| scholarly article | Q13442814 |
| P50 | author | Ling Qi | Q37375214 |
| Qiaoming Long | Q43194579 | ||
| Kezhong Zhang | Q64604817 | ||
| Asmita Bhattacharya | Q86563725 | ||
| P2093 | author name string | Sander Kersten | |
| Ming Liu | |||
| Lei Yin | |||
| Shengyi Sun | |||
| Heting Wang | |||
| P2860 | cites work | CREB-H: a novel mammalian transcription factor belonging to the CREB/ATF family and functioning via the box-B element with a liver-specific expression | Q24291231 |
| Endoplasmic reticulum stress activates cleavage of CREBH to induce a systemic inflammatory response | Q24304232 | ||
| FGF-21 as a novel metabolic regulator | Q24523933 | ||
| Fibroblast growth factor 21 regulates energy metabolism by activating the AMPK-SIRT1-PGC-1alpha pathway | Q24607012 | ||
| Endocrine fibroblast growth factors 15/19 and 21: from feast to famine | Q24629260 | ||
| ER stress controls iron metabolism through induction of hepcidin | Q24630714 | ||
| Inventing new medicines: The FGF21 story | Q26823319 | ||
| The delicate balance between secreted protein folding and endoplasmic reticulum-associated degradation in human physiology | Q27015793 | ||
| Hrd1 suppresses Nrf2-mediated cellular protection during liver cirrhosis | Q28385517 | ||
| Regulation of hepatic lipogenesis by the transcription factor XBP1 | Q28507784 | ||
| FGF21 is an endocrine signal of protein restriction | Q28572009 | ||
| Fibroblast growth factor 21 is induced by endoplasmic reticulum stress | Q28581415 | ||
| Deficiency of suppressor enhancer Lin12 1 like (SEL1L) in mice leads to systemic endoplasmic reticulum stress and embryonic lethality | Q28587457 | ||
| Essential role of synoviolin in embryogenesis | Q28588247 | ||
| The ER-associated degradation adaptor protein Sel1L regulates LPL secretion and lipid metabolism | Q28594858 | ||
| Hepatic fibroblast growth factor 21 is regulated by PPARalpha and is a key mediator of hepatic lipid metabolism in ketotic states | Q29615208 | ||
| Endocrine regulation of the fasting response by PPARalpha-mediated induction of fibroblast growth factor 21 | Q29615209 | ||
| A Phos-tag-based approach reveals the extent of physiological endoplasmic reticulum stress | Q33641274 | ||
| ATF4- and CHOP-dependent induction of FGF21 through endoplasmic reticulum stress | Q33681667 | ||
| Hepatic FGF21 expression is induced at birth via PPARalpha in response to milk intake and contributes to thermogenic activation of neonatal brown fat. | Q33762194 | ||
| The starvation hormone, fibroblast growth factor-21, extends lifespan in mice. | Q34306190 | ||
| FGF21 acts centrally to induce sympathetic nerve activity, energy expenditure, and weight loss. | Q34315567 | ||
| Fibroblast growth factor 21 is regulated by the IRE1α-XBP1 branch of the unfolded protein response and counteracts endoplasmic reticulum stress-induced hepatic steatosis | Q34396839 | ||
| Hrd1-mediated BLIMP-1 ubiquitination promotes dendritic cell MHCII expression for CD4 T cell priming during inflammation | Q34533090 | ||
| Acute exercise induces FGF21 expression in mice and in healthy humans | Q34718217 | ||
| The interplay of Hrd3 and the molecular chaperone system ensures efficient degradation of malfolded secretory proteins. | Q34960632 | ||
| The E3 ligase synoviolin controls body weight and mitochondrial biogenesis through negative regulation of PGC-1β | Q35527134 | ||
| Dissociation of inositol-requiring enzyme (IRE1α)-mediated c-Jun N-terminal kinase activation from hepatic insulin resistance in conditional X-box-binding protein-1 (XBP1) knock-out mice | Q35709954 | ||
| FGF21 regulates PGC-1α and browning of white adipose tissues in adaptive thermogenesis | Q35755169 | ||
| IRE1α activation protects mice against acetaminophen-induced hepatotoxicity | Q35760821 | ||
| Epithelial Sel1L is required for the maintenance of intestinal homeostasis | Q35859583 | ||
| Endoplasmic reticulum-tethered transcription factor cAMP responsive element-binding protein, hepatocyte specific, regulates hepatic lipogenesis, fatty acid oxidation, and lipolysis upon metabolic stress in mice | Q35870151 | ||
| The ATP-P2X7 signaling axis is dispensable for obesity-associated inflammasome activation in adipose tissue | Q35976791 | ||
| Silencing of lipid metabolism genes through IRE1α-mediated mRNA decay lowers plasma lipids in mice. | Q36330343 | ||
| IRE1α is an endogenous substrate of endoplasmic-reticulum-associated degradation | Q36348190 | ||
| Hepatic Xbp1 Gene Deletion Promotes Endoplasmic Reticulum Stress-induced Liver Injury and Apoptosis | Q36444262 | ||
| SEL1L nucleates a protein complex required for dislocation of misfolded glycoproteins | Q36858538 | ||
| Inhibition of growth hormone signaling by the fasting-induced hormone FGF21. | Q36953853 | ||
| Direct and essential function for Hrd3 in ER-associated degradation | Q36957336 | ||
| CREBH-FGF21 axis improves hepatic steatosis by suppressing adipose tissue lipolysis | Q37005595 | ||
| The ER membrane-anchored ubiquitin ligase Hrd1 is a positive regulator of T-cell immunity. | Q37099446 | ||
| FGF21 regulates metabolism and circadian behavior by acting on the nervous system | Q37161082 | ||
| Endoplasmic reticulum-resident E3 ubiquitin ligase Hrd1 controls B-cell immunity through degradation of the death receptor CD95/Fas | Q37264217 | ||
| Sel1L is indispensable for mammalian endoplasmic reticulum-associated degradation, endoplasmic reticulum homeostasis, and survival | Q37571396 | ||
| Liver-enriched transcription factor CREBH interacts with peroxisome proliferator-activated receptor α to regulate metabolic hormone FGF21. | Q37594100 | ||
| Transcriptional regulation of apolipoprotein A-IV by the transcription factor CREBH | Q37716353 | ||
| FGF21 as a therapeutic reagent | Q37991265 | ||
| Loss of Transcription Factor CREBH Accelerates Diet-Induced Atherosclerosis in Ldlr-/- Mice | Q38291709 | ||
| Understanding the Physiology of FGF21. | Q38665376 | ||
| FGF21 Regulates Metabolism Through Adipose-Dependent and -Independent Mechanisms | Q38852382 | ||
| FGF21 treatment ameliorates alcoholic fatty liver through activation of AMPK-SIRT1 pathway | Q38943985 | ||
| New Insights into the Physiological Role of Endoplasmic Reticulum-Associated Degradation | Q39107838 | ||
| The Sel1L-Hrd1 Endoplasmic Reticulum-Associated Degradation Complex Manages a Key Checkpoint in B Cell Development | Q39446306 | ||
| N-linked glycosylation is required for optimal proteolytic activation of membrane-bound transcription factor CREB-H. | Q39720383 | ||
| Detecting and quantitating physiological endoplasmic reticulum stress | Q39789909 | ||
| The IRE1alpha-XBP1 pathway of the unfolded protein response is required for adipogenesis | Q39843795 | ||
| The unfolded protein response transducer IRE1α prevents ER stress-induced hepatic steatosis | Q41904044 | ||
| FGF21 mediates the lipid metabolism response to amino acid starvation. | Q42034608 | ||
| ER-associated degradation is required for vasopressin prohormone processing and systemic water homeostasis. | Q47111673 | ||
| Hepatic CREB3L3 controls whole-body energy homeostasis and improves obesity and diabetes. | Q47727964 | ||
| Comparison of turnover rates of proteins of the brain, liver and kidney in mouse in vivo following long term labeling | Q48393340 | ||
| Hypothalamic ER-associated degradation regulates POMC maturation, feeding, and age-associated obesity. | Q49851092 | ||
| Trafficking of the bZIP transmembrane transcription factor CREB-H into alternate pathways of ERAD and stress-regulated intramembrane proteolysis | Q50336411 | ||
| Activating transcription factor 4-dependent induction of FGF21 during amino acid deprivation. | Q51362717 | ||
| In vivo ChIP for the analysis of microdissected tissue samples. | Q51544190 | ||
| Autophagy deficiency leads to protection from obesity and insulin resistance by inducing Fgf21 as a mitokine | Q63609769 | ||
| P4510 | describes a project that uses | ImageJ | Q1659584 |
| P433 | issue | 22 | |
| P407 | language of work or name | English | Q1860 |
| P577 | publication date | 2018-11-02 | |
| P1433 | published in | The EMBO Journal | Q1278554 |
| P1476 | title | Hepatic Sel1L-Hrd1 ER-associated degradation (ERAD) manages FGF21 levels and systemic metabolism via CREBH | |
| P478 | volume | 37 |
| Q91658827 | Elevation of the unfolded protein response increases RANKL expression |
| Q90470513 | Endoplasmic reticulum-associated degradation regulates mitochondrial dynamics in brown adipocytes |
| Q90224598 | Fibroblast Growth Factor 21 and the Adaptive Response to Nutritional Challenges |
| Q58616109 | Hepatic ERAD takes control of the organism |
| Q90390291 | Sel1L-Hrd1 ER-associated degradation maintains β-cell identity via TGFβ signaling |
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