hepatitis C virus particles | Q37819269 | ||
Autophagy as a target for anticancer therapy | Q37876679 | ||
Dengue virus and autophagy | Q37945240 | ||
Autophagy in regulation of Toll-like receptor signaling | Q37983945 | ||
Living in the liver: hepatic infections | Q37987860 | ||
Genome guardian p53 and viral infections | Q38068825 | ||
Hepatitis C virus proteins: from structure to function | Q38086751 | ||
West Nile virus differentially modulates the unfolded protein response to facilitate replication and immune evasion | Q41971863 | ||
Hepatitis C virus core protein activates autophagy through EIF2AK3 and ATF6 UPR pathway-mediated MAP1LC3B and ATG12 expression | Q41981056 | ||
Autophagy is an essential component of Drosophila immunity against vesicular stomatitis virus | Q42169591 | ||
Simeprevir plus sofosbuvir, with or without ribavirin, to treat chronic infection with hepatitis C virus genotype 1 in non-responders to pegylated interferon and ribavirin and treatment-naive patients: the COSMOS randomised study | Q42202756 | ||
All-oral daclatasvir plus asunaprevir for hepatitis C virus genotype 1b: a multinational, phase 3, multicohort study | Q42202767 | ||
Virus-induced unfolded protein response attenuates antiviral defenses via phosphorylation-dependent degradation of the type I interferon receptor | Q42551996 | ||
Randomized trial of peginterferon alfa-2b and ribavirin for 48 or 72 weeks in patients with hepatitis C virus genotype 1 and slow virologic response | Q42670765 | ||
p53-dependent transcriptional repression of p21(waf1) by hepatitis C virus NS3. | Q42826294 | ||
Hepatitis C virus NS5A protein modulates cell cycle regulatory genes and promotes cell growth | Q42993783 | ||
Activation of endoplasmic reticulum stress response by hepatitis viruses up-regulates protein phosphatase 2A. | Q43037981 | ||
In vivo hepatic endoplasmic reticulum stress in patients with chronic hepatitis C. | Q44332232 | ||
Endoplasmic reticulum stress induces hepatic steatosis via increased expression of the hepatic very low-density lipoprotein receptor | Q45200756 | ||
No evidence of the unfolded protein response in patients with chronic hepatitis C virus infection | Q45369635 | ||
Correction: SH2 Modified STAT1 Induces HLA-I Expression and Improves IFN-γ Signaling in IFN-α Resistant HCV Replicon Cells. | Q45766026 | ||
Hepatitis C virus RNA translation | Q38086752 | ||
Virion assembly and release | Q38086754 | ||
Emerging regulation and functions of autophagy | Q38118567 | ||
The autophagosome: origins unknown, biogenesis complex | Q38160660 | ||
Immune dysfunction in cirrhosis | Q38195835 | ||
Glucose-regulated proteins in cancer: molecular mechanisms and therapeutic potential | Q38198491 | ||
A new paradigm: innate immune sensing of viruses via the unfolded protein response | Q38218106 | ||
Unfolded protein response in hepatitis C virus infection | Q38218108 | ||
The impact of the endoplasmic reticulum protein-folding environment on cancer development | Q38241996 | ||
Endoplasmic reticulum stress is the crossroads of autophagy, inflammation, and apoptosis signaling pathways and participates in liver fibrosis | Q38257120 | ||
Persistent hepatitis C virus infection impairs ribavirin antiviral activity through clathrin-mediated trafficking of equilibrative nucleoside transporter 1. | Q38262107 | ||
Virology and cell biology of the hepatitis C virus life cycle: an update | Q38274457 | ||
Autophagosome formation during varicella-zoster virus infection following endoplasmic reticulum stress and the unfolded protein response | Q38286153 | ||
Hepatitis C virus RNA replication and assembly: living on the fat of the land | Q38294573 | ||
Hepatitis C virus core protein enhances p53 function through augmentation of DNA binding affinity and transcriptional ability | Q38309880 | ||
Single-point mutations of hepatitis C virus NS3 that impair p53 interaction and anti-apoptotic activity of NS3. | Q38317301 | ||
Complexes containing activating transcription factor (ATF)/cAMP-responsive-element-binding protein (CREB) interact with the CCAAT/enhancer-binding protein (C/EBP)-ATF composite site to regulate Gadd153 expression during the stress response | Q38326929 | ||
Endoplasmic reticulum stress, genome damage, and cancer. | Q38359856 | ||
Cross talk between ER stress, oxidative stress, and inflammation in health and disease | Q38389425 | ||
Cellular stress responses in hepatitis C virus infection: Mastering a two-edged sword | Q38402247 | ||
The evolving paradigm of cell-nonautonomous UPR-based regulation of immunity by cancer cells | Q38429699 | ||
Unfolded protein response pathways regulate Hepatitis C virus replication via modulation of autophagy | Q39197168 | ||
Hepatitis C virus inhibits AKT-tuberous sclerosis complex (TSC), the mechanistic target of rapamycin (MTOR) pathway, through endoplasmic reticulum stress to induce autophagy | Q39241528 | ||
Autophagy protects cells from HCV-induced defects in lipid metabolism | Q39430882 | ||
Dysfunction of autophagy participates in vacuole formation and cell death in cells replicating hepatitis C virus | Q39458949 | ||
Mechanism of HCV's resistance to IFN-α in cell culture involves expression of functional IFN-α receptor 1. | Q39506888 | ||
Cell culture and in vivo analyses of cytopathic hepatitis C virus mutants | Q39683340 | ||
Knockdown of autophagy-related gene decreases the production of infectious hepatitis C virus particles | Q39821948 | ||
Hepatitis B virus X protein (HBx) activates ATF6 and IRE1-XBP1 pathways of unfolded protein response | Q40210691 | ||
The role of BiP in endoplasmic reticulum-associated degradation of major histocompatibility complex class I heavy chain induced by cytomegalovirus proteins. | Q40274580 | ||
Alpha interferon inhibits translation mediated by the internal ribosome entry site of six different hepatitis C virus genotypes | Q40361695 | ||
Interferons alpha, beta, gamma each inhibit hepatitis C virus replication at the level of internal ribosome entry site-mediated translation | Q40418675 | ||
Modulation of p53 transcription regulatory activity and post-translational modification by hepatitis C virus core protein | Q40589165 | ||
Hepatitis C virus suppresses the IRE1-XBP1 pathway of the unfolded protein response | Q40590374 | ||
DOT1L Activity Promotes Proliferation and Protects Cortical Neural Stem Cells from Activation of ATF4-DDIT3-Mediated ER Stress In Vitro | Q40615754 | ||
HCV NS5A interacts with p53 and inhibits p53-mediated apoptosis | Q40720204 | ||
Activation of the promoters of genes associated with DNA damage, oxidative stress, ER stress and protein malfolding by the bile salt, deoxycholate | Q40931435 | ||
Hepatitis C virus core protein represses p21WAF1/Cip1/Sid1 promoter activity | Q41055268 | ||
The dysregulation of endoplasmic reticulum stress response in acute-on-chronic liver failure patients caused by acute exacerbation of chronic hepatitis B. | Q41076537 | ||
Activation of p53 tumor suppressor by hepatitis C virus core protein | Q41699712 | ||
When autophagy meets cancer through p62/SQSTM1. | Q41853953 | ||
Hepatitis C virus infection promotes hepatic gluconeogenesis through an NS5A-mediated, FoxO1-dependent pathway | Q30395512 | ||
Hepatitis C virus triggers mitochondrial fission and attenuates apoptosis to promote viral persistence | Q33674227 | ||
Herpes simplex virus 1 infection activates the endoplasmic reticulum resident kinase PERK and mediates eIF-2alpha dephosphorylation by the gamma(1)34.5 protein | Q33717136 | ||
SH2 modified STAT1 induces HLA-I expression and improves IFN-γ signaling in IFN-α resistant HCV replicon cells | Q33719537 | ||
Hepatitis C virus NS5A physically associates with p53 and regulates p21/waf1 gene expression in a p53-dependent manner | Q33836415 | ||
Integration of interferon-alpha/beta signalling to p53 responses in tumour suppression and antiviral defence. | Q33967257 | ||
ER stress, autophagy, and RNA viruses | Q34003896 | ||
Autophagy protein ATG5 interacts transiently with the hepatitis C virus RNA polymerase (NS5B) early during infection | Q34078007 | ||
Human immunodeficiency virus-1 inhibition of immunoamphisomes in dendritic cells impairs early innate and adaptive immune responses | Q34089158 | ||
Persistent expression of hepatitis C virus non-structural proteins leads to increased autophagy and mitochondrial injury in human hepatoma cells | Q34099283 | ||
IRGM is a common target of RNA viruses that subvert the autophagy network. | Q34102728 | ||
Hepatocyte Death: A Clear and Present Danger | Q34147508 | ||
Chaperone-mediated autophagy in protein quality control | Q34150995 | ||
Activation of the ATF6, XBP1 and grp78 genes in human hepatocellular carcinoma: a possible involvement of the ER stress pathway in hepatocarcinogenesis | Q34192744 | ||
Peptide sequences that target cytosolic proteins for lysosomal proteolysis | Q34229121 | ||
Intracellular expression of IRF9 Stat fusion protein overcomes the defective Jak-Stat signaling and inhibits HCV RNA replication | Q34243927 | ||
Regulation of p53 localization | Q34254613 | ||
Impaired expression of type I and type II interferon receptors in HCV-associated chronic liver disease and liver cirrhosis | Q34271143 | ||
Amino acid signalling upstream of mTOR | Q34325138 | ||
Mechanisms of HCV-induced liver cancer: what did we learn from in vitro and animal studies? | Q34358742 | ||
Structure and function of the p53 tumor suppressor gene: clues for rational cancer therapeutic strategies | Q34400583 | ||
Activation of the unfolded protein response and autophagy after hepatitis C virus infection suppresses innate antiviral immunity in vitro | Q34428880 | ||
Global distribution and prevalence of hepatitis C virus genotypes | Q34431175 | ||
Cytoprotective gene bi-1 is required for intrinsic protection from endoplasmic reticulum stress and ischemia-reperfusion injury | Q34479417 | ||
Viruses, endoplasmic reticulum stress, and interferon responses | Q34482331 | ||
Role of the host cell's unfolded protein response in arenavirus infection. | Q34529827 | ||
IL28B inhibits hepatitis C virus replication through the JAK-STAT pathway | Q34744565 | ||
The contribution of endoplasmic reticulum stress to liver diseases | Q34860549 | ||
Interferon and ribavirin combination treatment synergistically inhibit HCV internal ribosome entry site mediated translation at the level of polyribosome formation | Q34979630 | ||
Nutrient-sensing mechanisms and pathways. | Q35036374 | ||
Changes in autophagic response in patients with chronic hepatitis C virus infection | Q35071429 | ||
Temporal trends of nonalcoholic fatty liver disease-related hepatocellular carcinoma in the veteran affairs population | Q35096951 | ||
Decision making by p53: life, death and cancer | Q35116663 | ||
Rab5 and class III phosphoinositide 3-kinase Vps34 are involved in hepatitis C virus NS4B-induced autophagy. | Q35274953 | ||
P275 | copyright license | Creative Commons Attribution | Q6905323 |
P6216 | copyright status | copyrighted | Q50423863 |
P433 | issue | 5 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | hepatitis C | Q154869 |
autophagy | Q288322 | ||
Hepatitis C virus | Q708693 | ||
Hepacivirus | Q2082778 | ||
viral genome replication | Q3933202 | ||
host-pathogen interaction | Q5909198 | ||
endoplasmic reticulum unfolded protein response | Q14633900 | ||
endoplasmic reticulum stress | Q66305937 | ||
P5008 | on focus list of Wikimedia project | ScienceSource | Q55439927 |
P304 | page(s) | 150 | |
P577 | publication date | 2016-05-23 | |
P1433 | published in | Viruses | Q7935305 |
P1476 | title | Hepatitis C Virus Infection Induces Autophagy as a Prosurvival Mechanism to Alleviate Hepatic ER-Stress Response | |
P478 | volume | 8 |
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Q54253029 | Autophagy during viral infection - a double-edged sword. |
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Q39037205 | Autophagy-from molecular mechanisms to clinical relevance. |
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