| scholarly article | Q13442814 |
| P2093 | author name string | Chongmin Huan | |
| Devon John | |||
| Scott Tenner | |||
| Antonio E Alfonso | |||
| Kei Okochi | |||
| Kaylene Barrera | |||
| Albert Stanek | |||
| Cathy Mueller | |||
| Peiqi Ou | |||
| Zuzanna Niewiadomska | |||
| P2860 | cites work | A step-up approach or open necrosectomy for necrotizing pancreatitis | Q84090336 |
| Nuclear factor-κB in pancreatitis: Jack-of-all-trades, but which one is more important? | Q85484234 | ||
| Mammalian transcription factor ATF6 is synthesized as a transmembrane protein and activated by proteolysis in response to endoplasmic reticulum stress | Q22010800 | ||
| ER stress induces cleavage of membrane-bound ATF6 by the same proteases that process SREBPs | Q24290776 | ||
| XBP1 mRNA is induced by ATF6 and spliced by IRE1 in response to ER stress to produce a highly active transcription factor | Q24292102 | ||
| Hereditary pancreatitis is caused by a mutation in the cationic trypsinogen gene | Q24311652 | ||
| Protective effect against Parkinson's disease-related insults through the activation of XBP1 | Q24314837 | ||
| DAP-kinase-mediated phosphorylation on the BH3 domain of beclin 1 promotes dissociation of beclin 1 from Bcl-XL and induction of autophagy | Q24323360 | ||
| The molecular basis for selective inhibition of unconventional mRNA splicing by an IRE1-binding small molecule | Q24596804 | ||
| The ER stress factor XBP1s prevents amyloid-beta neurotoxicity | Q24598012 | ||
| Hereditary pancreatitis caused by mutation-induced misfolding of human cationic trypsinogen: a novel disease mechanism | Q24650338 | ||
| XBP-1 deficiency in the nervous system protects against amyotrophic lateral sclerosis by increasing autophagy | Q24655821 | ||
| Signal integration in the endoplasmic reticulum unfolded protein response | Q27860577 | ||
| Mutations in the unfolded protein response regulator ATF6 cause the cone dysfunction disorder achromatopsia | Q28115716 | ||
| IRE1 couples endoplasmic reticulum load to secretory capacity by processing the XBP-1 mRNA | Q28214814 | ||
| Gabexate for the prevention of pancreatic damage related to endoscopic retrograde cholangiopancreatography. Gabexate in digestive endoscopy--Italian Group | Q28288650 | ||
| Protein translation and folding are coupled by an endoplasmic-reticulum-resident kinase | Q28296183 | ||
| Oral treatment targeting the unfolded protein response prevents neurodegeneration and clinical disease in prion-infected mice | Q28300013 | ||
| Increased sensitivity to dextran sodium sulfate colitis in IRE1beta-deficient mice | Q28359871 | ||
| Regulated translation initiation controls stress-induced gene expression in mammalian cells | Q28506388 | ||
| Regulation of hepatic lipogenesis by the transcription factor XBP1 | Q28507784 | ||
| XBP-1 is required for biogenesis of cellular secretory machinery of exocrine glands | Q28510479 | ||
| CHOP induces death by promoting protein synthesis and oxidation in the stressed endoplasmic reticulum | Q28512249 | ||
| Endoplasmic reticulum stress links obesity, insulin action, and type 2 diabetes | Q28575190 | ||
| Regulation of glucose homeostasis through a XBP-1-FoxO1 interaction | Q28592129 | ||
| The unfolded protein response: from stress pathway to homeostatic regulation | Q29547396 | ||
| The presence of malfolded proteins in the endoplasmic reticulum signals the induction of glucose-regulated proteins | Q29620170 | ||
| PERK eIF2 alpha kinase is required to regulate the viability of the exocrine pancreas in mice | Q33295459 | ||
| IRE1α Disruption Causes Histological Abnormality of Exocrine Tissues, Increase of Blood Glucose Level, and Decrease of Serum Immunoglobulin Level | Q33707431 | ||
| Wolcott-Rallison syndrome | Q34148057 | ||
| Endoplasmic reticulum stress is chronically activated in chronic pancreatitis | Q34283421 | ||
| Burden of gastrointestinal disease in the United States: 2012 update | Q34293512 | ||
| American College of Gastroenterology guideline: management of acute pancreatitis | Q34360523 | ||
| ER stress activates NF-κB by integrating functions of basal IKK activity, IRE1 and PERK. | Q34460275 | ||
| Antiviral activity of a small-molecule inhibitor of arenavirus glycoprotein processing by the cellular site 1 protease | Q34485294 | ||
| Autocrine tumor necrosis factor alpha links endoplasmic reticulum stress to the membrane death receptor pathway through IRE1alpha-mediated NF-kappaB activation and down-regulation of TRAF2 expression | Q34563098 | ||
| Regulatory roles of the PI3K/Akt signaling pathway in rats with severe acute pancreatitis | Q35060697 | ||
| Pancreas-specific RelA/p65 truncation increases susceptibility of acini to inflammation-associated cell death following cerulein pancreatitis | Q35794043 | ||
| Targeting the UPR transcription factor XBP1 protects against Huntington's disease through the regulation of FoxO1 and autophagy | Q35909430 | ||
| The impact of the unfolded protein response on human disease | Q36062081 | ||
| Unfolded protein response transcription factor XBP-1 does not influence prion replication or pathogenesis. | Q36393034 | ||
| PERK is required in the adult pancreas and is essential for maintenance of glucose homeostasis | Q36435206 | ||
| Autophagy and pancreatitis | Q36454316 | ||
| Loss of acinar cell IKKα triggers spontaneous pancreatitis in mice | Q36793423 | ||
| The eIF2α/ATF4 pathway is essential for stress-induced autophagy gene expression | Q37148513 | ||
| From endoplasmic-reticulum stress to the inflammatory response | Q37225354 | ||
| C/EBP homologous protein deficiency aggravates acute pancreatitis and associated lung injury | Q37285290 | ||
| Activation of the Akt-NF-kappaB pathway by subtilase cytotoxin through the ATF6 branch of the unfolded protein response | Q37389501 | ||
| The XBP1 Arm of the Unfolded Protein Response Induces Fibrogenic Activity in Hepatic Stellate Cells Through Autophagy | Q37519318 | ||
| Cerulein-induced chronic pancreatitis does not require intra-acinar activation of trypsinogen in mice | Q37591305 | ||
| Binge ethanol exposure causes endoplasmic reticulum stress, oxidative stress and tissue injury in the pancreas | Q37688055 | ||
| Environmental and genetic stressors and the unfolded protein response in exocrine pancreatic function - a hypothesis | Q37864084 | ||
| Pathogenic mechanisms of acute pancreatitis | Q38034035 | ||
| The role of protein synthesis and digestive enzymes in acinar cell injury | Q38090903 | ||
| Druggable sensors of the unfolded protein response | Q38261088 | ||
| Pancreatic adaptive responses in alcohol abuse: Role of the unfolded protein response. | Q38367339 | ||
| Targeting the IRE1α-XBP1 branch of the unfolded protein response in human diseases | Q38489729 | ||
| IRE1α is essential for Xenopus pancreas development | Q38543615 | ||
| Combination of Alcohol and Cigarette Smoke Induces Endoplasmic Reticulum Stress and Cell Death in Pancreatic Acinar Cells | Q38694019 | ||
| Novel mechanism of enhancing IRE1α-XBP1 signalling via the PERK-ATF4 pathway | Q39941630 | ||
| Pharmacologic inhibition of site 1 protease activity inhibits sterol regulatory element-binding protein processing and reduces lipogenic enzyme gene expression and lipid synthesis in cultured cells and experimental animals | Q39968472 | ||
| STAT3 and STAT6 Signaling Pathways Synergize to Promote Cathepsin Secretion from Macrophages via IRE1α Activation | Q41434747 | ||
| Intra-acinar trypsinogen activation mediates early stages of pancreatic injury but not inflammation in mice with acute pancreatitis | Q41628354 | ||
| Constitutive IKK2 activation in acinar cells is sufficient to induce pancreatitis in vivo | Q41778161 | ||
| The CREB coactivator CRTC2 links hepatic ER stress and fasting gluconeogenesis | Q41791624 | ||
| Adaptive unfolded protein response attenuates alcohol-induced pancreatic damage | Q42026405 | ||
| Activating transcription factor 6 derepression mediates neuroprotection in Huntington disease. | Q42067930 | ||
| Induction of liver steatosis and lipid droplet formation in ATF6alpha-knockout mice burdened with pharmacological endoplasmic reticulum stress | Q42451739 | ||
| Activation of nuclear factor-κB in acinar cells increases the severity of pancreatitis in mice | Q42594838 | ||
| Physiological effects of enteral and parenteral feeding on pancreaticobiliary secretion in humans | Q44253277 | ||
| Acute pancreatitis caused by bortezomib. | Q53112700 | ||
| Early activation of endoplasmic reticulum stress is associated with arginine-induced acute pancreatitis. | Q53276855 | ||
| Deletion of IκBα Activates RelA to Reduce Acute Pancreatitis in Mice Through Up-regulation of Spi2A | Q60594555 | ||
| Localization of lysosomal and digestive enzymes in cytoplasmic vacuoles in caerulein-pancreatitis | Q68826236 | ||
| Acute pancreatitis | Q72384590 | ||
| Protease inhibitors in acute pancreatitis: lessons from the bench and failed clinical trials | Q81821242 | ||
| P433 | issue | 2 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | acute pancreatitis | Q854573 |
| P304 | page(s) | 37-46 | |
| P577 | publication date | 2018-09-29 | |
| P1433 | published in | World journal of gastrointestinal pathophysiology | Q27723538 |
| P1476 | title | Acinar cell injury induced by inadequate unfolded protein response in acute pancreatitis | |
| P478 | volume | 9 |
| Q93065136 | Commensal Escherichia coli Aggravates Acute Necrotizing Pancreatitis through Targeting of Intestinal Epithelial Cells | cites work | P2860 |
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