scholarly article | Q13442814 |
P50 | author | Stefan Knapp | Q40296391 |
James W. Murray | Q41621508 | ||
Apirat Chaikuad | Q42118607 | ||
Chancievan Thangaratnarajah | Q58203115 | ||
Frank von Delft | Q28050592 | ||
Tobias Krojer | Q28050596 | ||
Opher Gileadi | Q38321459 | ||
P2093 | author name string | Alex N Bullock | |
Ashley C W Pike | |||
Brian D Marsden | |||
Vikram Ayinampudi | |||
Christopher D O Cooper | |||
Peter Canning | |||
Tracy Keates | |||
Viktorija Hojzan | |||
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Structure of the SOCS4-ElonginB/C Complex Reveals a Distinct SOCS Box Interface and the Molecular Basis for SOCS-Dependent EGFR Degradation | Q27649020 | ||
Novel -Propeller of the BTB-Kelch Protein Krp1 Provides a Binding Site for Lasp-1 That Is Necessary for Pseudopodial Extension | Q27657284 | ||
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Keap1 recruits Neh2 through binding to ETGE and DLG motifs: characterization of the two-site molecular recognition model | Q28910182 | ||
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Keap1-dependent proteasomal degradation of transcription factor Nrf2 contributes to the negative regulation of antioxidant response element-driven gene expression | Q29615644 | ||
Structural basis for defects of Keap1 activity provoked by its point mutations in lung cancer | Q29616499 | ||
Keap1 is a forked-stem dimer structure with two large spheres enclosing the intervening, double glycine repeat, and C-terminal domains | Q33734064 | ||
Kelch-like homologue 9 mutation is associated with an early onset autosomal dominant distal myopathy | Q33944189 | ||
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Mutations in kelch-like 3 and cullin 3 cause hypertension and electrolyte abnormalities | Q35754537 | ||
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P433 | issue | 11 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | cell biology | Q7141 |
P304 | page(s) | 7803-14 | |
P577 | publication date | 2013-03-15 | |
P1433 | published in | Journal of Biological Chemistry | Q867727 |
P1476 | title | Structural Basis for Cul3 Protein Assembly with the BTB-Kelch Family of E3 Ubiquitin Ligases | |
P478 | volume | 288 |
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Q36994823 | Analysis of dimerization of BTB-IVR domains of Keap1 and its interaction with Cul3, by molecular modeling |
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Q41780358 | CUL3 and protein kinases: insights from PLK1/KLHL22 interaction |
Q38822665 | Characterizations of Three Major Cysteine Sensors of Keap1 in Stress Response. |
Q30952223 | Comparative Meta-Analysis of Transcriptomics Data during Cellular Senescence and In Vivo Tissue Ageing |
Q92134518 | Comparative structural and evolutionary analyses predict functional sites in the artemisinin resistance malaria protein K13 |
Q100311434 | Control of craniofacial and brain development by Cullin3-RING ubiquitin ligases: Lessons from human disease genetics |
Q38673650 | Crystal Structure of the Cul2-Rbx1-EloBC-VHL Ubiquitin Ligase Complex |
Q34661978 | Crystal structure of KLHL3 in complex with Cullin3 |
Q52678290 | Crystal structure of the Kelch domain of human NS1-binding protein at 1.98 Å resolution. |
Q26752753 | Cul3-KLHL20 ubiquitin ligase: physiological functions, stress responses, and disease implications |
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Q37133559 | Cullin 3 as a novel target in diverse pathologies |
Q57662146 | Cullin 3 targets methionine adenosyltransferase IIα for ubiquitylation-mediated degradation and regulates colorectal cancer cell proliferation |
Q47284253 | Cullin 3-Based Ubiquitin Ligases as Master Regulators of Mammalian Cell Differentiation. |
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Q47246481 | Deficiency in Kelch protein Klhl31 causes congenital myopathy in mice |
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Q60920505 | Dysregulation of Nrf2 in Hepatocellular Carcinoma: Role in Cancer Progression and Chemoresistance |
Q46331048 | GCL and CUL3 Control the Switch between Cell Lineages by Mediating Localized Degradation of an RTK. |
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Q34393793 | Germline loss-of-function mutations in LZTR1 predispose to an inherited disorder of multiple schwannomas |
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Q28975623 | Kelch Repeat and BTB Domain Containing Protein 5 (Kbtbd5) Regulates Skeletal Muscle Myogenesis through the E2F1-DP1 Complex |
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Q38694939 | Klhl6 Deficiency Impairs Transitional B Cell Survival and Differentiation. |
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Q28394732 | Mechanisms of activation of the transcription factor Nrf2 by redox stressors, nutrient cues, and energy status and the pathways through which it attenuates degenerative disease |
Q91826570 | Medulloblastomics revisited: biological and clinical insights from thousands of patients |
Q47257087 | Missense Variants in RHOBTB2 Cause a Developmental and Epileptic Encephalopathy in Humans, and Altered Levels Cause Neurological Defects in Drosophila. |
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