scholarly article | Q13442814 |
P50 | author | Edward Thorp | Q41305505 |
Tracie A. Seimon | Q59062028 | ||
P2093 | author name string | Gang Li | |
Ira Tabas | |||
George Kuriakose | |||
P2860 | cites work | NAD(P)H oxidase Nox-4 mediates 7-ketocholesterol-induced endoplasmic reticulum stress and apoptosis in human aortic smooth muscle cells | Q24559719 |
CHOP is implicated in programmed cell death in response to impaired function of the endoplasmic reticulum | Q28509611 | ||
IRE1 signaling affects cell fate during the unfolded protein response | Q29615502 | ||
Chemical chaperones reduce ER stress and restore glucose homeostasis in a mouse model of type 2 diabetes | Q29615503 | ||
TDAG51 Is Induced by Homocysteine, Promotes Detachment-mediated Programmed Cell Death, and Contributes to the Development of Atherosclerosis in Hyperhomocysteinemia | Q30927502 | ||
Endothelial cell apoptosis: biochemical characteristics and potential implications for atherosclerosis | Q34363721 | ||
The unfolding tale of the unfolded protein response | Q34486042 | ||
Animal models of spontaneous plaque rupture: the holy grail of experimental atherosclerosis research. | Q34586858 | ||
Translational control in the endoplasmic reticulum stress response | Q35001600 | ||
Orchestrating the unfolded protein response in health and disease | Q35001604 | ||
Pathologic assessment of the vulnerable human coronary plaque | Q35583211 | ||
Niemann-Pick C heterozygosity confers resistance to lesional necrosis and macrophage apoptosis in murine atherosclerosis | Q35918384 | ||
Consequences and therapeutic implications of macrophage apoptosis in atherosclerosis: the importance of lesion stage and phagocytic efficiency | Q36248636 | ||
Cholesterol-induced macrophage apoptosis requires ER stress pathways and engagement of the type A scavenger receptor | Q36320445 | ||
Phagocytosis in atherosclerosis: Molecular mechanisms and implications for plaque progression and stability. | Q36644805 | ||
The emerging role of vascular smooth muscle cell apoptosis in atherosclerosis and plaque stability | Q36678905 | ||
Apoptosis and efferocytosis in mouse models of atherosclerosis | Q37065937 | ||
Minimally modified LDL binds to CD14, induces macrophage spreading via TLR4/MD-2, and inhibits phagocytosis of apoptotic cells | Q40691206 | ||
Induction of CHOP and apoptosis by nitric oxide in p53-deficient microglial cells | Q40776210 | ||
Atherosclerosis regression, plaque disruption, and cardiovascular events: a rationale for lipid lowering in coronary artery disease | Q40897992 | ||
Macrophages and atherosclerotic plaque stability. | Q41235093 | ||
Mertk receptor mutation reduces efferocytosis efficiency and promotes apoptotic cell accumulation and plaque necrosis in atherosclerotic lesions of apoe-/- mice | Q42134595 | ||
The unfolded protein response is an important regulator of inflammatory genes in endothelial cells | Q42500350 | ||
The endoplasmic reticulum is the site of cholesterol-induced cytotoxicity in macrophages | Q44542540 | ||
Oxidized low-density lipoproteins trigger endoplasmic reticulum stress in vascular cells: prevention by oxygen-regulated protein 150 expression. | Q46184567 | ||
Activation of the unfolded protein response occurs at all stages of atherosclerotic lesion development in apolipoprotein E-deficient mice | Q46421418 | ||
Peroxynitrite causes endoplasmic reticulum stress and apoptosis in human vascular endothelium: implications in atherogenesis | Q46740172 | ||
Cholesterol-induced apoptotic macrophages elicit an inflammatory response in phagocytes, which is partially attenuated by the Mer receptor | Q46870067 | ||
Phagocytosis of apoptotic cells by macrophages is impaired in atherosclerosis. | Q50769120 | ||
Free cholesterol loading of macrophages induces apoptosis involving the fas pathway. | Q54053135 | ||
Macrophage insulin receptor deficiency increases ER stress-induced apoptosis and necrotic core formation in advanced atherosclerotic lesions. | Q54610885 | ||
Amino Acid Limitation Induces Expression ofCHOP, a CCAAT/Enhancer Binding Protein-related Gene, at Both Transcriptional and Post-transcriptional Levels | Q57033253 | ||
Apoptosis in the atherosclerotic plaque: quantitative and qualitative aspects | Q77380909 | ||
Increased endoplasmic reticulum stress in atherosclerotic plaques associated with acute coronary syndrome | Q80819961 | ||
P433 | issue | 5 | |
P921 | main subject | atherosclerosis | Q12252367 |
apoptotic process | Q14599311 | ||
P304 | page(s) | 474-481 | |
P577 | publication date | 2009-05-01 | |
P1433 | published in | Cell Metabolism | Q1254684 |
P1476 | title | Reduced apoptosis and plaque necrosis in advanced atherosclerotic lesions of Apoe-/- and Ldlr-/- mice lacking CHOP. | |
P478 | volume | 9 |
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Q55456504 | Endoplasmic Reticulum Stress in Metabolic Disorders. |
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