| scholarly article | Q13442814 |
| P819 | ADS bibcode | 2008PNAS..10518907D |
| P356 | DOI | 10.1073/PNAS.0810111105 |
| P932 | PMC publication ID | 2585942 |
| P698 | PubMed publication ID | 19028876 |
| P5875 | ResearchGate publication ID | 23492775 |
| P2093 | author name string | Jessica L Goodman | |
| L Charles Murtaugh | |||
| Lyska L Emerson | |||
| Jean-Paul De La O | |||
| Andrew B Curtis | |||
| Benjamin E Illum | |||
| Scott C Froebe | |||
| P2860 | cites work | Notch signaling controls multiple steps of pancreatic differentiation | Q24569636 |
| Beta-catenin is essential for pancreatic acinar but not islet development | Q28512693 | ||
| Direct evidence for the pancreatic lineage: NGN3+ cells are islet progenitors and are distinct from duct progenitors | Q29619012 | ||
| Preinvasive and invasive ductal pancreatic cancer and its early detection in the mouse | Q29620396 | ||
| Pancreatic cancer. | Q30490247 | ||
| A PCR primer bank for quantitative gene expression analysis | Q33447710 | ||
| Gene regulatory factors in pancreatic development | Q34286940 | ||
| Activated Kras and Ink4a/Arf deficiency cooperate to produce metastatic pancreatic ductal adenocarcinoma | Q34642090 | ||
| The Nestin progenitor lineage is the compartment of origin for pancreatic intraepithelial neoplasia | Q35663506 | ||
| Acinar cells contribute to the molecular heterogeneity of pancreatic intraepithelial neoplasia | Q35928158 | ||
| The love-hate relationship between Ras and Notch. | Q36231474 | ||
| Pathology of genetically engineered mouse models of pancreatic exocrine cancer: consensus report and recommendations. | Q36362278 | ||
| In vivo lineage tracing defines the role of acinar-to-ductal transdifferentiation in inflammatory ductal metaplasia | Q36478584 | ||
| Notch signaling is required for exocrine regeneration after acute pancreatitis. | Q40017803 | ||
| Analysis of lung tumor initiation and progression using conditional expression of oncogenic K-ras | Q40424687 | ||
| Tumor induction by an endogenous K-ras oncogene is highly dependent on cellular context | Q40637553 | ||
| Increased expression of hypoxia-inducible factor-1alpha, p48, and the Notch signaling cascade during acute pancreatitis in mice | Q44715476 | ||
| Recapitulation of elements of embryonic development in adult mouse pancreatic regeneration | Q46376132 | ||
| Pten constrains centroacinar cell expansion and malignant transformation in the pancreas | Q46708964 | ||
| Preinvasive pancreatic neoplasia of ductal phenotype induced by acinar cell targeting of mutant Kras in transgenic mice | Q53379866 | ||
| The mutant K-ras oncogene causes pancreatic periductal lymphocytic infiltration and gastric mucous neck cell hyperplasia in transgenic mice | Q53379869 | ||
| Chronic Pancreatitis Is Essential for Induction of Pancreatic Ductal Adenocarcinoma by K-Ras Oncogenes in Adult Mice | Q61776577 | ||
| Isolation of RNA using guanidinium salts | Q69005967 | ||
| Notch mediates TGF alpha-induced changes in epithelial differentiation during pancreatic tumorigenesis | Q73614651 | ||
| Activation of Notch-1 signaling maintains the neoplastic phenotype in human Ras-transformed cells | Q74624551 | ||
| Pancreatic epithelial plasticity mediated by acinar cell transdifferentiation and generation of nestin-positive intermediates | Q80378426 | ||
| Mist1-KrasG12D knock-in mice develop mixed differentiation metastatic exocrine pancreatic carcinoma and hepatocellular carcinoma | Q82193149 | ||
| P4510 | describes a project that uses | ImageJ | Q1659584 |
| P433 | issue | 48 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | intraepithelial neoplasia | Q6058399 |
| P304 | page(s) | 18907-18912 | |
| P577 | publication date | 2008-11-21 | |
| P1433 | published in | Proceedings of the National Academy of Sciences of the United States of America | Q1146531 |
| P1476 | title | Notch and Kras reprogram pancreatic acinar cells to ductal intraepithelial neoplasia | |
| P478 | volume | 105 |
| Q28082890 | A Revised Classification System and Recommendations From the Baltimore Consensus Meeting for Neoplastic Precursor Lesions in the Pancreas |
| Q38852413 | A bright future for protein kinase D1 as a drug target to prevent or treat pancreatic cancer |
| Q91711463 | ATDC is required for the initiation of KRAS-induced pancreatic tumorigenesis |
| Q38922230 | Acinar cell plasticity and development of pancreatic ductal adenocarcinoma |
| Q38459854 | Acinar cell reprogramming: a clinically important target in pancreatic disease |
| Q42282476 | Acinar cells in the neonatal pancreas grow by self-duplication and not by neogenesis from duct cells |
| Q82181907 | Acinar‐to‐ductal metaplasia accompanies c‐myc‐induced exocrine pancreatic cancer progression in transgenic rodents |
| Q38461863 | Activated Notch1 induces lung adenomas in mice and cooperates with Myc in the generation of lung adenocarcinoma. |
| Q54526478 | Activation of protein kinase Cδ leads to increased pancreatic acinar cell dedifferentiation in the absence of MIST1 |
| Q27312357 | Acute pancreatitis accelerates initiation and progression to pancreatic cancer in mice expressing oncogenic Kras in the nestin cell lineage |
| Q34082522 | Acute pancreatitis markedly accelerates pancreatic cancer progression in mice expressing oncogenic Kras |
| Q34046479 | Adult cell plasticity in vivo: de-differentiation and transdifferentiation are back in style |
| Q90240762 | Aldh1b1 expression defines progenitor cells in the adult pancreas and is required for Kras-induced pancreatic cancer |
| Q37628092 | Analysis of the tumor-initiating and metastatic capacity of PDX1-positive cells from the adult pancreas |
| Q89285906 | Are Gastric and Esophageal Metaplasia Relatives? The Case for Barrett's Stemming from SPEM |
| Q35176052 | Aspartate β-Hydroxylase expression promotes a malignant pancreatic cellular phenotype |
| Q33832727 | Basal but not luminal mammary epithelial cells require PI3K/mTOR signaling for Ras-driven overgrowth. |
| Q33604913 | Beta-catenin blocks Kras-dependent reprogramming of acini into pancreatic cancer precursor lesions in mice |
| Q37182612 | Bmi1 lineage tracing identifies a self-renewing pancreatic acinar cell subpopulation capable of maintaining pancreatic organ homeostasis |
| Q35228755 | Brg1 promotes both tumor-suppressive and oncogenic activities at distinct stages of pancreatic cancer formation |
| Q36823886 | Cell Fate Decisions During Breast Cancer Development |
| Q33767473 | Cell state plasticity, stem cells, EMT, and the generation of intra-tumoral heterogeneity |
| Q29619557 | Cells of origin in cancer |
| Q39161835 | Cells of origin of pancreatic neoplasms |
| Q39379039 | Cellular Prion Protein Mediates Pancreatic Cancer Cell Survival and Invasion through Association with and Enhanced Signaling of Notch1. |
| Q36096239 | Cellular features of senescence during the evolution of human and murine ductal pancreatic cancer |
| Q37709553 | Cellular plasticity within the pancreas--lessons learned from development |
| Q37411831 | Challenges and advances in mouse modeling for human pancreatic tumorigenesis and metastasis |
| Q91526219 | Characterization of genetic subclonal evolution in pancreatic cancer mouse models |
| Q36929317 | Characterization of transgenic mice expressing cancer-associated variants of human NOTCH1 |
| Q39051424 | Combined inhibition of Notch and JAK/STAT is superior to monotherapies and impairs pancreatic cancer progression |
| Q34620985 | Context-dependent transformation of adult pancreatic cells by oncogenic K-Ras |
| Q27021239 | Control of Cell Identity in Pancreas Development and Regeneration |
| Q35578568 | Crosstalk between the canonical NF-κB and Notch signaling pathways inhibits Pparγ expression and promotes pancreatic cancer progression in mice |
| Q34201623 | Cyclin-dependent kinase inhibitor Dinaciclib (SCH727965) inhibits pancreatic cancer growth and progression in murine xenograft models |
| Q41906889 | DCLK1 marks a morphologically distinct subpopulation of cells with stem cell properties in preinvasive pancreatic cancer |
| Q42826326 | DYRK1B-dependent autocrine-to-paracrine shift of Hedgehog signaling by mutant RAS. |
| Q40003054 | Dclk1 Defines Quiescent Pancreatic Progenitors that Promote Injury-Induced Regeneration and Tumorigenesis |
| Q35622126 | Deploying mouse models of pancreatic cancer for chemoprevention studies |
| Q26774942 | Developmental Pathways Direct Pancreatic Cancer Initiation from Its Cellular Origin |
| Q35432049 | Dicer is required for maintenance of adult pancreatic acinar cell identity and plays a role in Kras-driven pancreatic neoplasia |
| Q35161464 | Dicer regulates differentiation and viability during mouse pancreatic cancer initiation |
| Q39278421 | Differential expression of Notch1 intracellular domain and p21 proteins, and their clinical significance in gastric cancer |
| Q39384816 | Differentiation and Inflammation: 'Best Enemies' in Gastrointestinal Carcinogenesis |
| Q36673216 | Direct reprogramming by oncogenic Ras and Myc. |
| Q41845030 | Disputed Paternity: The Uncertain Ancestry of Pancreatic Ductal Neoplasia |
| Q90139280 | Ductal vs. acinar? Recent insights into identifying cell lineage of pancreatic ductal adenocarcinoma |
| Q55616605 | E47 Governs the MYC-CDKN1B/p27KIP1-RB Network to Growth Arrest PDA Cells Independent of CDKN2A/p16INK4A and Wild-Type p53. |
| Q89151859 | EMT Subtype Influences Epithelial Plasticity and Mode of Cell Migration |
| Q47281933 | EVI1 modulates oncogenic role of GPC1 in pancreatic carcinogenesis |
| Q38314944 | EVI1 oncogene promotes KRAS pathway through suppression of microRNA-96 in pancreatic carcinogenesis |
| Q38673285 | EWS/FLI is a Master Regulator of Metabolic Reprogramming in Ewing Sarcoma |
| Q33818230 | Epithelial Tissues Have Varying Degrees of Susceptibility to KrasG12D-Initiated Tumorigenesis in a Mouse Model |
| Q34456195 | Epithelial mesenchymal transition and pancreatic tumor initiating CD44+/EpCAM+ cells are inhibited by γ-secretase inhibitor IX. |
| Q47121552 | Epithelial-Myeloid cell crosstalk regulates acinar cell plasticity and pancreatic remodeling in mice. |
| Q38022404 | Exocrine ontogenies: on the development of pancreatic acinar, ductal and centroacinar cells |
| Q28482954 | Exocrine pancreatic carcinogenesis and autotaxin expression |
| Q30422901 | Focused ultrasound-mediated drug delivery to pancreatic cancer in a mouse model. |
| Q27004534 | From fly wings to targeted cancer therapies: a centennial for notch signaling |
| Q37821995 | GI GEMs: Genetically Engineered Mouse Models of Gastrointestinal Disease |
| Q30620697 | Galectin-1 drives pancreatic carcinogenesis through stroma remodeling and Hedgehog signaling activation |
| Q34454159 | Gene delivery to pancreatic exocrine cells in vivo and in vitro. |
| Q88688533 | Genetically Engineered Mouse Models of K-Ras-Driven Lung and Pancreatic Tumors: Validation of Therapeutic Targets |
| Q27002973 | Genetically engineered mouse models of pancreatic adenocarcinoma |
| Q36675786 | Genetically engineered mouse models of pancreatic cancer |
| Q42738166 | Genetically-engineered mouse models for pancreatic cancer: Advances and current limitations |
| Q39556322 | Hepatitis B and C virus infections as possible risk factor for pancreatic adenocarcinoma |
| Q37103319 | High NOTCH activity induces radiation resistance in non small cell lung cancer |
| Q27023576 | Human Correlates of Provocative Questions in Pancreatic Pathology |
| Q52603542 | Id3 modulates cellular localization of bHLH Ptf1‐p48 protein |
| Q41856856 | Identification of Sox9-Dependent Acinar-to-Ductal Reprogramming as the Principal Mechanism for Initiation of Pancreatic Ductal Adenocarcinoma |
| Q33921337 | Identification of novel targets of CSL-dependent Notch signaling in hematopoiesis |
| Q41854156 | Impact of Sox9 dosage and Hes1-mediated Notch signaling in controlling the plasticity of adult pancreatic duct cells in mice. |
| Q40730244 | Impaired JNK signaling cooperates with KrasG12D expression to accelerate pancreatic ductal adenocarcinoma. |
| Q55252335 | In vivo reprogramming drives Kras-induced cancer development. |
| Q37068621 | Inactivating mutations of RNF43 confer Wnt dependency in pancreatic ductal adenocarcinoma |
| Q33486497 | Inactivation of TIF1gamma cooperates with Kras to induce cystic tumors of the pancreas |
| Q39885220 | Inhibition of gamma-secretase activity inhibits tumor progression in a mouse model of pancreatic ductal adenocarcinoma |
| Q50087583 | Injury, repair, inflammation and metaplasia in the stomach. |
| Q37334274 | Interaction of stellate cells with pancreatic carcinoma cells |
| Q36068611 | K-Ras and cyclooxygenase-2 coactivation augments intraductal papillary mucinous neoplasm and Notch1 mimicking human pancreas lesions |
| Q35083740 | KRAS Mouse Models: Modeling Cancer Harboring KRAS Mutations |
| Q33858490 | KRAS, Hedgehog, Wnt and the twisted developmental biology of pancreatic ductal adenocarcinoma. |
| Q100428129 | Kras mutation rate precisely orchestrates ductal derived pancreatic intraepithelial neoplasia and pancreatic cancer |
| Q30396675 | Leptin-Notch signaling axis is involved in pancreatic cancer progression |
| Q50517982 | Lineage Tracing Evidence for Transdifferentiation of Acinar to Duct Cells and Plasticity of Human Pancreas |
| Q41602913 | Lineage tracing reveals the dynamic contribution of Hes1+ cells to the developing and adult pancreas. |
| Q53176733 | Liver and pancreas: do similar embryonic development and tissue organization lead to similar mechanisms of tumorigenesis? |
| Q33754775 | Loss of the acinar-restricted transcription factor Mist1 accelerates Kras-induced pancreatic intraepithelial neoplasia |
| Q53215067 | Lunatic Fringe is a potent tumor suppressor in Kras-initiated pancreatic cancer |
| Q47246517 | Luteolin inhibits pancreatitis‑induced acinar‑ductal metaplasia, proliferation and epithelial‑mesenchymal transition of acinar cells. |
| Q35388201 | MT1-MMP cooperates with Kras(G12D) to promote pancreatic fibrosis through increased TGF-β signaling |
| Q38680703 | Macrophages and pancreatic ductal adenocarcinoma |
| Q39337159 | Maintenance of acinar cell organization is critical to preventing Kras-induced acinar-ductal metaplasia. |
| Q37998643 | Malignant glioma: lessons from genomics, mouse models, and stem cells |
| Q37421292 | Mechanisms of parenchymal injury and signaling pathways in ectatic ducts of chronic pancreatitis: implications for pancreatic carcinogenesis |
| Q50964950 | Mechanistic insights into the activity of Ptf1-p48 (pancreas transcription factor 1a): probing the interactions levels of Ptf1-p48 with E2A-E47 (transcription factor E2-alpha) and ID3 (inhibitor of DNA binding 3). |
| Q39250469 | Mesenchymal stem cells regulate epithelial-mesenchymal transition and tumor progression of pancreatic cancer cells |
| Q27334957 | Met receptor tyrosine kinase signaling induces secretion of the angiogenic chemokine interleukin-8/CXCL8 in pancreatic cancer |
| Q91871177 | Mimicking and Manipulating Pancreatic Acinar-to-Ductal Metaplasia in 3-dimensional Cell Culture |
| Q39441268 | Molecular Events in the Natural History of Pancreatic Cancer |
| Q37538446 | Molecular and cellular regulation of pancreatic acinar cell function |
| Q37809242 | Molecular biology of pancreatic ductal adenocarcinoma progression: aberrant activation of developmental pathways |
| Q26999345 | Molecular mechanism underlying lymphatic metastasis in pancreatic cancer |
| Q37176648 | Molecular signature of pancreatic adenocarcinoma: an insight from genotype to phenotype and challenges for targeted therapy |
| Q26860574 | Mouse models of pancreatic cancer |
| Q38974147 | NACK is an integral component of the Notch transcriptional activation complex and is critical for development and tumorigenesis. |
| Q26771258 | Notch Signaling in Pancreatic Development |
| Q41858645 | Notch and Kras in pancreatic cancer: at the crossroads of mutation, differentiation and signaling |
| Q39116742 | Notch as a tumour suppressor. |
| Q38094850 | Notch signaling in pancreatic cancer: oncogene or tumor suppressor? |
| Q37785204 | Notch signaling in solid tumors. |
| Q36881446 | Notch signaling pathway targeted therapy suppresses tumor progression and metastatic spread in pancreatic cancer. |
| Q34982347 | Notch signaling proteins: legitimate targets for cancer therapy |
| Q37867484 | Notch signalling in solid tumours: a little bit of everything but not all the time |
| Q39169243 | Notch1 Contributes to Chemoresistance to Gemcitabine and Serves as an Unfavorable Prognostic Indicator in Pancreatic Cancer |
| Q34533078 | Notch1 Is Not Required for Acinar-to-Ductal Metaplasia in a Model of Kras-Induced Pancreatic Ductal Adenocarcinoma |
| Q40865845 | Notch1 functions as a tumor suppressor in a model of K-ras-induced pancreatic ductal adenocarcinoma |
| Q34068002 | Notch2 is required for progression of pancreatic intraepithelial neoplasia and development of pancreatic ductal adenocarcinoma |
| Q39156056 | Nr5a2 maintains acinar cell differentiation and constrains oncogenic Kras-mediated pancreatic neoplastic initiation |
| Q42937848 | Numb regulates acinar cell dedifferentiation and survival during pancreatic damage and acinar-to-ductal metaplasia |
| Q38329689 | Ongoing Notch signaling maintains phenotypic fidelity in the adult exocrine pancreas. |
| Q34456438 | Organoid models of human and mouse ductal pancreatic cancer |
| Q87195787 | Orthotopic inflammation-related pancreatic carcinogenesis in a wild-type mouse induced by combined application of caerulein and dimethylbenzanthracene |
| Q51228273 | Overview of the pancreatic toxicity and carcinogenesis session |
| Q34101017 | PD2/Paf1 depletion in pancreatic acinar cells promotes acinar-to-ductal metaplasia |
| Q37587988 | PDX1 dynamically regulates pancreatic ductal adenocarcinoma initiation and maintenance |
| Q34616196 | PI3K regulation of RAC1 is required for KRAS-induced pancreatic tumorigenesis in mice |
| Q34134396 | PTEN loss accelerates KrasG12D-induced pancreatic cancer development |
| Q92078077 | PYK2 Is Involved in Premalignant Acinar Cell Reprogramming and Pancreatic Ductal Adenocarcinoma Maintenance by Phosphorylating β-CateninY654 |
| Q92959992 | Pancreatic Ductal Deletion of Hnf1b Disrupts Exocrine Homeostasis, Leads to Pancreatitis, and Facilitates Tumorigenesis |
| Q42556570 | Pancreatic cancer: translational research aspects and clinical implications. |
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| Q37249748 | Pancreatic duct glands are distinct ductal compartments that react to chronic injury and mediate Shh-induced metaplasia |
| Q27006729 | Pancreatic ductal cells in development, regeneration, and neoplasia |
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| Q42573716 | Pancreatitis-induced inflammation contributes to pancreatic cancer by inhibiting oncogene-induced senescence |
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| Q54273913 | Phosphorylation of Ribosomal Protein S6 Attenuates DNA Damage and Tumor Suppression during Development of Pancreatic Cancer |
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| Q36367896 | Quantitative Proteomic Analysis of Differentially Expressed Protein Profiles Involved in Pancreatic Ductal Adenocarcinoma |
| Q39841946 | Ras activity levels control the development of pancreatic diseases. |
| Q38014542 | Recent developments of transgenic and xenograft mouse models of pancreatic cancer for translational research |
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