Point mutations in the proline-rich region of p22phox are dominant inhibitors of Nox1- and Nox2-dependent reactive oxygen generation.

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Point mutations in the proline-rich region of p22phox are dominant inhibitors of Nox1- and Nox2-dependent reactive oxygen generation. is …
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scholarly articleQ13442814

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P356DOI10.1074/JBC.M501882200
P698PubMed publication ID15994299

P50authorGuangjie ChengQ50636884
P2093author name stringJ David Lambeth
Darren Ritsick
Tsukasa Kawahara
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NOX enzymes and the biology of reactive oxygenQ29547517
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Assembly and activation of the phagocyte NADPH oxidase. Specific interaction of the N-terminal Src homology 3 domain of p47phox with p22phox is required for activation of the NADPH oxidaseQ30176829
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The NADPH oxidase Nox3 constitutively produces superoxide in a p22phox-dependent manner: its regulation by oxidase organizers and activatorsQ33213948
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Ultrastructural localization of cytochrome b in the membranes of resting and phagocytosing human granulocytesQ34245500
Mechanism of Ca2+ activation of the NADPH oxidase 5 (NOX5).Q34300862
Nox3 regulation by NOXO1, p47phox, and p67phox.Q34324985
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Reactive oxygen species in the vasculature: molecular and cellular mechanismsQ35569201
The importance of redox regulated pathways in sperm cell biology.Q35756574
Deficiency of NADPH Oxidase Components p47phox and gp91phox Caused Granulomatous Synovitis and Increased Connective Tissue Destruction in Experimental Arthritis ModelsQ35792117
Structure and regulation of the neutrophil respiratory burst oxidase: comparison with nonphagocyte oxidasesQ35829421
Molecular events associated with reactive oxygen species and cell cycle progression in mammalian cellsQ35846749
P433issue36
P407language of work or nameEnglishQ1860
P1104number of pages11
P304page(s)31859-31869
P577publication date2005-06-30
P1433published inJournal of Biological ChemistryQ867727
P1476titlePoint mutations in the proline-rich region of p22phox are dominant inhibitors of Nox1- and Nox2-dependent reactive oxygen generation
P478volume280

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