The role of X-box binding protein 1 in the hepatic response to refeeding in mice

scientific article published on 27 November 2018

The role of X-box binding protein 1 in the hepatic response to refeeding in mice is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1194/JLR.M086413
P932PMC publication ID6358297
P698PubMed publication ID30482806

P2093author name stringShantel Olivares
Anne S Henkel
P2860cites workXBP1 mRNA is induced by ATF6 and spliced by IRE1 in response to ER stress to produce a highly active transcription factorQ24292102
TRB3, a novel ER stress-inducible gene, is induced via ATF4-CHOP pathway and is involved in cell deathQ24298384
pXBP1(U) encoded in XBP1 pre-mRNA negatively regulates unfolded protein response activator pXBP1(S) in mammalian ER stress responseQ24303865
Unconventional splicing of XBP1 mRNA occurs in the cytoplasm during the mammalian unfolded protein responseQ24316572
A novel ER alpha-mannosidase-like protein accelerates ER-associated degradationQ24522524
Endoplasmic reticulum stress: cell life and death decisions.Q24536108
FGF19 as a postprandial, insulin-independent activator of hepatic protein and glycogen synthesisQ24617480
The ERO1 gene of yeast is required for oxidation of protein dithiols in the endoplasmic reticulumQ27936188
A critical role for the peroxisome proliferator-activated receptor alpha (PPARalpha) in the cellular fasting response: the PPARalpha-null mouse as a model of fatty acid oxidation disordersQ28116120
Nutrient control of glucose homeostasis through a complex of PGC-1alpha and SIRT1Q28237795
Protein translation and folding are coupled by an endoplasmic-reticulum-resident kinaseQ28296183
Regulation of hepatic lipogenesis by the transcription factor XBP1Q28507784
CHOP is implicated in programmed cell death in response to impaired function of the endoplasmic reticulumQ28509611
Gadd153 sensitizes cells to endoplasmic reticulum stress by down-regulating Bcl2 and perturbing the cellular redox stateQ28572472
Rapamycin inhibits postprandial-mediated X-box-binding protein-1 splicing in rat liverQ28577788
XBP-1 Regulates a Subset of Endoplasmic Reticulum Resident Chaperone Genes in the Unfolded Protein ResponseQ28585314
Rapid turnover of unspliced Xbp-1 as a factor that modulates the unfolded protein responseQ28589998
ATF6 activated by proteolysis binds in the presence of NF-Y (CBF) directly to the cis-acting element responsible for the mammalian unfolded protein responseQ28611502
The unfolded protein response: from stress pathway to homeostatic regulationQ29547396
ER stress triggers apoptosis by activating BH3-only protein BimQ29617730
Homocysteine supplementation attenuates the unfolded protein response in a murine nutritional model of steatohepatitisQ33553744
AT-1 is the ER membrane acetyl-CoA transporter and is essential for cell viabilityQ34129501
P58IPK, a novel endoplasmic reticulum stress-inducible protein and potential negative regulator of eIF2alpha signalingQ34179244
SLC33A1/AT-1 protein regulates the induction of autophagy downstream of IRE1/XBP1 pathwayQ34287569
A chronic high-cholesterol diet paradoxically suppresses hepatic CYP7A1 expression in FVB/NJ miceQ34503382
Dual and opposing roles of the unfolded protein response regulated by IRE1alpha and XBP1 in proinsulin processing and insulin secretionQ35008217
ER stress and the unfolded protein responseQ35986601
Silencing of lipid metabolism genes through IRE1α-mediated mRNA decay lowers plasma lipids in mice.Q36330343
Role of the cytoplasmic segments of Sec61alpha in the ribosome-binding and translocation-promoting activities of the Sec61 complexQ36342470
Hepatic Xbp1 Gene Deletion Promotes Endoplasmic Reticulum Stress-induced Liver Injury and ApoptosisQ36444262
The Xbp1s/GalE axis links ER stress to postprandial hepatic metabolismQ36497151
Diet-induced obesity alters protein synthesis: tissue-specific effects in fasted versus fed miceQ36570540
IRE1α-XBP1s induces PDI expression to increase MTP activity for hepatic VLDL assembly and lipid homeostasisQ36600595
Endoplasmic reticulum stress signaling in diseaseQ36610455
Insulin selectively increases SREBP-1c mRNA in the livers of rats with streptozotocin-induced diabetesQ36698641
Regulation of sterol regulatory element binding proteins in livers of fasted and refed miceQ37375695
Accelerated fatty acid oxidation in muscle averts fasting-induced hepatic steatosis in SJL/J miceQ37433454
Hepatic Elovl6 gene expression is regulated by the synergistic action of ChREBP and SREBP-1cQ38291264
Physiological and molecular mechanisms involved in nutritional regulation of fatty acid synthesisQ40576744
UPR pathways combine to prevent hepatic steatosis caused by ER stress-mediated suppression of transcriptional master regulators.Q42041480
Insulin and dietary fructose induce stearoyl-CoA desaturase 1 gene expression of diabetic miceQ42282460
Concerted elevation of acyl-coenzyme A:diacylglycerol acyltransferase (DGAT) activity through independent stimulation of mRNA expression of DGAT1 and DGAT2 by carbohydrate and insulinQ42527526
Differentiation-induced gene expression in 3T3-L1 preadipocytes. Characterization of a differentially expressed gene encoding stearoyl-CoA desaturaseQ42801595
Diminished hepatic response to fasting/refeeding and liver X receptor agonists in mice with selective deficiency of sterol regulatory element-binding protein-1cQ43848640
Metabolomics (liver and blood profiling) in a mouse model in response to fasting: a study of hepatic steatosisQ46975368
Induction of fibroblast growth factor 21 does not require activation of the hepatic X-box binding protein 1 in miceQ47104419
Role of Sec61alpha in the regulated transfer of the ribosome-nascent chain complex from the signal recognition particle to the translocation channelQ73453521
That which does not kill me makes me stronger: adapting to chronic ER stressQ81405125
P433issue2
P304page(s)353-359
P577publication date2018-11-27
P1433published inJournal of Lipid ResearchQ6295449
P1476titleThe role of X-box binding protein 1 in the hepatic response to refeeding in mice
P478volume60

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